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Journal Abstract Search
264 related items for PubMed ID: 20228680
1. Toll-like receptor 2 plays a critical role in cardiac dysfunction during polymicrobial sepsis. Zou L, Feng Y, Chen YJ, Si R, Shen S, Zhou Q, Ichinose F, Scherrer-Crosbie M, Chao W. Crit Care Med; 2010 May; 38(5):1335-42. PubMed ID: 20228680 [Abstract] [Full Text] [Related]
2. MyD88 and Trif signaling play distinct roles in cardiac dysfunction and mortality during endotoxin shock and polymicrobial sepsis. Feng Y, Zou L, Zhang M, Li Y, Chen C, Chao W. Anesthesiology; 2011 Sep; 115(3):555-67. PubMed ID: 21792053 [Abstract] [Full Text] [Related]
3. Does polymicrobial sepsis exert cardiac dysfunction directly through toll-like receptor 2? Mersmann J, Zacharowski K. Crit Care Med; 2010 May; 38(5):1384-5. PubMed ID: 20404635 [No Abstract] [Full Text] [Related]
5. Nonhematopoietic toll-like receptor 2 contributes to neutrophil and cardiac function impairment during polymicrobial sepsis. Zou L, Feng Y, Zhang M, Li Y, Chao W. Shock; 2011 Oct; 36(4):370-80. PubMed ID: 21701420 [Abstract] [Full Text] [Related]
9. Importance of Toll-like receptor 2 in mitochondrial dysfunction during polymicrobial sepsis. Gong Y, Zou L, Feng Y, Li D, Cai J, Chen D, Chao W. Anesthesiology; 2014 Dec; 121(6):1236-47. PubMed ID: 25272245 [Abstract] [Full Text] [Related]
10. Modulation of tissue Toll-like receptor 2 and 4 during the early phases of polymicrobial sepsis correlates with mortality. Williams DL, Ha T, Li C, Kalbfleisch JH, Schweitzer J, Vogt W, Browder IW. Crit Care Med; 2003 Jun; 31(6):1808-18. PubMed ID: 12794424 [Abstract] [Full Text] [Related]
14. CCR9+ macrophages are required for eradication of peritoneal bacterial infections and prevention of polymicrobial sepsis. Mizukami T, Kanai T, Mikami Y, Hayashi A, Doi T, Handa T, Matsumoto A, Jun L, Matsuoka K, Sato T, Hisamatsu T, Hibi T. Immunol Lett; 2012 Sep; 147(1-2):75-9. PubMed ID: 22771342 [Abstract] [Full Text] [Related]
16. Scavenger receptor class a plays a central role in mediating mortality and the development of the pro-inflammatory phenotype in polymicrobial sepsis. Ozment TR, Ha T, Breuel KF, Ford TR, Ferguson DA, Kalbfleisch J, Schweitzer JB, Kelley JL, Li C, Williams DL. PLoS Pathog; 2012 Sep; 8(10):e1002967. PubMed ID: 23071440 [Abstract] [Full Text] [Related]