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230 related items for PubMed ID: 20335317

  • 1. Pathobiology of renal-specific oxidoreductase/myo-inositol oxygenase in diabetic nephropathy: its implications in tubulointerstitial fibrosis.
    Xie P, Sun L, Oates PJ, Srivastava SK, Kanwar YS.
    Am J Physiol Renal Physiol; 2010 Jun; 298(6):F1393-404. PubMed ID: 20335317
    [Abstract] [Full Text] [Related]

  • 2. myo-Inositol Oxygenase Overexpression Accentuates Generation of Reactive Oxygen Species and Exacerbates Cellular Injury following High Glucose Ambience: A NEW MECHANISM RELEVANT TO THE PATHOGENESIS OF DIABETIC NEPHROPATHY.
    Sun L, Dutta RK, Xie P, Kanwar YS.
    J Biol Chem; 2016 Mar 11; 291(11):5688-5707. PubMed ID: 26792859
    [Abstract] [Full Text] [Related]

  • 3. Disruption of renal tubular mitochondrial quality control by Myo-inositol oxygenase in diabetic kidney disease.
    Zhan M, Usman IM, Sun L, Kanwar YS.
    J Am Soc Nephrol; 2015 Jun 11; 26(6):1304-21. PubMed ID: 25270067
    [Abstract] [Full Text] [Related]

  • 4. Transcriptional and post-translational modulation of myo-inositol oxygenase by high glucose and related pathobiological stresses.
    Nayak B, Kondeti VK, Xie P, Lin S, Viswakarma N, Raparia K, Kanwar YS.
    J Biol Chem; 2011 Aug 05; 286(31):27594-611. PubMed ID: 21652700
    [Abstract] [Full Text] [Related]

  • 5. High Glucose-Induced Hypomethylation Promotes Binding of Sp-1 to Myo-Inositol Oxygenase: Implication in the Pathobiology of Diabetic Tubulopathy.
    Sharma I, Dutta RK, Singh NK, Kanwar YS.
    Am J Pathol; 2017 Apr 05; 187(4):724-739. PubMed ID: 28208054
    [Abstract] [Full Text] [Related]

  • 6. Transcriptional and Translational Modulation of myo-Inositol Oxygenase (Miox) by Fatty Acids: IMPLICATIONS IN RENAL TUBULAR INJURY INDUCED IN OBESITY AND DIABETES.
    Tominaga T, Dutta RK, Joladarashi D, Doi T, Reddy JK, Kanwar YS.
    J Biol Chem; 2016 Jan 15; 291(3):1348-67. PubMed ID: 26578517
    [Abstract] [Full Text] [Related]

  • 7. Contribution of myo-inositol oxygenase in AGE:RAGE-mediated renal tubulointerstitial injury in the context of diabetic nephropathy.
    Sharma I, Tupe RS, Wallner AK, Kanwar YS.
    Am J Physiol Renal Physiol; 2018 Jan 01; 314(1):F107-F121. PubMed ID: 28931523
    [Abstract] [Full Text] [Related]

  • 8. Increased expression of myo-inositol oxygenase is involved in the tubulointerstitial injury of diabetic nephropathy.
    Lu Y, Liu C, Miao X, Xu K, Wu X, Liu C.
    Exp Clin Endocrinol Diabetes; 2009 Jun 01; 117(6):257-65. PubMed ID: 19053028
    [Abstract] [Full Text] [Related]

  • 9. Myo-inositol Oxygenase (MIOX) Overexpression Drives the Progression of Renal Tubulointerstitial Injury in Diabetes.
    Sharma I, Deng F, Liao Y, Kanwar YS.
    Diabetes; 2020 Jun 01; 69(6):1248-1263. PubMed ID: 32169892
    [Abstract] [Full Text] [Related]

  • 10. Myo-inositol oxygenase accentuates renal tubular injury initiated by endoplasmic reticulum stress.
    Tominaga T, Sharma I, Fujita Y, Doi T, Wallner AK, Kanwar YS.
    Am J Physiol Renal Physiol; 2019 Feb 01; 316(2):F301-F315. PubMed ID: 30539651
    [Abstract] [Full Text] [Related]

