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335 related items for PubMed ID: 2049075

  • 1. Phorbol ester and bryostatin differentially regulate the hydrolysis of phosphatidylethanolamine in Ha-ras- and raf-oncogene-transformed NIH 3T3 cells.
    Kiss Z, Rapp UR, Pettit GR, Anderson WB.
    Biochem J; 1991 Jun 01; 276 ( Pt 2)(Pt 2):505-9. PubMed ID: 2049075
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  • 3. Regulation of phospholipase D by sphingosine involves both protein kinase C-dependent and -independent mechanisms in NIH 3T3 fibroblasts.
    Kiss Z, Deli E.
    Biochem J; 1992 Dec 15; 288 ( Pt 3)(Pt 3):853-8. PubMed ID: 1472000
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  • 4. Involvement of phospholipase D and protein kinase C in phorbol ester and fatty acid stimulated turnover of phosphatidylcholine and phosphatidylethanolamine in neural cells.
    Cook HW, Ridgway ND, Byers DM.
    Biochim Biophys Acta; 1998 Feb 05; 1390(1):103-17. PubMed ID: 9487144
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  • 5. Stimulation of phosphatidylcholine synthesis by activators of protein kinase C is dissociable from increased phospholipid hydrolysis.
    Kiss Z, Chattopadhyay J, Pettit GR.
    Biochem J; 1991 Jan 01; 273(Pt 1)(Pt 1):189-94. PubMed ID: 1989580
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  • 6. Cooperative effects of ethanol and protein kinase C activators on phospholipase-D-mediated hydrolysis of phosphatidylethanolamine in NIH 3T3 fibroblasts.
    Kiss Z.
    Biochim Biophys Acta; 1992 Dec 15; 1175(1):88-94. PubMed ID: 1482699
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  • 7. Protein kinase Calpha is a major mediator of the stimulatory effect of phorbol ester on phospholipase D-mediated hydrolysis of phosphatidylethanolamine.
    Mukherjee JJ, Chung T, Ways DK, Kiss Z.
    J Biol Chem; 1996 Nov 15; 271(46):28912-7. PubMed ID: 8910539
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  • 9. ATP stimulates the hydrolysis of phosphatidylethanolamine in NIH 3T3 cells. Potentiating effects of guanosine triphosphates and sphingosine.
    Kiss Z, Anderson WB.
    J Biol Chem; 1990 May 05; 265(13):7345-50. PubMed ID: 2185245
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  • 12. Vitamin K3 preferentially inhibits stimulation of phospholipase D-mediated hydrolysis of phosphatidylethanolamine by protein kinase C activators in NIH 3T3 fibroblasts.
    Tomono M, Kiss Z.
    Arch Biochem Biophys; 1994 Oct 05; 314(1):217-23. PubMed ID: 7944397
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  • 14. Raf revertant cells resist transformation by non-nuclear oncogenes and are deficient in the induction of early response genes by TPA and serum.
    Kolch W, Heidecker G, Troppmair J, Yanagihara K, Bassin RH, Rapp UR.
    Oncogene; 1993 Feb 05; 8(2):361-70. PubMed ID: 8426742
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  • 17. Ethanol potentiates the stimulatory effects of phorbol ester, sphingosine and 4-hydroxynonenal on the hydrolysis of phosphatidylethanolamine in NIH 3T3 cells.
    Kiss Z, Crilly K, Chattopadhyay J.
    Eur J Biochem; 1991 May 08; 197(3):785-90. PubMed ID: 2029907
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  • 19. Phorbol ester treatment of intact rabbit platelets greatly enhances both the basal and guanosine 5'-[gamma-thio]triphosphate-stimulated phospholipase D activities of isolated platelet membranes. Physiological activation of phospholipase D may be secondary to activation of phospholipase C.
    Van der Meulen J, Haslam RJ.
    Biochem J; 1990 Nov 01; 271(3):693-700. PubMed ID: 2123096
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  • 20. Posttranslational regulation of Lck and a p36-38 protein by activators of protein kinase C: differential effects of the tumor promoter, PMA, and the non-tumor-promoter, bryostatin.
    Galron D, Ansotegui IJ, Isakov N.
    Cell Immunol; 1997 Jun 15; 178(2):141-51. PubMed ID: 9225005
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