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Journal Abstract Search

367 related items for PubMed ID: 20184888

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  • 3. Hypophosphorylation of the Stiff N2B titin isoform raises cardiomyocyte resting tension in failing human myocardium.
    Borbély A, Falcao-Pires I, van Heerebeek L, Hamdani N, Edes I, Gavina C, Leite-Moreira AF, Bronzwaer JG, Papp Z, van der Velden J, Stienen GJ, Paulus WJ.
    Circ Res; 2009 Mar 27; 104(6):780-6. PubMed ID: 19179657
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  • 4. Protein kinase G modulates human myocardial passive stiffness by phosphorylation of the titin springs.
    Krüger M, Kötter S, Grützner A, Lang P, Andresen C, Redfield MM, Butt E, dos Remedios CG, Linke WA.
    Circ Res; 2009 Jan 02; 104(1):87-94. PubMed ID: 19023132
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  • 6. Deranged myofilament phosphorylation and function in experimental heart failure with preserved ejection fraction.
    Hamdani N, Bishu KG, von Frieling-Salewsky M, Redfield MM, Linke WA.
    Cardiovasc Res; 2013 Mar 01; 97(3):464-71. PubMed ID: 23213108
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  • 7. Hypothyroidism leads to increased collagen-based stiffness and re-expression of large cardiac titin isoforms with high compliance.
    Wu Y, Peng J, Campbell KB, Labeit S, Granzier H.
    J Mol Cell Cardiol; 2007 Jan 01; 42(1):186-95. PubMed ID: 17069849
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  • 10. Changes in titin and collagen underlie diastolic stiffness diversity of cardiac muscle.
    Wu Y, Cazorla O, Labeit D, Labeit S, Granzier H.
    J Mol Cell Cardiol; 2000 Dec 01; 32(12):2151-62. PubMed ID: 11112991
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  • 11. Developmental changes in passive stiffness and myofilament Ca2+ sensitivity due to titin and troponin-I isoform switching are not critically triggered by birth.
    Krüger M, Kohl T, Linke WA.
    Am J Physiol Heart Circ Physiol; 2006 Aug 01; 291(2):H496-506. PubMed ID: 16679402
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  • 12. Titin isoform switch in ischemic human heart disease.
    Neagoe C, Kulke M, del Monte F, Gwathmey JK, de Tombe PP, Hajjar RJ, Linke WA.
    Circulation; 2002 Sep 10; 106(11):1333-41. PubMed ID: 12221049
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  • 13. Protein kinase-A phosphorylates titin in human heart muscle and reduces myofibrillar passive tension.
    Krüger M, Linke WA.
    J Muscle Res Cell Motil; 2006 Sep 10; 27(5-7):435-44. PubMed ID: 16897574
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  • 14. Developmental control of titin isoform expression and passive stiffness in fetal and neonatal myocardium.
    Lahmers S, Wu Y, Call DR, Labeit S, Granzier H.
    Circ Res; 2004 Mar 05; 94(4):505-13. PubMed ID: 14707027
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  • 15. Role of inducible nitric oxide synthase in induction of RhoA expression in hearts from diabetic rats.
    Soliman H, Craig GP, Nagareddy P, Yuen VG, Lin G, Kumar U, McNeill JH, Macleod KM.
    Cardiovasc Res; 2008 Jul 15; 79(2):322-30. PubMed ID: 18411229
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  • 16. Titin-based regulations of diastolic and systolic functions of mammalian cardiac muscle.
    Fukuda N, Terui T, Ishiwata S, Kurihara S.
    J Mol Cell Cardiol; 2010 May 15; 48(5):876-81. PubMed ID: 19962382
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  • 17. Sense and stretchability: the role of titin and titin-associated proteins in myocardial stress-sensing and mechanical dysfunction.
    Linke WA.
    Cardiovasc Res; 2008 Mar 01; 77(4):637-48. PubMed ID: 17475230
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  • 18. Diabetes-Induced Cardiomyocyte Passive Stiffening Is Caused by Impaired Insulin-Dependent Titin Modification and Can Be Modulated by Neuregulin-1.
    Hopf AE, Andresen C, Kötter S, Isić M, Ulrich K, Sahin S, Bongardt S, Röll W, Drove F, Scheerer N, Vandekerckhove L, De Keulenaer GW, Hamdani N, Linke WA, Krüger M.
    Circ Res; 2018 Jul 20; 123(3):342-355. PubMed ID: 29760016
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  • 19. Differential changes in titin domain phosphorylation increase myofilament stiffness in failing human hearts.
    Kötter S, Gout L, Von Frieling-Salewsky M, Müller AE, Helling S, Marcus K, Dos Remedios C, Linke WA, Krüger M.
    Cardiovasc Res; 2013 Sep 01; 99(4):648-56. PubMed ID: 23764881
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  • 20. IGF-I attenuates diabetes-induced cardiac contractile dysfunction in ventricular myocytes.
    Norby FL, Wold LE, Duan J, Hintz KK, Ren J.
    Am J Physiol Endocrinol Metab; 2002 Oct 01; 283(4):E658-66. PubMed ID: 12217882
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