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204 related items for PubMed ID: 20420578
1. Dominant-negative VDAC1 mutants reveal oligomeric VDAC1 to be the active unit in mitochondria-mediated apoptosis. Mader A, Abu-Hamad S, Arbel N, Gutiérrez-Aguilar M, Shoshan-Barmatz V. Biochem J; 2010 Jul 01; 429(1):147-55. PubMed ID: 20420578 [Abstract] [Full Text] [Related]
2. Mapping the ruthenium red-binding site of the voltage-dependent anion channel-1. Israelson A, Zaid H, Abu-Hamad S, Nahon E, Shoshan-Barmatz V. Cell Calcium; 2008 Feb 01; 43(2):196-204. PubMed ID: 17590433 [Abstract] [Full Text] [Related]
3. The voltage-dependent anion channel-1 modulates apoptotic cell death. Zaid H, Abu-Hamad S, Israelson A, Nathan I, Shoshan-Barmatz V. Cell Death Differ; 2005 Jul 01; 12(7):751-60. PubMed ID: 15818409 [Abstract] [Full Text] [Related]
4. VDAC1 cysteine residues: topology and function in channel activity and apoptosis. Aram L, Geula S, Arbel N, Shoshan-Barmatz V. Biochem J; 2010 Apr 14; 427(3):445-54. PubMed ID: 20192921 [Abstract] [Full Text] [Related]
5. Localization of the voltage-dependent anion channel-1 Ca2+-binding sites. Israelson A, Abu-Hamad S, Zaid H, Nahon E, Shoshan-Barmatz V. Cell Calcium; 2007 Mar 14; 41(3):235-44. PubMed ID: 16930689 [Abstract] [Full Text] [Related]
6. The VDAC1 N-terminus is essential both for apoptosis and the protective effect of anti-apoptotic proteins. Abu-Hamad S, Arbel N, Calo D, Arzoine L, Israelson A, Keinan N, Ben-Romano R, Friedman O, Shoshan-Barmatz V. J Cell Sci; 2009 Jun 01; 122(Pt 11):1906-16. PubMed ID: 19461077 [Abstract] [Full Text] [Related]
7. Apoptosis is regulated by the VDAC1 N-terminal region and by VDAC oligomerization: release of cytochrome c, AIF and Smac/Diablo. Shoshan-Barmatz V, Keinan N, Abu-Hamad S, Tyomkin D, Aram L. Biochim Biophys Acta; 2010 Jun 01; 1797(6-7):1281-91. PubMed ID: 20214874 [Abstract] [Full Text] [Related]
8. The mitochondrial voltage-dependent anion channel 1 in tumor cells. Shoshan-Barmatz V, Ben-Hail D, Admoni L, Krelin Y, Tripathi SS. Biochim Biophys Acta; 2015 Oct 01; 1848(10 Pt B):2547-75. PubMed ID: 25448878 [Abstract] [Full Text] [Related]
10. VDAC1, having a shorter N-terminus than VDAC2 but showing the same migration in an SDS-polyacrylamide gel, is the predominant form expressed in mitochondria of various tissues. Yamamoto T, Yamada A, Watanabe M, Yoshimura Y, Yamazaki N, Yoshimura Y, Yamauchi T, Kataoka M, Nagata T, Terada H, Shinohara Y. J Proteome Res; 2006 Dec 25; 5(12):3336-44. PubMed ID: 17137335 [Abstract] [Full Text] [Related]
11. In self-defence: hexokinase promotes voltage-dependent anion channel closure and prevents mitochondria-mediated apoptotic cell death. Azoulay-Zohar H, Israelson A, Abu-Hamad S, Shoshan-Barmatz V. Biochem J; 2004 Jan 15; 377(Pt 2):347-55. PubMed ID: 14561215 [Abstract] [Full Text] [Related]
12. A soluble form of the pilus protein FimA targets the VDAC-hexokinase complex at mitochondria to suppress host cell apoptosis. Sukumaran SK, Fu NY, Tin CB, Wan KF, Lee SS, Yu VC. Mol Cell; 2010 Mar 26; 37(6):768-83. PubMed ID: 20347420 [Abstract] [Full Text] [Related]
13. Hexokinase-I protection against apoptotic cell death is mediated via interaction with the voltage-dependent anion channel-1: mapping the site of binding. Abu-Hamad S, Zaid H, Israelson A, Nahon E, Shoshan-Barmatz V. J Biol Chem; 2008 May 09; 283(19):13482-90. PubMed ID: 18308720 [Abstract] [Full Text] [Related]
14. Oligomerization of the mitochondrial protein VDAC1: from structure to function and cancer therapy. Shoshan-Barmatz V, Mizrachi D, Keinan N. Prog Mol Biol Transl Sci; 2013 May 09; 117():303-34. PubMed ID: 23663973 [Abstract] [Full Text] [Related]
15. The expression level of the voltage-dependent anion channel controls life and death of the cell. Abu-Hamad S, Sivan S, Shoshan-Barmatz V. Proc Natl Acad Sci U S A; 2006 Apr 11; 103(15):5787-92. PubMed ID: 16585511 [Abstract] [Full Text] [Related]
16. VDAC1-interacting anion transport inhibitors inhibit VDAC1 oligomerization and apoptosis. Ben-Hail D, Shoshan-Barmatz V. Biochim Biophys Acta; 2016 Jul 11; 1863(7 Pt A):1612-23. PubMed ID: 27064145 [Abstract] [Full Text] [Related]
17. Outer membrane VDAC1 controls permeability transition of the inner mitochondrial membrane in cellulo during stress-induced apoptosis. Tomasello F, Messina A, Lartigue L, Schembri L, Medina C, Reina S, Thoraval D, Crouzet M, Ichas F, De Pinto V, De Giorgi F. Cell Res; 2009 Dec 11; 19(12):1363-76. PubMed ID: 19668262 [Abstract] [Full Text] [Related]
18. An N-terminal nucleotide-binding site in VDAC1: involvement in regulating mitochondrial function. Yehezkel G, Abu-Hamad S, Shoshan-Barmatz V. J Cell Physiol; 2007 Aug 11; 212(2):551-61. PubMed ID: 17503466 [Abstract] [Full Text] [Related]
19. Novel Compounds Targeting the Mitochondrial Protein VDAC1 Inhibit Apoptosis and Protect against Mitochondrial Dysfunction. Ben-Hail D, Begas-Shvartz R, Shalev M, Shteinfer-Kuzmine A, Gruzman A, Reina S, De Pinto V, Shoshan-Barmatz V. J Biol Chem; 2016 Nov 25; 291(48):24986-25003. PubMed ID: 27738100 [Abstract] [Full Text] [Related]
20. VDAC1 as a Player in Mitochondria-Mediated Apoptosis and Target for Modulating Apoptosis. Shoshan-Barmatz V, Krelin Y, Chen Q. Curr Med Chem; 2017 Nov 25; 24(40):4435-4446. PubMed ID: 28618997 [Abstract] [Full Text] [Related] Page: [Next] [New Search]