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Journal Abstract Search

161 related items for PubMed ID: 20298204

  • 21. Peptide and protein mimetics inhibiting amyloid beta-peptide aggregation.
    Takahashi T, Mihara H.
    Acc Chem Res; 2008 Oct; 41(10):1309-18. PubMed ID: 18937396
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  • 23. Molecular pathogenesis of protein misfolding diseases: pathological molecular environments versus quality control systems against misfolded proteins.
    Naiki H, Nagai Y.
    J Biochem; 2009 Dec; 146(6):751-6. PubMed ID: 19643812
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  • 24. [At last new light for the treatment of amyloidosis].
    Pettersson T.
    Duodecim; 2003 Dec; 119(12):1097-8. PubMed ID: 12868354
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  • 25. Targeted treatment for amyloidosis.
    Pepys-Vered ME, Pepys MB.
    Isr Med Assoc J; 2014 May; 16(5):277-80. PubMed ID: 24979830
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  • 29. Concentration of serum amyloid P component in the CSF as a possible marker of cerebral amyloid deposits in Alzheimer's disease.
    Hawkins PN, Rossor MN, Gallimore JR, Miller B, Moore EG, Pepys MB.
    Biochem Biophys Res Commun; 1994 Jun 15; 201(2):722-6. PubMed ID: 7516157
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  • 30. Selection of peptides binding to the amyloid b-protein reveals potential inhibitors of amyloid formation.
    Schwarzman AL, Tsiper M, Gregori L, Goldgaber D, Frakowiak J, Mazur-Kolecka B, Taraskina A, Pchelina S, Vitek MP.
    Amyloid; 2005 Dec 15; 12(4):199-209. PubMed ID: 16399644
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  • 34. Preferential association of serum amyloid P component with fibrillar deposits in familial British and Danish dementias: similarities with Alzheimer's disease.
    Rostagno A, Lashley T, Ng D, Meyerson J, Braendgaard H, Plant G, Bojsen-Møller M, Holton J, Frangione B, Revesz T, Ghiso J.
    J Neurol Sci; 2007 Jun 15; 257(1-2):88-96. PubMed ID: 17374542
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  • 39. Biophysical analyses of synthetic amyloid-beta(1-42) aggregates before and after covalent cross-linking. Implications for deducing the structure of endogenous amyloid-beta oligomers.
    Moore BD, Rangachari V, Tay WM, Milkovic NM, Rosenberry TL.
    Biochemistry; 2009 Dec 15; 48(49):11796-806. PubMed ID: 19916493
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