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Journal Abstract Search

354 related items for PubMed ID: 20852043

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  • 26. Pioglitazone, a peroxisome proliferator-activated receptor-gamma agonist, attenuates left ventricular remodeling and failure after experimental myocardial infarction.
    Shiomi T, Tsutsui H, Hayashidani S, Suematsu N, Ikeuchi M, Wen J, Ishibashi M, Kubota T, Egashira K, Takeshita A.
    Circulation; 2002 Dec 10; 106(24):3126-32. PubMed ID: 12473562
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  • 27. Acute left ventricular remodeling following myocardial infarction: coupling of regional healing with remote extracellular matrix expansion.
    Chan W, Duffy SJ, White DA, Gao XM, Du XJ, Ellims AH, Dart AM, Taylor AJ.
    JACC Cardiovasc Imaging; 2012 Sep 10; 5(9):884-93. PubMed ID: 22974800
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  • 29. Early matrix metalloproteinase-9 inhibition post-myocardial infarction worsens cardiac dysfunction by delaying inflammation resolution.
    Iyer RP, de Castro Brás LE, Patterson NL, Bhowmick M, Flynn ER, Asher M, Cannon PL, Deleon-Pennell KY, Fields GB, Lindsey ML.
    J Mol Cell Cardiol; 2016 Nov 10; 100():109-117. PubMed ID: 27746126
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  • 32. C-reactive protein during and after myocardial infarction in relation to cardiac injury and left ventricular function at follow-up.
    Vanhaverbeke M, Veltman D, Pattyn N, De Crem N, Gillijns H, Cornelissen V, Janssens S, Sinnaeve PR.
    Clin Cardiol; 2018 Sep 10; 41(9):1201-1206. PubMed ID: 29952015
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  • 33. Exaggerated left ventricular dilation and reduced collagen deposition after myocardial infarction in mice lacking osteopontin.
    Trueblood NA, Xie Z, Communal C, Sam F, Ngoy S, Liaw L, Jenkins AW, Wang J, Sawyer DB, Bing OH, Apstein CS, Colucci WS, Singh K.
    Circ Res; 2001 May 25; 88(10):1080-7. PubMed ID: 11375279
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  • 34. Endothelial cells overexpressing IL-8 receptor reduce cardiac remodeling and dysfunction following myocardial infarction.
    Zhao X, Zhang W, Xing D, Li P, Fu J, Gong K, Hage FG, Oparil S, Chen YF.
    Am J Physiol Heart Circ Physiol; 2013 Aug 15; 305(4):H590-8. PubMed ID: 23771691
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  • 35. Susceptibility to systolic dysfunction in the myocardium from chronically infarcted spontaneously hypertensive rats.
    Norton GR, Veliotes DG, Osadchii O, Woodiwiss AJ, Thomas DP.
    Am J Physiol Heart Circ Physiol; 2008 Jan 15; 294(1):H372-8. PubMed ID: 17993597
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  • 37. Early activation of matrix metalloproteinases underlies the exacerbated systolic and diastolic dysfunction in mice lacking TIMP3 following myocardial infarction.
    Kandalam V, Basu R, Abraham T, Wang X, Awad A, Wang W, Lopaschuk GD, Maeda N, Oudit GY, Kassiri Z.
    Am J Physiol Heart Circ Physiol; 2010 Oct 15; 299(4):H1012-23. PubMed ID: 20675565
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  • 39. IL-10 inhibits inflammation and attenuates left ventricular remodeling after myocardial infarction via activation of STAT3 and suppression of HuR.
    Krishnamurthy P, Rajasingh J, Lambers E, Qin G, Losordo DW, Kishore R.
    Circ Res; 2009 Jan 30; 104(2):e9-18. PubMed ID: 19096025
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  • 40. Exogenous CXCL4 infusion inhibits macrophage phagocytosis by limiting CD36 signalling to enhance post-myocardial infarction cardiac dilation and mortality.
    Lindsey ML, Jung M, Yabluchanskiy A, Cannon PL, Iyer RP, Flynn ER, DeLeon-Pennell KY, Valerio FM, Harrison CL, Ripplinger CM, Hall ME, Ma Y.
    Cardiovasc Res; 2019 Feb 01; 115(2):395-408. PubMed ID: 30169632
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