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Journal Abstract Search

249 related items for PubMed ID: 20506216

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  • 2. Global T cell dysregulation in non-autoimmune-prone mice promotes rapid development of BAFF-independent, systemic lupus erythematosus-like autoimmunity.
    Stohl W, Jacob N, Quinn WJ, Cancro MP, Gao H, Putterman C, Gao X, Pricop L, Koss MN.
    J Immunol; 2008 Jul 01; 181(1):833-41. PubMed ID: 18566449
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  • 3. BAFF overexpression and accelerated glomerular disease in mice with an incomplete genetic predisposition to systemic lupus erythematosus.
    Stohl W, Xu D, Kim KS, Koss MN, Jorgensen TN, Deocharan B, Metzger TE, Bixler SA, Hong YS, Ambrose CM, Mackay F, Morel L, Putterman C, Kotzin BL, Kalled SL.
    Arthritis Rheum; 2005 Jul 01; 52(7):2080-91. PubMed ID: 15986357
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  • 7. Dispensability of APRIL to the development of systemic lupus erythematosus in NZM 2328 mice.
    Jacob CO, Guo S, Jacob N, Pawar RD, Putterman C, Quinn WJ, Cancro MP, Migone TS, Stohl W.
    Arthritis Rheum; 2012 May 01; 64(5):1610-9. PubMed ID: 22127792
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  • 8. Altered BAFF-receptor signaling and additional modifier loci contribute to systemic autoimmunity in A/WySnJ mice.
    Mayne CG, Nashold FE, Sasaki Y, Hayes CE.
    Eur J Immunol; 2009 Feb 01; 39(2):589-99. PubMed ID: 19152335
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  • 10. Development of Murine Systemic Lupus Erythematosus in the Absence of BAFF.
    Stohl W, Yu N, Chalmers S, Putterman C, Jacob CO.
    Arthritis Rheumatol; 2020 Feb 01; 72(2):292-302. PubMed ID: 31493335
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  • 11. Differential Development of Systemic Lupus Erythematosus in NZM 2328 Mice Deficient in Discrete Pairs of BAFF Receptors.
    Jacob CO, Yu N, Sindhava V, Cancro MP, Pawar RD, Putterman C, Stohl W.
    Arthritis Rheumatol; 2015 Sep 01; 67(9):2523-35. PubMed ID: 25989238
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  • 15. B cell-activating factor (BAFF) from dendritic cells, monocytes and neutrophils is required for B cell maturation and autoantibody production in SLE-like autoimmune disease.
    Giordano D, Kuley R, Draves KE, Elkon KB, Giltiay NV, Clark EA.
    Front Immunol; 2023 Sep 01; 14():1050528. PubMed ID: 36923413
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  • 16. Deleting the BAFF receptor TACI protects against systemic lupus erythematosus without extensive reduction of B cell numbers.
    Figgett WA, Deliyanti D, Fairfax KA, Quah PS, Wilkinson-Berka JL, Mackay F.
    J Autoimmun; 2015 Jul 01; 61():9-16. PubMed ID: 26027434
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  • 17. TACI deletion protects against progressive murine lupus nephritis induced by BAFF overexpression.
    Arkatkar T, Jacobs HM, Du SW, Li QZ, Hudkins KL, Alpers CE, Rawlings DJ, Jackson SW.
    Kidney Int; 2018 Oct 01; 94(4):728-740. PubMed ID: 29907458
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  • 18. CD19 hyperexpression augments Sle1-induced humoral autoimmunity but not clinical nephritis.
    Shi X, Xie C, Chang S, Zhou XJ, Tedder T, Mohan C.
    Arthritis Rheum; 2007 Sep 01; 56(9):3057-69. PubMed ID: 17763445
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  • 19. BAFF-driven autoimmunity requires CD19 expression.
    Fairfax KA, Tsantikos E, Figgett WA, Vincent FB, Quah PS, LePage M, Hibbs ML, Mackay F.
    J Autoimmun; 2015 Aug 01; 62():1-10. PubMed ID: 26103922
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  • 20. Epistatic suppression of systemic lupus erythematosus: fine mapping of Sles1 to less than 1 mb.
    Subramanian S, Yim YS, Liu K, Tus K, Zhou XJ, Wakeland EK.
    J Immunol; 2005 Jul 15; 175(2):1062-72. PubMed ID: 16002707
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