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162 related items for PubMed ID: 20615469

  • 1. Amyloid-β peptide alteration of tau exon-10 splicing via the GSK3β-SC35 pathway.
    Chen KL, Yuan RY, Hu CJ, Hsu CY.
    Neurobiol Dis; 2010 Nov; 40(2):378-85. PubMed ID: 20615469
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  • 2. Glycogen synthase kinase-3 plays a crucial role in tau exon 10 splicing and intranuclear distribution of SC35. Implications for Alzheimer's disease.
    Hernández F, Pérez M, Lucas JJ, Mata AM, Bhat R, Avila J.
    J Biol Chem; 2004 Jan 30; 279(5):3801-6. PubMed ID: 14602710
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  • 3. SIRT1 Deacetylates SC35 and Suppresses Its Function in Tau Exon 10 Inclusion.
    Yin X, Jiang X, Wang J, Qian S, Liu F, Qian W.
    J Alzheimers Dis; 2018 Jan 30; 61(2):561-570. PubMed ID: 29226865
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  • 4. Splicing factor SC35 promotes tau expression through stabilization of its mRNA.
    Qian W, Iqbal K, Grundke-Iqbal I, Gong CX, Liu F.
    FEBS Lett; 2011 Mar 23; 585(6):875-80. PubMed ID: 21333649
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  • 9. Antibodies against beta-amyloid reduce Abeta oligomers, glycogen synthase kinase-3beta activation and tau phosphorylation in vivo and in vitro.
    Ma QL, Lim GP, Harris-White ME, Yang F, Ambegaokar SS, Ubeda OJ, Glabe CG, Teter B, Frautschy SA, Cole GM.
    J Neurosci Res; 2006 Feb 15; 83(3):374-84. PubMed ID: 16385556
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  • 10. Curcumin attenuates amyloid-β-induced tau hyperphosphorylation in human neuroblastoma SH-SY5Y cells involving PTEN/Akt/GSK-3β signaling pathway.
    Huang HC, Tang D, Xu K, Jiang ZF.
    J Recept Signal Transduct Res; 2014 Feb 15; 34(1):26-37. PubMed ID: 24188406
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  • 13. Abeta(1-42) induces abnormal alternative splicing of tau exons 2/3 in NGF-induced PC12 cells.
    Lagunes T, Herrera-Rivero M, Hernández-Aguilar ME, Aranda-Abreu GE.
    An Acad Bras Cienc; 2014 Dec 15; 86(4):1927-34. PubMed ID: 25590729
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  • 16. Transactive response DNA-binding protein 43 (TDP-43) regulates alternative splicing of tau exon 10: Implications for the pathogenesis of tauopathies.
    Gu J, Chen F, Iqbal K, Gong CX, Wang X, Liu F.
    J Biol Chem; 2017 Jun 23; 292(25):10600-10612. PubMed ID: 28487370
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  • 17. Chronic lithium administration to FTDP-17 tau and GSK-3beta overexpressing mice prevents tau hyperphosphorylation and neurofibrillary tangle formation, but pre-formed neurofibrillary tangles do not revert.
    Engel T, Goñi-Oliver P, Lucas JJ, Avila J, Hernández F.
    J Neurochem; 2006 Dec 23; 99(6):1445-55. PubMed ID: 17059563
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