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Journal Abstract Search

672 related items for PubMed ID: 20007902

  • 21. KLF15 overexpression in myocytes fails to ameliorate ALS-related pathology or extend the lifespan of SOD1G93A mice.
    Massopust R, Juros D, Shapiro D, Lopes M, Haldar SM, Taetzsch T, Valdez G.
    Neurobiol Dis; 2022 Jan; 162():105583. PubMed ID: 34902552
    [Abstract] [Full Text] [Related]

  • 22. VAChT overexpression increases acetylcholine at the synaptic cleft and accelerates aging of neuromuscular junctions.
    Sugita S, Fleming LL, Wood C, Vaughan SK, Gomes MP, Camargo W, Naves LA, Prado VF, Prado MA, Guatimosim C, Valdez G.
    Skelet Muscle; 2016 Jan; 6():31. PubMed ID: 27713817
    [Abstract] [Full Text] [Related]

  • 23. Wnt antagonist FRZB is a muscle biomarker of denervation atrophy in amyotrophic lateral sclerosis.
    Kwan T, Kazamel M, Thoenes K, Si Y, Jiang N, King PH.
    Sci Rep; 2020 Oct 07; 10(1):16679. PubMed ID: 33028902
    [Abstract] [Full Text] [Related]

  • 24. The anabolic/androgenic steroid nandrolone exacerbates gene expression modifications induced by mutant SOD1 in muscles of mice models of amyotrophic lateral sclerosis.
    Galbiati M, Onesto E, Zito A, Crippa V, Rusmini P, Mariotti R, Bentivoglio M, Bendotti C, Poletti A.
    Pharmacol Res; 2012 Feb 07; 65(2):221-30. PubMed ID: 22178654
    [Abstract] [Full Text] [Related]

  • 25. Skeletal muscle in amyotrophic lateral sclerosis.
    Shefner JM, Musaro A, Ngo ST, Lunetta C, Steyn FJ, Robitaille R, De Carvalho M, Rutkove S, Ludolph AC, Dupuis L.
    Brain; 2023 Nov 02; 146(11):4425-4436. PubMed ID: 37327376
    [Abstract] [Full Text] [Related]

  • 26. Macrophage-mediated inflammation and glial response in the skeletal muscle of a rat model of familial amyotrophic lateral sclerosis (ALS).
    Van Dyke JM, Smit-Oistad IM, Macrander C, Krakora D, Meyer MG, Suzuki M.
    Exp Neurol; 2016 Mar 02; 277():275-282. PubMed ID: 26775178
    [Abstract] [Full Text] [Related]

  • 27. Skeletal muscle-restricted expression of human SOD1 causes motor neuron degeneration in transgenic mice.
    Wong M, Martin LJ.
    Hum Mol Genet; 2010 Jun 01; 19(11):2284-302. PubMed ID: 20223753
    [Abstract] [Full Text] [Related]

  • 28. Neuromuscular junction destruction during amyotrophic lateral sclerosis: insights from transgenic models.
    Dupuis L, Loeffler JP.
    Curr Opin Pharmacol; 2009 Jun 01; 9(3):341-6. PubMed ID: 19386549
    [Abstract] [Full Text] [Related]

  • 29. Amyloid precursor protein (APP) contributes to pathology in the SOD1(G93A) mouse model of amyotrophic lateral sclerosis.
    Bryson JB, Hobbs C, Parsons MJ, Bosch KD, Pandraud A, Walsh FS, Doherty P, Greensmith L.
    Hum Mol Genet; 2012 Sep 01; 21(17):3871-82. PubMed ID: 22678056
    [Abstract] [Full Text] [Related]

  • 30. Dihydrotestosterone ameliorates degeneration in muscle, axons and motoneurons and improves motor function in amyotrophic lateral sclerosis model mice.
    Yoo YE, Ko CP.
    PLoS One; 2012 Sep 01; 7(5):e37258. PubMed ID: 22606355
    [Abstract] [Full Text] [Related]

  • 31. Muscle microRNA signatures as biomarkers of disease progression in amyotrophic lateral sclerosis.
    Si Y, Cui X, Crossman DK, Hao J, Kazamel M, Kwon Y, King PH.
    Neurobiol Dis; 2018 Jun 01; 114():85-94. PubMed ID: 29486297
    [Abstract] [Full Text] [Related]

