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Journal Abstract Search

309 related items for PubMed ID: 20838240

  • 1. Downregulation of the potassium chloride cotransporter KCC2 in vulnerable motoneurons in the SOD1-G93A mouse model of amyotrophic lateral sclerosis.
    Fuchs A, Ringer C, Bilkei-Gorzo A, Weihe E, Roeper J, Schütz B.
    J Neuropathol Exp Neurol; 2010 Oct; 69(10):1057-70. PubMed ID: 20838240
    [Abstract] [Full Text] [Related]

  • 2. Implication of 5-HT in the Dysregulation of Chloride Homeostasis in Prenatal Spinal Motoneurons from the G93A Mouse Model of Amyotrophic Lateral Sclerosis.
    Martin E, Cazenave W, Allain AE, Cattaert D, Branchereau P.
    Int J Mol Sci; 2020 Feb 07; 21(3):. PubMed ID: 32046135
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  • 3. Knocking down metabotropic glutamate receptor 1 improves survival and disease progression in the SOD1(G93A) mouse model of amyotrophic lateral sclerosis.
    Milanese M, Giribaldi F, Melone M, Bonifacino T, Musante I, Carminati E, Rossi PI, Vergani L, Voci A, Conti F, Puliti A, Bonanno G.
    Neurobiol Dis; 2014 Apr 07; 64():48-59. PubMed ID: 24361555
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  • 4. Glycine receptor channels in spinal motoneurons are abnormal in a transgenic mouse model of amyotrophic lateral sclerosis.
    Chang Q, Martin LJ.
    J Neurosci; 2011 Feb 23; 31(8):2815-27. PubMed ID: 21414903
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  • 5. Human Cu/Zn superoxide dismutase (SOD1) overexpression in mice causes mitochondrial vacuolization, axonal degeneration, and premature motoneuron death and accelerates motoneuron disease in mice expressing a familial amyotrophic lateral sclerosis mutant SOD1.
    Jaarsma D, Haasdijk ED, Grashorn JA, Hawkins R, van Duijn W, Verspaget HW, London J, Holstege JC.
    Neurobiol Dis; 2000 Dec 23; 7(6 Pt B):623-43. PubMed ID: 11114261
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  • 8. Ablation of P2X7 receptor exacerbates gliosis and motoneuron death in the SOD1-G93A mouse model of amyotrophic lateral sclerosis.
    Apolloni S, Amadio S, Montilli C, Volonté C, D'Ambrosi N.
    Hum Mol Genet; 2013 Oct 15; 22(20):4102-16. PubMed ID: 23736299
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  • 9. In vivo quantification of spinal and bulbar motor neuron degeneration in the G93A-SOD1 transgenic mouse model of ALS by T2 relaxation time and apparent diffusion coefficient.
    Niessen HG, Angenstein F, Sander K, Kunz WS, Teuchert M, Ludolph AC, Heinze HJ, Scheich H, Vielhaber S.
    Exp Neurol; 2006 Oct 15; 201(2):293-300. PubMed ID: 16740261
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  • 10. Calreticulin levels determine onset of early muscle denervation by fast motoneurons of ALS model mice.
    Bernard-Marissal N, Sunyach C, Marissal T, Raoul C, Pettmann B.
    Neurobiol Dis; 2015 Jan 15; 73():130-6. PubMed ID: 25277755
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  • 11. Neuregulin 1 confers neuroprotection in SOD1-linked amyotrophic lateral sclerosis mice via restoration of C-boutons of spinal motor neurons.
    Lasiene J, Komine O, Fujimori-Tonou N, Powers B, Endo F, Watanabe S, Shijie J, Ravits J, Horner P, Misawa H, Yamanaka K.
    Acta Neuropathol Commun; 2016 Feb 18; 4():15. PubMed ID: 26891847
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  • 12. Parvalbumin and calbindin D-28k immunoreactivity in transgenic mice with a G93A mutant SOD1 gene.
    Sasaki S, Warita H, Komori T, Murakami T, Abe K, Iwata M.
    Brain Res; 2006 Apr 14; 1083(1):196-203. PubMed ID: 16546142
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  • 14. Voltage-gated calcium channels are abnormal in cultured spinal motoneurons in the G93A-SOD1 transgenic mouse model of ALS.
    Chang Q, Martin LJ.
    Neurobiol Dis; 2016 Sep 14; 93():78-95. PubMed ID: 27151771
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  • 15. Developmental up-regulation of the potassium-chloride cotransporter type 2 in the rat lumbar spinal cord.
    Stil A, Liabeuf S, Jean-Xavier C, Brocard C, Viemari JC, Vinay L.
    Neuroscience; 2009 Dec 01; 164(2):809-21. PubMed ID: 19699273
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  • 16. Hyperexcitability of cultured spinal motoneurons from presymptomatic ALS mice.
    Kuo JJ, Schonewille M, Siddique T, Schults AN, Fu R, Bär PR, Anelli R, Heckman CJ, Kroese AB.
    J Neurophysiol; 2004 Jan 01; 91(1):571-5. PubMed ID: 14523070
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  • 17. Differential involvement of vesicular and glial glutamate transporters around spinal α-motoneurons in the pathogenesis of SOD1G93A mouse model of amyotrophic lateral sclerosis.
    Ohgomori T, Yamasaki R, Takeuchi H, Kadomatsu K, Kira JI, Jinno S.
    Neuroscience; 2017 Jul 25; 356():114-124. PubMed ID: 28526579
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  • 18. Imbalanced excitatory to inhibitory synaptic input precedes motor neuron degeneration in an animal model of amyotrophic lateral sclerosis.
    Schütz B.
    Neurobiol Dis; 2005 Oct 25; 20(1):131-40. PubMed ID: 16137574
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  • 19. Postnatal electrical and morphological abnormalities in lumbar motoneurons from transgenic mouse models of amyotrophic lateral sclerosis.
    Amendola J, Gueritaud JP, Lamotte d'Incamps B, Bories C, Liabeuf S, Allene C, Pambo-Pambo A, Durand J.
    Arch Ital Biol; 2007 Nov 25; 145(3-4):311-23. PubMed ID: 18075124
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  • 20. Progressive changes in synaptic inputs to motoneurons in adult sacral spinal cord of a mouse model of amyotrophic lateral sclerosis.
    Jiang M, Schuster JE, Fu R, Siddique T, Heckman CJ.
    J Neurosci; 2009 Dec 02; 29(48):15031-8. PubMed ID: 19955354
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