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Journal Abstract Search

298 related items for PubMed ID: 20302875

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  • 2. Catecholaminergic polymorphic ventricular tachycardia: recent mechanistic insights.
    Kontula K, Laitinen PJ, Lehtonen A, Toivonen L, Viitasalo M, Swan H.
    Cardiovasc Res; 2005 Aug 15; 67(3):379-87. PubMed ID: 15913575
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  • 4. Modest reductions of cardiac calsequestrin increase sarcoplasmic reticulum Ca2+ leak independent of luminal Ca2+ and trigger ventricular arrhythmias in mice.
    Chopra N, Kannankeril PJ, Yang T, Hlaing T, Holinstat I, Ettensohn K, Pfeifer K, Akin B, Jones LR, Franzini-Armstrong C, Knollmann BC.
    Circ Res; 2007 Sep 14; 101(6):617-26. PubMed ID: 17656677
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  • 6. Sarcoplasmic reticulum Ca2+ release in neonatal rat cardiac myocytes.
    Gergs U, Kirchhefer U, Buskase J, Kiele-Dunsche K, Buchwalow IB, Jones LR, Schmitz W, Traub O, Neumann J.
    J Mol Cell Cardiol; 2011 Nov 14; 51(5):682-8. PubMed ID: 21871897
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  • 7. Abnormal interactions of calsequestrin with the ryanodine receptor calcium release channel complex linked to exercise-induced sudden cardiac death.
    Terentyev D, Nori A, Santoro M, Viatchenko-Karpinski S, Kubalova Z, Gyorke I, Terentyeva R, Vedamoorthyrao S, Blom NA, Valle G, Napolitano C, Williams SC, Volpe P, Priori SG, Gyorke S.
    Circ Res; 2006 May 12; 98(9):1151-8. PubMed ID: 16601229
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  • 12. Ryanodine receptor mutations associated with stress-induced ventricular tachycardia mediate increased calcium release in stimulated cardiomyocytes.
    George CH, Higgs GV, Lai FA.
    Circ Res; 2003 Sep 19; 93(6):531-40. PubMed ID: 12919952
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  • 14. Modulation of ryanodine receptor by luminal calcium and accessory proteins in health and cardiac disease.
    Györke S, Terentyev D.
    Cardiovasc Res; 2008 Jan 15; 77(2):245-55. PubMed ID: 18006456
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  • 15. Catecholaminergic polymorphic ventricular tachycardia from bedside to bench and beyond.
    Katz G, Arad M, Eldar M.
    Curr Probl Cardiol; 2009 Jan 15; 34(1):9-43. PubMed ID: 19068246
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  • 16. Transgenic triadin 1 overexpression alters SR Ca2+ handling and leads to a blunted contractile response to beta-adrenergic agonists.
    Kirchhefer U, Jones LR, Begrow F, Boknik P, Hein L, Lohse MJ, Riemann B, Schmitz W, Stypmann J, Neumann J.
    Cardiovasc Res; 2004 Apr 01; 62(1):122-34. PubMed ID: 15023559
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  • 17. Effects of calsequestrin over-expression on excitation-contraction coupling in isolated rabbit cardiomyocytes.
    Miller SL, Currie S, Loughrey CM, Kettlewell S, Seidler T, Reynolds DF, Hasenfuss G, Smith GL.
    Cardiovasc Res; 2005 Sep 01; 67(4):667-77. PubMed ID: 15913577
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  • 18. Protein protein interactions between triadin and calsequestrin are involved in modulation of sarcoplasmic reticulum calcium release in cardiac myocytes.
    Terentyev D, Viatchenko-Karpinski S, Vedamoorthyrao S, Oduru S, Györke I, Williams SC, Györke S.
    J Physiol; 2007 Aug 15; 583(Pt 1):71-80. PubMed ID: 17569730
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  • 20. Overexpression of junctin causes adaptive changes in cardiac myocyte Ca(2+) signaling.
    Kirchhefer U, Hanske G, Jones LR, Justus I, Kaestner L, Lipp P, Schmitz W, Neumann J.
    Cell Calcium; 2006 Feb 15; 39(2):131-42. PubMed ID: 16289269
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