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Journal Abstract Search

270 related items for PubMed ID: 20508195

  • 1. The Akt-endothelial nitric oxide synthase pathway in lipopolysaccharide preconditioning-induced hypoxic-ischemic tolerance in the neonatal rat brain.
    Lin HY, Wu CL, Huang CC.
    Stroke; 2010 Jul; 41(7):1543-51. PubMed ID: 20508195
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  • 2. Ischemic preconditioning reduces neurovascular damage after hypoxia-ischemia via the cellular inhibitor of apoptosis 1 in neonatal brain.
    Lin WY, Chang YC, Ho CJ, Huang CC.
    Stroke; 2013 Jan; 44(1):162-9. PubMed ID: 23192759
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  • 3. Lipopolysaccharide preconditioning reduces neuroinflammation against hypoxic ischemia and provides long-term outcome of neuroprotection in neonatal rat.
    Lin HY, Huang CC, Chang KF.
    Pediatr Res; 2009 Sep; 66(3):254-9. PubMed ID: 19531979
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  • 4. Prophylactic but not delayed administration of simvastatin protects against long-lasting cognitive and morphological consequences of neonatal hypoxic-ischemic brain injury, reduces interleukin-1beta and tumor necrosis factor-alpha mRNA induction, and does not affect endothelial nitric oxide synthase expression.
    Balduini W, Mazzoni E, Carloni S, De Simoni MG, Perego C, Sironi L, Cimino M.
    Stroke; 2003 Aug; 34(8):2007-12. PubMed ID: 12829860
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  • 5. Preconditioning suppresses inflammation in neonatal hypoxic ischemia via Akt activation.
    Yin W, Signore AP, Iwai M, Cao G, Gao Y, Johnnides MJ, Hickey RW, Chen J.
    Stroke; 2007 Mar; 38(3):1017-24. PubMed ID: 17272774
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  • 6. Insulin Receptor Substrate-1 Activation Mediated p53 Downregulation Protects Against Hypoxic-Ischemia in the Neonatal Brain.
    Tu YF, Jiang ST, Chow YH, Huang CC, Ho CJ, Chou YP.
    Mol Neurobiol; 2016 Aug; 53(6):3658-3669. PubMed ID: 26111627
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  • 7. Hypoxic preconditioning provides neuroprotection and increases vascular endothelial growth factor A, preserves the phosphorylation of Akt-Ser-473 and diminishes the increase in caspase-3 activity in neonatal rat hypoxic-ischemic model.
    Feng Y, Rhodes PG, Bhatt AJ.
    Brain Res; 2010 Apr 14; 1325():1-9. PubMed ID: 20153736
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  • 8. Protective effects of lipopolysaccharide preconditioning against nitric oxide neurotoxicity.
    Huang CY, Yang HI, Chen SD, Shaw FZ, Yang DI.
    J Neurosci Res; 2008 May 01; 86(6):1277-89. PubMed ID: 18092358
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  • 9. Reduced nitric oxide is involved in prenatal ischemia-induced tolerance to neonatal hypoxic-ischemic brain injury in rats.
    Xiao F, Fratkin JD, Rhodes PG, Cai Z.
    Neurosci Lett; 2000 May 05; 285(1):5-8. PubMed ID: 10788694
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  • 13. Remote limb ischemic postconditioning protects against neonatal hypoxic-ischemic brain injury in rat pups by the opioid receptor/Akt pathway.
    Zhou Y, Fathali N, Lekic T, Ostrowski RP, Chen C, Martin RD, Tang J, Zhang JH.
    Stroke; 2011 Feb 05; 42(2):439-44. PubMed ID: 21183744
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  • 14. Isoflurane posttreatment reduces neonatal hypoxic-ischemic brain injury in rats by the sphingosine-1-phosphate/phosphatidylinositol-3-kinase/Akt pathway.
    Zhou Y, Lekic T, Fathali N, Ostrowski RP, Martin RD, Tang J, Zhang JH.
    Stroke; 2010 Jul 05; 41(7):1521-7. PubMed ID: 20508187
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  • 17. Recombinant human erythropoietin preconditioning attenuates liver ischemia reperfusion injury through the phosphatidylinositol-3 kinase/AKT/endothelial nitric oxide synthase pathway.
    Fu W, Liao X, Ruan J, Li X, Chen L, Wang B, Wang K, Zhou J.
    J Surg Res; 2013 Aug 05; 183(2):876-84. PubMed ID: 23490139
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  • 18. Up-regulation of endothelial nitric oxide synthase via phosphatidylinositol 3-kinase pathway contributes to ischemic tolerance in the CA1 subfield of gerbil hippocampus.
    Hashiguchi A, Yano S, Morioka M, Hamada J, Ushio Y, Takeuchi Y, Fukunaga K.
    J Cereb Blood Flow Metab; 2004 Mar 05; 24(3):271-9. PubMed ID: 15091107
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  • 19. TNFR1-JNK signaling is the shared pathway of neuroinflammation and neurovascular damage after LPS-sensitized hypoxic-ischemic injury in the immature brain.
    Wang LW, Chang YC, Chen SJ, Tseng CH, Tu YF, Liao NS, Huang CC, Ho CJ.
    J Neuroinflammation; 2014 Dec 24; 11():215. PubMed ID: 25540015
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