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197 related items for PubMed ID: 2046673

  • 1. Depletion of c-myc with specific antisense sequences reverses the transformed phenotype in ras oncogene-transformed NIH 3T3 cells.
    Sklar MD, Thompson E, Welsh MJ, Liebert M, Harney J, Grossman HB, Smith M, Prochownik EV.
    Mol Cell Biol; 1991 Jul; 11(7):3699-710. PubMed ID: 2046673
    [Abstract] [Full Text] [Related]

  • 2. Increased cell-substrate adhesion accompanies conditional reversion to the normal phenotype in ras-oncogene-transformed NIH-3T3 cells.
    Shumaker DK, Sklar MD, Prochownik EV, Varani J.
    Exp Cell Res; 1994 Oct; 214(2):440-6. PubMed ID: 7925639
    [Abstract] [Full Text] [Related]

  • 3. Permanent conversion of mouse and human cells transformed by activated ras or raf genes to apparently normal cells by treatment with the antibiotic azatyrosine.
    Shindo-Okada N, Makabe O, Nagahara H, Nishimura S.
    Mol Carcinog; 1989 Oct; 2(3):159-67. PubMed ID: 2675904
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  • 4. Antisense-fos RNA causes partial reversion of the transformed phenotypes induced by the c-Ha-ras oncogene.
    Ledwith BJ, Manam S, Kraynak AR, Nichols WW, Bradley MO.
    Mol Cell Biol; 1990 Apr; 10(4):1545-55. PubMed ID: 1690847
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  • 8. Differential dependence of the tumorigenicity of chemically transformed rat liver epithelial cells on autocrine production of transforming growth factor alpha.
    Duddy SK, Earp HS, Russell WE, Smith GJ, Grisham JW.
    Cell Growth Differ; 1995 Mar; 6(3):251-61. PubMed ID: 7794793
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  • 9. Partial transformation of mouse fibroblastic and epithelial cell lines with the v-myc oncogene.
    Falcone G, Summerhayes IC, Paterson H, Marshall CJ, Hall A.
    Exp Cell Res; 1987 Jan; 168(1):273-84. PubMed ID: 3023119
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  • 10. Decreased tumorigenicity of c-Myc-transformed fibroblasts expressing active USF2.
    Choe C, Chen N, Sawadogo M.
    Exp Cell Res; 2005 Jan 01; 302(1):1-10. PubMed ID: 15541720
    [Abstract] [Full Text] [Related]

  • 11. Specific viral oncogenes cause differential effects on cell-to-cell communication, relevant to the suppression of the transformed phenotype by normal cells.
    Bignami M, Rosa S, Falcone G, Tató F, Katoh F, Yamasaki H.
    Mol Carcinog; 1988 Jan 01; 1(1):67-75. PubMed ID: 2475135
    [Abstract] [Full Text] [Related]

  • 12. Antisense DNA inhibition of tumor growth induced by c-Ha-ras oncogene in nude mice.
    Gray GD, Hernandez OM, Hebel D, Root M, Pow-Sang JM, Wickstrom E.
    Cancer Res; 1993 Feb 01; 53(3):577-80. PubMed ID: 8425190
    [Abstract] [Full Text] [Related]

  • 13. Detection of genes with a potential for suppressing the transformed phenotype associated with activated ras genes.
    Noda M, Kitayama H, Matsuzaki T, Sugimoto Y, Okayama H, Bassin RH, Ikawa Y.
    Proc Natl Acad Sci U S A; 1989 Jan 01; 86(1):162-6. PubMed ID: 2463620
    [Abstract] [Full Text] [Related]

  • 14. Detection and cloning of potent transforming gene(s) from chewing tobacco-related human oral carcinomas.
    Saranath D, Bhoite LT, Deo MG, Tandle AT, D'Costa J, Kolhapure RN, Govardhan MK, Banerjee K.
    Eur J Cancer B Oral Oncol; 1994 Jul 01; 30B(4):268-77. PubMed ID: 7950842
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  • 15. Transfection of EK-3, a subline of NIH 3T3, with the oncogene Ha-ras does not abolish its anchorage dependence.
    Katz E, Samid D.
    Eur J Cell Biol; 1989 Aug 01; 49(2):221-4. PubMed ID: 2673787
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  • 16. Introduction of the activated N-ras oncogene into human fibroblasts by retroviral vector induces morphological transformation and tumorigenicity.
    Kinsella AR, Fiszer-Maliszewska L, Mitchell EL, Guo YP, Fox M, Scott D.
    Carcinogenesis; 1990 Oct 01; 11(10):1803-9. PubMed ID: 2208593
    [Abstract] [Full Text] [Related]

  • 17. Resistance to oncogenic transformation in revertant R1 of human ras-transformed NIH 3T3 cells.
    Kuzumaki N, Ogiso Y, Oda A, Fujita H, Suzuki H, Sato C, Müllauer L.
    Mol Cell Biol; 1989 May 01; 9(5):2258-63. PubMed ID: 2664473
    [Abstract] [Full Text] [Related]

  • 18. Oncogene transformation frequency of nonsenescent SFME cells is increased by c-myc.
    Rawson C, Shirahata S, Collodi P, Natsuno T, Barnes D.
    Oncogene; 1991 Mar 01; 6(3):487-9. PubMed ID: 2011405
    [Abstract] [Full Text] [Related]

  • 19. Thy-1 as a negative growth regulator in ras-transformed mouse fibroblasts.
    Sugimoto Y, Ikawa Y, Nakauchi H.
    Cancer Res; 1991 Jan 01; 51(1):99-104. PubMed ID: 1670996
    [Abstract] [Full Text] [Related]

  • 20. A modest reduction in c-myc expression has minimal effects on cell growth and apoptosis but dramatically reduces susceptibility to Ras and Raf transformation.
    Bazarov AV, Adachi S, Li SF, Mateyak MK, Wei S, Sedivy JM.
    Cancer Res; 2001 Feb 01; 61(3):1178-86. PubMed ID: 11221849
    [Abstract] [Full Text] [Related]

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