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PUBMED FOR HANDHELDS

Journal Abstract Search


371 related items for PubMed ID: 20060491

  • 1. Spatiotemporal characterization of ionizing radiation induced DNA damage foci and their relation to chromatin organization.
    Costes SV, Chiolo I, Pluth JM, Barcellos-Hoff MH, Jakob B.
    Mutat Res; 2010; 704(1-3):78-87. PubMed ID: 20060491
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  • 2. Persistent DNA damage after high dose in vivo gamma exposure of minipig skin.
    Ahmed EA, Agay D, Schrock G, Drouet M, Meineke V, Scherthan H.
    PLoS One; 2012; 7(6):e39521. PubMed ID: 22761813
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  • 3. Clustered DNA damage induces pan-nuclear H2AX phosphorylation mediated by ATM and DNA-PK.
    Meyer B, Voss KO, Tobias F, Jakob B, Durante M, Taucher-Scholz G.
    Nucleic Acids Res; 2013 Jul; 41(12):6109-18. PubMed ID: 23620287
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  • 5. Growth of persistent foci of DNA damage checkpoint factors is essential for amplification of G1 checkpoint signaling.
    Yamauchi M, Oka Y, Yamamoto M, Niimura K, Uchida M, Kodama S, Watanabe M, Sekine I, Yamashita S, Suzuki K.
    DNA Repair (Amst); 2008 Mar 01; 7(3):405-17. PubMed ID: 18248856
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  • 8. Smad7 foci are present in micronuclei induced by heavy particle radiation.
    Wang M, Saha J, Cucinotta FA.
    Mutat Res; 2013 Aug 30; 756(1-2):108-14. PubMed ID: 23643526
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  • 10. Laser-induced radiation microbeam technology and simultaneous real-time fluorescence imaging in live cells.
    Botchway SW, Reynolds P, Parker AW, O'Neill P.
    Methods Enzymol; 2012 Aug 30; 504():3-28. PubMed ID: 22264527
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  • 11. The influence of heterochromatin on DNA double strand break repair: Getting the strong, silent type to relax.
    Goodarzi AA, Jeggo P, Lobrich M.
    DNA Repair (Amst); 2010 Dec 10; 9(12):1273-82. PubMed ID: 21036673
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  • 12. p53 binding protein 1 (53BP1) is an early participant in the cellular response to DNA double-strand breaks.
    Schultz LB, Chehab NH, Malikzay A, Halazonetis TD.
    J Cell Biol; 2000 Dec 25; 151(7):1381-90. PubMed ID: 11134068
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  • 16. Evidence for the direct binding of phosphorylated p53 to sites of DNA breaks in vivo.
    Al Rashid ST, Dellaire G, Cuddihy A, Jalali F, Vaid M, Coackley C, Folkard M, Xu Y, Chen BP, Chen DJ, Lilge L, Prise KM, Bazett Jones DP, Bristow RG.
    Cancer Res; 2005 Dec 01; 65(23):10810-21. PubMed ID: 16322227
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  • 17. S1219 residue of 53BP1 is phosphorylated by ATM kinase upon DNA damage and required for proper execution of DNA damage response.
    Lee H, Kwak HJ, Cho IT, Park SH, Lee CH.
    Biochem Biophys Res Commun; 2009 Jan 02; 378(1):32-6. PubMed ID: 18996087
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  • 18. Tumor suppressor p53 binding protein 1 (53BP1) is involved in DNA damage-signaling pathways.
    Rappold I, Iwabuchi K, Date T, Chen J.
    J Cell Biol; 2001 Apr 30; 153(3):613-20. PubMed ID: 11331310
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  • 19. In vivo formation of gamma-H2AX and 53BP1 DNA repair foci in blood cells after radioiodine therapy of differentiated thyroid cancer.
    Lassmann M, Hänscheid H, Gassen D, Biko J, Meineke V, Reiners C, Scherthan H.
    J Nucl Med; 2010 Aug 30; 51(8):1318-25. PubMed ID: 20660387
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