  • 11. Modulation of renal-specific oxidoreductase/myo-inositol oxygenase by high-glucose ambience.
    Nayak B, Xie P, Akagi S, Yang Q, Sun L, Wada J, Thakur A, Danesh FR, Chugh SS, Kanwar YS.
    Proc Natl Acad Sci U S A; 2005 Dec 13; 102(50):17952-7. PubMed ID: 16330753
    [Abstract] [Full Text] [Related]

  • 12. Renal-specific oxidoreductase biphasic expression under high glucose ambience during fetal versus neonatal development.
    Kanwar YS, Akagi S, Nayak B, Sun L, Wada J, Xie P, Thakur A, Chugh SS, Danesh FR.
    Kidney Int; 2005 Oct 13; 68(4):1670-83. PubMed ID: 16164644
    [Abstract] [Full Text] [Related]

  • 13. Gene regulation of aldose-, aldehyde- and a renal specific oxido reductase (RSOR) in the pathobiology of diabetes mellitus.
    Danesh FR, Wada J, Wallner EI, Sahai A, Srivastava SK, Kanwar YS.
    Curr Med Chem; 2003 Aug 13; 10(15):1399-406. PubMed ID: 12871137
    [Abstract] [Full Text] [Related]

  • 14. Myo-inositol oxygenase overexpression exacerbates cadmium-induced kidney injury via oxidant stress and necroptosis.
    Zheng X, Deng F, Sharma I, Kanwar YS.
    Am J Physiol Renal Physiol; 2022 Mar 01; 322(3):F344-F359. PubMed ID: 35100813
    [Abstract] [Full Text] [Related]

  • 15. Up-regulation of human myo-inositol oxygenase by hyperosmotic stress in renal proximal tubular epithelial cells.
    Prabhu KS, Arner RJ, Vunta H, Reddy CC.
    J Biol Chem; 2005 May 20; 280(20):19895-901. PubMed ID: 15778219
    [Abstract] [Full Text] [Related]

  • 16. Relevance of renal-specific oxidoreductase in tubulogenesis during mammalian nephron development.
    Kanwar YS, Yang Q, Tian Y, Lin S, Wada J, Chugh S, Srivastava SK.
    Am J Physiol Renal Physiol; 2002 Apr 20; 282(4):F752-62. PubMed ID: 11880337
    [Abstract] [Full Text] [Related]

  • 17. Beneficial Effects of Myo-Inositol Oxygenase Deficiency in Cisplatin-Induced AKI.
    Dutta RK, Kondeti VK, Sharma I, Chandel NS, Quaggin SE, Kanwar YS.
    J Am Soc Nephrol; 2017 May 20; 28(5):1421-1436. PubMed ID: 27895157
    [Abstract] [Full Text] [Related]

  • 18. Renal depletion of myo-inositol is associated with its increased degradation in animal models of metabolic disease.
    Chang HH, Chao HN, Walker CS, Choong SY, Phillips A, Loomes KM.
    Am J Physiol Renal Physiol; 2015 Nov 01; 309(9):F755-63. PubMed ID: 26311112
    [Abstract] [Full Text] [Related]

  • 19. WJ-39, an Aldose Reductase Inhibitor, Ameliorates Renal Lesions in Diabetic Nephropathy by Activating Nrf2 Signaling.
    Zhou X, Liu Z, Ying K, Wang H, Liu P, Ji X, Chi T, Zou L, Wang S, He Z.
    Oxid Med Cell Longev; 2020 Nov 01; 2020():7950457. PubMed ID: 32566101
    [Abstract] [Full Text] [Related]

  • 20. Transforming growth factor-β1-mediated renal fibrosis is dependent on the regulation of transforming growth factor receptor 1 expression by let-7b.
    Wang B, Jha JC, Hagiwara S, McClelland AD, Jandeleit-Dahm K, Thomas MC, Cooper ME, Kantharidis P.
    Kidney Int; 2014 Feb 01; 85(2):352-61. PubMed ID: 24088962
    [Abstract] [Full Text] [Related]

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