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Journal Abstract Search

196 related items for PubMed ID: 20844317

  • 1. ATM-independent, high-fidelity nonhomologous end joining predominates in human embryonic stem cells.
    Adams BR, Hawkins AJ, Povirk LF, Valerie K.
    Aging (Albany NY); 2010 Sep; 2(9):582-96. PubMed ID: 20844317
    [Abstract] [Full Text] [Related]

  • 2. Dynamic dependence on ATR and ATM for double-strand break repair in human embryonic stem cells and neural descendants.
    Adams BR, Golding SE, Rao RR, Valerie K.
    PLoS One; 2010 Apr 02; 5(4):e10001. PubMed ID: 20368801
    [Abstract] [Full Text] [Related]

  • 3. Inhibition of poly (ADP-ribose) polymerase activates ATM which is required for subsequent homologous recombination repair.
    Bryant HE, Helleday T.
    Nucleic Acids Res; 2006 Apr 02; 34(6):1685-91. PubMed ID: 16556909
    [Abstract] [Full Text] [Related]

  • 4. Aberrant V(D)J recombination in ataxia telangiectasia mutated-deficient lymphocytes is dependent on nonhomologous DNA end joining.
    Bredemeyer AL, Huang CY, Walker LM, Bassing CH, Sleckman BP.
    J Immunol; 2008 Aug 15; 181(4):2620-5. PubMed ID: 18684952
    [Abstract] [Full Text] [Related]

  • 5. DNA repair factors and telomere-chromosome integrity in mammalian cells.
    Hande MP.
    Cytogenet Genome Res; 2004 Aug 15; 104(1-4):116-22. PubMed ID: 15162024
    [Abstract] [Full Text] [Related]

  • 6. Ataxia telangiectasia-mutated protein and DNA-dependent protein kinase have complementary V(D)J recombination functions.
    Zha S, Jiang W, Fujiwara Y, Patel H, Goff PH, Brush JW, Dubois RL, Alt FW.
    Proc Natl Acad Sci U S A; 2011 Feb 01; 108(5):2028-33. PubMed ID: 21245310
    [Abstract] [Full Text] [Related]

  • 7. Double strand break repair by homologous recombination is regulated by cell cycle-independent signaling via ATM in human glioma cells.
    Golding SE, Rosenberg E, Khalil A, McEwen A, Holmes M, Neill S, Povirk LF, Valerie K.
    J Biol Chem; 2004 Apr 09; 279(15):15402-10. PubMed ID: 14744854
    [Abstract] [Full Text] [Related]

  • 8. Mystery of DNA repair: the role of the MRN complex and ATM kinase in DNA damage repair.
    Czornak K, Chughtai S, Chrzanowska KH.
    J Appl Genet; 2008 Apr 09; 49(4):383-96. PubMed ID: 19029686
    [Abstract] [Full Text] [Related]

  • 9. Ataxia telangiectasia mutated (ATM) is essential for DNA-PKcs phosphorylations at the Thr-2609 cluster upon DNA double strand break.
    Chen BP, Uematsu N, Kobayashi J, Lerenthal Y, Krempler A, Yajima H, Löbrich M, Shiloh Y, Chen DJ.
    J Biol Chem; 2007 Mar 02; 282(9):6582-7. PubMed ID: 17189255
    [Abstract] [Full Text] [Related]

  • 10. Genetic interaction between DNA repair factors PAXX, XLF, XRCC4 and DNA-PKcs in human cells.
    Xing M, Oksenych V.
    FEBS Open Bio; 2019 Jul 02; 9(7):1315-1326. PubMed ID: 31141305
    [Abstract] [Full Text] [Related]

  • 11. DNA-PKcs and ATM co-regulate DNA double-strand break repair.
    Shrivastav M, Miller CA, De Haro LP, Durant ST, Chen BP, Chen DJ, Nickoloff JA.
    DNA Repair (Amst); 2009 Aug 06; 8(8):920-9. PubMed ID: 19535303
    [Abstract] [Full Text] [Related]

  • 12. Unique and redundant functions of ATM and DNA-PKcs during V(D)J recombination.
    Gapud EJ, Sleckman BP.
    Cell Cycle; 2011 Jun 15; 10(12):1928-35. PubMed ID: 21673501
    [Abstract] [Full Text] [Related]

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  • 15. Ataxia telangiectasia mutated and checkpoint kinase 2 regulate BRCA1 to promote the fidelity of DNA end-joining.
    Wang HC, Chou WC, Shieh SY, Shen CY.
    Cancer Res; 2006 Feb 01; 66(3):1391-400. PubMed ID: 16452194
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  • 17. Extracellular signal-related kinase positively regulates ataxia telangiectasia mutated, homologous recombination repair, and the DNA damage response.
    Golding SE, Rosenberg E, Neill S, Dent P, Povirk LF, Valerie K.
    Cancer Res; 2007 Feb 01; 67(3):1046-53. PubMed ID: 17283137
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  • 19. Regulation of DNA double-strand break repair pathway choice.
    Shrivastav M, De Haro LP, Nickoloff JA.
    Cell Res; 2008 Jan 01; 18(1):134-47. PubMed ID: 18157161
    [Abstract] [Full Text] [Related]

  • 20. Requirement for Parp-1 and DNA ligases 1 or 3 but not of Xrcc1 in chromosomal translocation formation by backup end joining.
    Soni A, Siemann M, Grabos M, Murmann T, Pantelias GE, Iliakis G.
    Nucleic Acids Res; 2014 Jun 01; 42(10):6380-92. PubMed ID: 24748665
    [Abstract] [Full Text] [Related]

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