  • 32. ATF3 expression improves motor function in the ALS mouse model by promoting motor neuron survival and retaining muscle innervation.
    Seijffers R, Zhang J, Matthews JC, Chen A, Tamrazian E, Babaniyi O, Selig M, Hynynen M, Woolf CJ, Brown RH.
    Proc Natl Acad Sci U S A; 2014 Jan 28; 111(4):1622-7. PubMed ID: 24474789
    [Abstract] [Full Text] [Related]

  • 33. Time course of preferential motor unit loss in the SOD1 G93A mouse model of amyotrophic lateral sclerosis.
    Hegedus J, Putman CT, Gordon T.
    Neurobiol Dis; 2007 Nov 28; 28(2):154-64. PubMed ID: 17766128
    [Abstract] [Full Text] [Related]

  • 34. Overview of Impaired BDNF Signaling, Their Coupled Downstream Serine-Threonine Kinases and SNARE/SM Complex in the Neuromuscular Junction of the Amyotrophic Lateral Sclerosis Model SOD1-G93A Mice.
    Just-Borràs L, Hurtado E, Cilleros-Mañé V, Biondi O, Charbonnier F, Tomàs M, Garcia N, Lanuza MA, Tomàs J.
    Mol Neurobiol; 2019 Oct 28; 56(10):6856-6872. PubMed ID: 30929165
    [Abstract] [Full Text] [Related]

  • 35. Regeneration of neuromuscular synapses: action of microRNA-206.
    Lin N, Friedlander RM.
    Neurosurgery; 2010 Apr 28; 66(4):N19-20. PubMed ID: 20305481
    [No Abstract] [Full Text] [Related]

  • 36. Severe muscle wasting and denervation in mice lacking the RNA-binding protein ZFP106.
    Anderson DM, Cannavino J, Li H, Anderson KM, Nelson BR, McAnally J, Bezprozvannaya S, Liu Y, Lin W, Liu N, Bassel-Duby R, Olson EN.
    Proc Natl Acad Sci U S A; 2016 Aug 02; 113(31):E4494-503. PubMed ID: 27418600
    [Abstract] [Full Text] [Related]

  • 37. Effects of Class II-Selective Histone Deacetylase Inhibitor on Neuromuscular Function and Disease Progression in SOD1-ALS Mice.
    Buonvicino D, Felici R, Ranieri G, Caramelli R, Lapucci A, Cavone L, Muzzi M, Di Pietro L, Bernardini C, Zwergel C, Valente S, Mai A, Chiarugi A.
    Neuroscience; 2018 May 21; 379():228-238. PubMed ID: 29588251
    [Abstract] [Full Text] [Related]

  • 38. Glycoprotein nonmetastatic melanoma protein B ameliorates skeletal muscle lesions in a SOD1G93A mouse model of amyotrophic lateral sclerosis.
    Nagahara Y, Shimazawa M, Tanaka H, Ono Y, Noda Y, Ohuchi K, Tsuruma K, Katsuno M, Sobue G, Hara H.
    J Neurosci Res; 2015 Oct 21; 93(10):1552-66. PubMed ID: 26140698
    [Abstract] [Full Text] [Related]

  • 39. Intact single muscle fibres from SOD1G93A amyotrophic lateral sclerosis mice display preserved specific force, fatigue resistance and training-like adaptations.
    Cheng AJ, Allodi I, Chaillou T, Schlittler M, Ivarsson N, Lanner JT, Thams S, Hedlund E, Andersson DC.
    J Physiol; 2019 Jun 21; 597(12):3133-3146. PubMed ID: 31074054
    [Abstract] [Full Text] [Related]

  • 40. Muscle specific kinase (MuSK) activation preserves neuromuscular junctions in the diaphragm but is not sufficient to provide a functional benefit in the SOD1G93A mouse model of ALS.
    Sengupta-Ghosh A, Dominguez SL, Xie L, Barck KH, Jiang Z, Earr T, Imperio J, Phu L, Budayeva HG, Kirkpatrick DS, Cai H, Eastham-Anderson J, Ngu H, Foreman O, Hedehus M, Reichelt M, Hotzel I, Shang Y, Carano RAD, Ayalon G, Easton A.
    Neurobiol Dis; 2019 Apr 21; 124():340-352. PubMed ID: 30528255
    [Abstract] [Full Text] [Related]

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