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Journal Abstract Search

739 related items for PubMed ID: 20008901

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  • 4. Knocking down metabotropic glutamate receptor 1 improves survival and disease progression in the SOD1(G93A) mouse model of amyotrophic lateral sclerosis.
    Milanese M, Giribaldi F, Melone M, Bonifacino T, Musante I, Carminati E, Rossi PI, Vergani L, Voci A, Conti F, Puliti A, Bonanno G.
    Neurobiol Dis; 2014 Apr; 64():48-59. PubMed ID: 24361555
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  • 5. Human Cu/Zn superoxide dismutase (SOD1) overexpression in mice causes mitochondrial vacuolization, axonal degeneration, and premature motoneuron death and accelerates motoneuron disease in mice expressing a familial amyotrophic lateral sclerosis mutant SOD1.
    Jaarsma D, Haasdijk ED, Grashorn JA, Hawkins R, van Duijn W, Verspaget HW, London J, Holstege JC.
    Neurobiol Dis; 2000 Dec; 7(6 Pt B):623-43. PubMed ID: 11114261
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  • 6. Complete dissociation of motor neuron death from motor dysfunction by Bax deletion in a mouse model of ALS.
    Gould TW, Buss RR, Vinsant S, Prevette D, Sun W, Knudson CM, Milligan CE, Oppenheim RW.
    J Neurosci; 2006 Aug 23; 26(34):8774-86. PubMed ID: 16928866
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  • 7. RNS60 exerts therapeutic effects in the SOD1 ALS mouse model through protective glia and peripheral nerve rescue.
    Vallarola A, Sironi F, Tortarolo M, Gatto N, De Gioia R, Pasetto L, De Paola M, Mariani A, Ghosh S, Watson R, Kalmes A, Bonetto V, Bendotti C.
    J Neuroinflammation; 2018 Mar 01; 15(1):65. PubMed ID: 29495962
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  • 9. Effect of thymic stimulation of CD4+ T cell expansion on disease onset and progression in mutant SOD1 mice.
    Sheean RK, Weston RH, Perera ND, D'Amico A, Nutt SL, Turner BJ.
    J Neuroinflammation; 2015 Feb 27; 12():40. PubMed ID: 25889790
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  • 10. Inducible nitric oxide synthase is present in motor neuron mitochondria and Schwann cells and contributes to disease mechanisms in ALS mice.
    Chen K, Northington FJ, Martin LJ.
    Brain Struct Funct; 2010 Mar 27; 214(2-3):219-34. PubMed ID: 19888600
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  • 12. Novel behavioural characteristics of the superoxide dismutase 1 G93A (SOD1G93A ) mouse model of amyotrophic lateral sclerosis include sex-dependent phenotypes.
    Kreilaus F, Guerra S, Masanetz R, Menne V, Yerbury J, Karl T.
    Genes Brain Behav; 2020 Feb 27; 19(2):e12604. PubMed ID: 31412164
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  • 13. Overexpression of metallothionein-I, a copper-regulating protein, attenuates intracellular copper dyshomeostasis and extends lifespan in a mouse model of amyotrophic lateral sclerosis caused by mutant superoxide dismutase-1.
    Tokuda E, Okawa E, Watanabe S, Ono S.
    Hum Mol Genet; 2014 Mar 01; 23(5):1271-85. PubMed ID: 24163136
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  • 14. Nuclear localization of human SOD1 and mutant SOD1-specific disruption of survival motor neuron protein complex in transgenic amyotrophic lateral sclerosis mice.
    Gertz B, Wong M, Martin LJ.
    J Neuropathol Exp Neurol; 2012 Feb 01; 71(2):162-77. PubMed ID: 22249462
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  • 15. Over-expression of Hsp27 does not influence disease in the mutant SOD1(G93A) mouse model of amyotrophic lateral sclerosis.
    Krishnan J, Vannuvel K, Andries M, Waelkens E, Robberecht W, Van Den Bosch L.
    J Neurochem; 2008 Sep 01; 106(5):2170-83. PubMed ID: 18624915
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  • 19. A potential role for the p75 low-affinity neurotrophin receptor in spinal motor neuron degeneration in murine and human amyotrophic lateral sclerosis.
    Lowry KS, Murray SS, McLean CA, Talman P, Mathers S, Lopes EC, Cheema SS.
    Amyotroph Lateral Scler Other Motor Neuron Disord; 2001 Sep 01; 2(3):127-34. PubMed ID: 11771768
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