These tools will no longer be maintained as of December 31, 2024. Archived website can be found here. PubMed4Hh GitHub repository can be found here. Contact NLM Customer Service if you have questions.
Pubmed for Handhelds
PUBMED FOR HANDHELDS
Journal Abstract Search
926 related items for PubMed ID: 20056917
1. TIMP2 deficiency accelerates adverse post-myocardial infarction remodeling because of enhanced MT1-MMP activity despite lack of MMP2 activation. Kandalam V, Basu R, Abraham T, Wang X, Soloway PD, Jaworski DM, Oudit GY, Kassiri Z. Circ Res; 2010 Mar 05; 106(4):796-808. PubMed ID: 20056917 [Abstract] [Full Text] [Related]
2. Lack of tissue inhibitor of metalloproteinases 2 leads to exacerbated left ventricular dysfunction and adverse extracellular matrix remodeling in response to biomechanical stress. Kandalam V, Basu R, Moore L, Fan D, Wang X, Jaworski DM, Oudit GY, Kassiri Z. Circulation; 2011 Nov 08; 124(19):2094-105. PubMed ID: 21986284 [Abstract] [Full Text] [Related]
3. Myocardial overexpression of TIMP3 after myocardial infarction exerts beneficial effects by promoting angiogenesis and suppressing early proteolysis. Takawale A, Zhang P, Azad A, Wang W, Wang X, Murray AG, Kassiri Z. Am J Physiol Heart Circ Physiol; 2017 Aug 01; 313(2):H224-H236. PubMed ID: 28550172 [Abstract] [Full Text] [Related]
6. Expression of the tissue inhibitor of metalloproteinase-3 by transplanted VSMCs modifies heart structure and function after myocardial infarction. Jia ZB, Tian H, Kang K, Miao HZ, Liu KY, Jiang SL, Wang LP. Transpl Immunol; 2014 May 01; 30(4):149-58. PubMed ID: 24727088 [Abstract] [Full Text] [Related]
8. Early activation of matrix metalloproteinases underlies the exacerbated systolic and diastolic dysfunction in mice lacking TIMP3 following myocardial infarction. Kandalam V, Basu R, Abraham T, Wang X, Awad A, Wang W, Lopaschuk GD, Maeda N, Oudit GY, Kassiri Z. Am J Physiol Heart Circ Physiol; 2010 Oct 01; 299(4):H1012-23. PubMed ID: 20675565 [Abstract] [Full Text] [Related]
10. Heterogeneity in MT1-MMP activity with ischemia-reperfusion and previous myocardial infarction: relation to regional myocardial function. Dixon JA, Gaillard WF, Rivers WT, Koval CN, Stroud RE, Mukherjee R, Spinale FG. Am J Physiol Heart Circ Physiol; 2010 Dec 01; 299(6):H1947-58. PubMed ID: 20935147 [Abstract] [Full Text] [Related]
11. Myocardial recovery from ischemia-reperfusion is compromised in the absence of tissue inhibitor of metalloproteinase 4. Takawale A, Fan D, Basu R, Shen M, Parajuli N, Wang W, Wang X, Oudit GY, Kassiri Z. Circ Heart Fail; 2014 Jul 01; 7(4):652-62. PubMed ID: 24842912 [Abstract] [Full Text] [Related]
14. Age-related differences in postinfarct left ventricular rupture and remodeling. Yang Y, Ma Y, Han W, Li J, Xiang Y, Liu F, Ma X, Zhang J, Fu Z, Su YD, Du XJ, Gao XM. Am J Physiol Heart Circ Physiol; 2008 Apr 01; 294(4):H1815-22. PubMed ID: 18263717 [Abstract] [Full Text] [Related]
17. Inhibition of matrix metalloproteinases improves left ventricular function in mice lacking osteopontin after myocardial infarction. Krishnamurthy P, Peterson JT, Subramanian V, Singh M, Singh K. Mol Cell Biochem; 2009 Feb 01; 322(1-2):53-62. PubMed ID: 18979185 [Abstract] [Full Text] [Related]
18. Differential role of TIMP2 and TIMP3 in cardiac hypertrophy, fibrosis, and diastolic dysfunction. Fan D, Takawale A, Basu R, Patel V, Lee J, Kandalam V, Wang X, Oudit GY, Kassiri Z. Cardiovasc Res; 2014 Jul 15; 103(2):268-80. PubMed ID: 24692173 [Abstract] [Full Text] [Related]