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446 related items for PubMed ID: 20501641

  • 1. Disruption of AMPKalpha1 signaling prevents AICAR-induced inhibition of AS160/TBC1D4 phosphorylation and glucose uptake in primary rat adipocytes.
    Gaidhu MP, Perry RL, Noor F, Ceddia RB.
    Mol Endocrinol; 2010 Jul; 24(7):1434-40. PubMed ID: 20501641
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  • 2. Prior treatment with the AMPK activator AICAR induces subsequently enhanced glucose uptake in isolated skeletal muscles from 24-month-old rats.
    Oki K, Arias EB, Kanzaki M, Cartee GD.
    Appl Physiol Nutr Metab; 2018 Aug; 43(8):795-805. PubMed ID: 29518344
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  • 4. Discovery of TBC1D1 as an insulin-, AICAR-, and contraction-stimulated signaling nexus in mouse skeletal muscle.
    Taylor EB, An D, Kramer HF, Yu H, Fujii NL, Roeckl KS, Bowles N, Hirshman MF, Xie J, Feener EP, Goodyear LJ.
    J Biol Chem; 2008 Apr 11; 283(15):9787-96. PubMed ID: 18276596
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  • 5. 5-Aminoimidazole-4-carboxamide-1-beta-D-ribofuranoside-induced AMP-activated protein kinase phosphorylation inhibits basal and insulin-stimulated glucose uptake, lipid synthesis, and fatty acid oxidation in isolated rat adipocytes.
    Gaidhu MP, Fediuc S, Ceddia RB.
    J Biol Chem; 2006 Sep 08; 281(36):25956-64. PubMed ID: 16816404
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  • 7. Stimulation of glucose transport in response to activation of distinct AMPK signaling pathways.
    Jing M, Cheruvu VK, Ismail-Beigi F.
    Am J Physiol Cell Physiol; 2008 Nov 08; 295(5):C1071-82. PubMed ID: 18701654
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  • 10. A Tbc1d1 Ser231Ala-knockin mutation partially impairs AICAR- but not exercise-induced muscle glucose uptake in mice.
    Chen Q, Xie B, Zhu S, Rong P, Sheng Y, Ducommun S, Chen L, Quan C, Li M, Sakamoto K, MacKintosh C, Chen S, Wang HY.
    Diabetologia; 2017 Feb 08; 60(2):336-345. PubMed ID: 27826658
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  • 15. Activation of phosphatidylinositol-3 kinase, AMP-activated kinase and Akt substrate-160 kDa by trans-10, cis-12 conjugated linoleic acid mediates skeletal muscle glucose uptake.
    Mohankumar SK, Taylor CG, Siemens L, Zahradka P.
    J Nutr Biochem; 2013 Feb 08; 24(2):445-56. PubMed ID: 22704782
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  • 16. Thr649Ala-AS160 knock-in mutation does not impair contraction/AICAR-induced glucose transport in mouse muscle.
    Ducommun S, Wang HY, Sakamoto K, MacKintosh C, Chen S.
    Am J Physiol Endocrinol Metab; 2012 May 15; 302(9):E1036-43. PubMed ID: 22318952
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  • 17. “Deletion of both Rab-GTPase–activating proteins TBC1D1 and TBC1D4 in mice eliminates insulin- and AICAR-stimulated glucose transport [corrected].
    Chadt A, Immisch A, de Wendt C, Springer C, Zhou Z, Stermann T, Holman GD, Loffing-Cueni D, Loffing J, Joost HG, Al-Hasani H.
    Diabetes; 2015 Mar 15; 64(3):746-59. PubMed ID: 25249576
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  • 18. Restoring AS160 phosphorylation rescues skeletal muscle insulin resistance and fatty acid oxidation while not reducing intramuscular lipids.
    Alkhateeb H, Chabowski A, Glatz JF, Gurd B, Luiken JJ, Bonen A.
    Am J Physiol Endocrinol Metab; 2009 Nov 15; 297(5):E1056-66. PubMed ID: 19724017
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  • 19. Activation of AMPK alleviates cardiopulmonary bypass-induced cardiac injury via ameliorating acute cardiac glucose metabolic disorder.
    Wang Z, Cao Y, Yin Q, Han Y, Wang Y, Sun G, Zhu H, Xu M, Gu C.
    Cardiovasc Ther; 2018 Dec 15; 36(6):e12482. PubMed ID: 30632675
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