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PUBMED FOR HANDHELDS

Journal Abstract Search


148 related items for PubMed ID: 20460998

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  • 4. Bradykinin prevents reperfusion injury by targeting mitochondrial permeability transition pore through glycogen synthase kinase 3beta.
    Park SS, Zhao H, Mueller RA, Xu Z.
    J Mol Cell Cardiol; 2006 May; 40(5):708-16. PubMed ID: 16516918
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  • 5. Signaling pathways involved in desflurane-induced postconditioning in human atrial myocardium in vitro.
    Lemoine S, Beauchef G, Zhu L, Renard E, Lepage O, Massetti M, Khayat A, Galera P, Gérard JL, Hanouz JL.
    Anesthesiology; 2008 Dec; 109(6):1036-44. PubMed ID: 19034100
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  • 9. Reactive oxygen species mediate sevoflurane- and desflurane-induced preconditioning in isolated human right atria in vitro.
    Hanouz JL, Zhu L, Lemoine S, Durand C, Lepage O, Massetti M, Khayat A, Plaud B, Gérard JL.
    Anesth Analg; 2007 Dec; 105(6):1534-9, table of contents. PubMed ID: 18042844
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  • 10. Sevoflurane confers additive cardioprotection to ethanol preconditioning associated with enhanced phosphorylation of glycogen synthase kinase-3β and inhibition of mitochondrial permeability transition pore opening.
    Onishi A, Miyamae M, Inoue H, Kaneda K, Okusa C, Inamura Y, Shiomi M, Koshinuma S, Momota Y, Figueredo VM.
    J Cardiothorac Vasc Anesth; 2013 Oct; 27(5):916-24. PubMed ID: 23266287
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  • 12. Aprotinin abolishes sevoflurane postconditioning by inhibiting nitric oxide production and phosphorylation of protein kinase C-delta and glycogen synthase kinase 3beta.
    Inamura Y, Miyamae M, Sugioka S, Kaneda K, Okusa C, Onishi A, Domae N, Kotani J, Figueredo VM.
    Anesthesiology; 2009 Nov; 111(5):1036-43. PubMed ID: 19809279
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  • 14. Effect of desflurane-induced preconditioning following ischemia-reperfusion on nitric oxide release in rabbits.
    Tsai SK, Lin SM, Huang CH, Hung WC, Chih CL, Huang SS.
    Life Sci; 2004 Dec 24; 76(6):651-60. PubMed ID: 15567190
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  • 15. Inhibition of phosphodiesterases leads to prevention of the mitochondrial permeability transition pore opening and reperfusion injury in cardiac H9c2 cells.
    Chanoit G, Zhou J, Lee S, McIntosh R, Shen X, Zvara DA, Xu Z.
    Cardiovasc Drugs Ther; 2011 Aug 24; 25(4):299-306. PubMed ID: 21643720
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  • 16. [D-Ala2,D-Leu5]-enkephalin (DADLE) and morphine-induced postconditioning by inhibition of mitochondrial permeability transition pore, in human myocardium.
    Fuardo M, Lemoine S, Lo Coco C, Hanouz JL, Massetti M.
    Exp Biol Med (Maywood); 2013 Apr 24; 238(4):426-32. PubMed ID: 23436882
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  • 19. NECA at reperfusion limits infarction and inhibits formation of the mitochondrial permeability transition pore by activating p70S6 kinase.
    Förster K, Paul I, Solenkova N, Staudt A, Cohen MV, Downey JM, Felix SB, Krieg T.
    Basic Res Cardiol; 2006 Jul 24; 101(4):319-26. PubMed ID: 16604438
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  • 20. Effect of pressure overload on cardioprotection of mitochondrial KATP channels and GSK-3beta: interaction with the MPT pore.
    Mozaffari MS, Schaffer SW.
    Am J Hypertens; 2008 May 24; 21(5):570-5. PubMed ID: 18437149
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