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Journal Abstract Search

354 related items for PubMed ID: 20852043

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  • 3. Myocardial overexpression of TIMP3 after myocardial infarction exerts beneficial effects by promoting angiogenesis and suppressing early proteolysis.
    Takawale A, Zhang P, Azad A, Wang W, Wang X, Murray AG, Kassiri Z.
    Am J Physiol Heart Circ Physiol; 2017 Aug 01; 313(2):H224-H236. PubMed ID: 28550172
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  • 7. Cardiac overexpression of monocyte chemoattractant protein-1 in transgenic mice prevents cardiac dysfunction and remodeling after myocardial infarction.
    Morimoto H, Takahashi M, Izawa A, Ise H, Hongo M, Kolattukudy PE, Ikeda U.
    Circ Res; 2006 Oct 13; 99(8):891-9. PubMed ID: 16990567
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  • 8. Anti-monocyte chemoattractant protein-1 gene therapy attenuates left ventricular remodeling and failure after experimental myocardial infarction.
    Hayashidani S, Tsutsui H, Shiomi T, Ikeuchi M, Matsusaka H, Suematsu N, Wen J, Egashira K, Takeshita A.
    Circulation; 2003 Oct 28; 108(17):2134-40. PubMed ID: 14517168
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  • 10. Antibody against interleukin-6 receptor attenuates left ventricular remodelling after myocardial infarction in mice.
    Kobara M, Noda K, Kitamura M, Okamoto A, Shiraishi T, Toba H, Matsubara H, Nakata T.
    Cardiovasc Res; 2010 Aug 01; 87(3):424-30. PubMed ID: 20211866
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  • 11. Splenic Ly6Chi monocytes contribute to adverse late post-ischemic left ventricular remodeling in heme oxygenase-1 deficient mice.
    Tomczyk M, Kraszewska I, Szade K, Bukowska-Strakova K, Meloni M, Jozkowicz A, Dulak J, Jazwa A.
    Basic Res Cardiol; 2017 Jul 01; 112(4):39. PubMed ID: 28534119
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  • 12. Cardiomyocyte-specific overexpression of nitric oxide synthase 3 improves left ventricular performance and reduces compensatory hypertrophy after myocardial infarction.
    Janssens S, Pokreisz P, Schoonjans L, Pellens M, Vermeersch P, Tjwa M, Jans P, Scherrer-Crosbie M, Picard MH, Szelid Z, Gillijns H, Van de Werf F, Collen D, Bloch KD.
    Circ Res; 2004 May 14; 94(9):1256-62. PubMed ID: 15044322
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  • 14. Cardiac-specific overexpression of diacylglycerol kinase zeta attenuates left ventricular remodeling and improves survival after myocardial infarction.
    Niizeki T, Takeishi Y, Arimoto T, Takahashi H, Shishido T, Koyama Y, Goto K, Walsh RA, Kubota I.
    Am J Physiol Heart Circ Physiol; 2007 Feb 14; 292(2):H1105-12. PubMed ID: 17071729
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  • 15. Association of dietary iron restriction with left ventricular remodeling after myocardial infarction in mice.
    Eguchi A, Naito Y, Iwasaku T, Okuhara Y, Morisawa D, Sawada H, Nishimura K, Oboshi M, Fujii K, Mano T, Masuyama T, Hirotani S.
    Heart Vessels; 2016 Feb 14; 31(2):222-9. PubMed ID: 25573257
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  • 19. IL-33 induces type-2-cytokine phenotype but exacerbates cardiac remodeling post-myocardial infarction with eosinophil recruitment, worsened systolic dysfunction, and ventricular wall rupture.
    Ghali R, Habeichi NJ, Kaplan A, Tannous C, Abidi E, Bekdash A, Farhat R, Itani H, Jurjus A, Booz GW, Mallat Z, Zouein FA.
    Clin Sci (Lond); 2020 Jun 12; 134(11):1191-1218. PubMed ID: 32432676
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  • 20. Overexpression of human C-reactive protein exacerbates left ventricular remodeling in diabetic cardiomyopathy.
    Mano Y, Anzai T, Kaneko H, Nagatomo Y, Nagai T, Anzai A, Maekawa Y, Takahashi T, Meguro T, Yoshikawa T, Fukuda K.
    Circ J; 2011 Jun 12; 75(7):1717-27. PubMed ID: 21519150
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