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Journal Abstract Search


174 related items for PubMed ID: 20884027

  • 1. Transient mitochondrial permeability transition pore opening after neonatal cardioplegic arrest.
    Leung CH, Wang L, Fu YY, Yuen W, Caldarone CA.
    J Thorac Cardiovasc Surg; 2011 Apr; 141(4):975-82. PubMed ID: 20884027
    [Abstract] [Full Text] [Related]

  • 2. Cyclosporine A prevents apoptosis-related mitochondrial dysfunction after neonatal cardioplegic arrest.
    Oka N, Wang L, Mi W, Zhu W, Honjo O, Caldarone CA.
    J Thorac Cardiovasc Surg; 2008 Jan; 135(1):123-30, 130.e1-2. PubMed ID: 18179928
    [Abstract] [Full Text] [Related]

  • 3. Inhibition of mitochondrial remodeling by cyclosporine A preserves myocardial performance in a neonatal rabbit model of cardioplegic arrest.
    Oka N, Wang L, Mi W, Caldarone CA.
    J Thorac Cardiovasc Surg; 2008 Mar; 135(3):585-93. PubMed ID: 18329475
    [Abstract] [Full Text] [Related]

  • 4. Remote ischemic preconditioning elaborates a transferable blood-borne effector that protects mitochondrial structure and function and preserves myocardial performance after neonatal cardioplegic arrest.
    Wang L, Oka N, Tropak M, Callahan J, Lee J, Wilson G, Redington A, Caldarone CA.
    J Thorac Cardiovasc Surg; 2008 Aug; 136(2):335-42. PubMed ID: 18692639
    [Abstract] [Full Text] [Related]

  • 5. Orientin-induced cardioprotection against reperfusion is associated with attenuation of mitochondrial permeability transition.
    Lu N, Sun Y, Zheng X.
    Planta Med; 2011 Jul; 77(10):984-91. PubMed ID: 21283956
    [Abstract] [Full Text] [Related]

  • 6. Mitochondrial permeability transition relevance for apoptotic triggering in the post-ischemic heart.
    Correa F, Soto V, Zazueta C.
    Int J Biochem Cell Biol; 2007 Jul; 39(4):787-98. PubMed ID: 17306600
    [Abstract] [Full Text] [Related]

  • 7. Inhibition of mitochondrial permeability transition pore opening is involved in the protective effects of mortalin overexpression against beta-amyloid-induced apoptosis in SH-SY5Y cells.
    Qu M, Zhou Z, Chen C, Li M, Pei L, Yang J, Wang Y, Li L, Liu C, Zhang G, Yu Z, Wang D.
    Neurosci Res; 2012 Jan; 72(1):94-102. PubMed ID: 22001761
    [Abstract] [Full Text] [Related]

  • 8. Mechanical stretch induces mitochondria-dependent apoptosis in neonatal rat cardiomyocytes and G2/M accumulation in cardiac fibroblasts.
    Liao XD, Wang XH, Jin HJ, Chen LY, Chen Q.
    Cell Res; 2004 Feb; 14(1):16-26. PubMed ID: 15040886
    [Abstract] [Full Text] [Related]

  • 9. Increased expression and intramitochondrial translocation of cyclophilin-D associates with increased vulnerability of the permeability transition pore to stress-induced opening during compensated ventricular hypertrophy.
    Matas J, Young NT, Bourcier-Lucas C, Ascah A, Marcil M, Deschepper CF, Burelle Y.
    J Mol Cell Cardiol; 2009 Mar; 46(3):420-30. PubMed ID: 19094991
    [Abstract] [Full Text] [Related]

  • 10. Inhibition of Bcl-2 sensitizes mitochondrial permeability transition pore (MPTP) opening in ischemia-damaged mitochondria.
    Chen Q, Xu H, Xu A, Ross T, Bowler E, Hu Y, Lesnefsky EJ.
    PLoS One; 2015 Mar; 10(3):e0118834. PubMed ID: 25756500
    [Abstract] [Full Text] [Related]

  • 11. Apoptosis-related mitochondrial dysfunction in the early postoperative neonatal lamb heart.
    Caldarone CA, Barner EW, Wang L, Karimi M, Mascio CE, Hammel JM, Segar JL, Du C, Scholz TD.
    Ann Thorac Surg; 2004 Sep; 78(3):948-55. PubMed ID: 15337026
    [Abstract] [Full Text] [Related]

  • 12. A tale of two mitochondrial channels, MAC and PTP, in apoptosis.
    Kinnally KW, Antonsson B.
    Apoptosis; 2007 May; 12(5):857-68. PubMed ID: 17294079
    [Abstract] [Full Text] [Related]

  • 13. Detailing the role of Bax translocation, cytochrome c release, and perinuclear clustering of the mitochondria in the killing of HeLa cells by TNF.
    Pucci B, Bertani F, Karpinich NO, Indelicato M, Russo MA, Farber JL, Tafani M.
    J Cell Physiol; 2008 Nov; 217(2):442-9. PubMed ID: 18546202
    [Abstract] [Full Text] [Related]

  • 14. Caspase-dependent alteration of the ADP/ATP translocator triggers the mitochondrial permeability transition which is not required for the low-potassium-dependent apoptosis of cerebellar granule cells.
    Atlante A, Bobba A, de Bari L, Fontana F, Calissano P, Marra E, Passarella S.
    J Neurochem; 2006 May; 97(4):1166-81. PubMed ID: 16606362
    [Abstract] [Full Text] [Related]

  • 15. Vitamin E protects against the mitochondrial damage caused by cyclosporin A in LLC-PK1 cells.
    de Arriba G, de Hornedo JP, Rubio SR, Fernández MC, Martínez SB, Camarero MM, Cid TP.
    Toxicol Appl Pharmacol; 2009 Sep 15; 239(3):241-50. PubMed ID: 19523970
    [Abstract] [Full Text] [Related]

  • 16. Danthron, an anthraquinone derivative, induces DNA damage and caspase cascades-mediated apoptosis in SNU-1 human gastric cancer cells through mitochondrial permeability transition pores and Bax-triggered pathways.
    Chiang JH, Yang JS, Ma CY, Yang MD, Huang HY, Hsia TC, Kuo HM, Wu PP, Lee TH, Chung JG.
    Chem Res Toxicol; 2011 Jan 14; 24(1):20-9. PubMed ID: 21126053
    [Abstract] [Full Text] [Related]

  • 17. Blockade of electron transport during ischemia preserves bcl-2 and inhibits opening of the mitochondrial permeability transition pore.
    Chen Q, Lesnefsky EJ.
    FEBS Lett; 2011 Mar 23; 585(6):921-6. PubMed ID: 21354418
    [Abstract] [Full Text] [Related]

  • 18. High fluence low-power laser irradiation induces mitochondrial permeability transition mediated by reactive oxygen species.
    Wu S, Xing D, Gao X, Chen WR.
    J Cell Physiol; 2009 Mar 23; 218(3):603-11. PubMed ID: 19006121
    [Abstract] [Full Text] [Related]

  • 19. Preservation of mitochondrial structure and function after cardioplegic arrest in the neonate using a selective mitochondrial KATP channel opener.
    Wang L, Kinnear C, Hammel JM, Zhu W, Hua Z, Mi W, Caldarone CA.
    Ann Thorac Surg; 2006 May 23; 81(5):1817-23. PubMed ID: 16631678
    [Abstract] [Full Text] [Related]

  • 20. Bcl-2 inhibitors sensitize tumor necrosis factor-related apoptosis-inducing ligand-induced apoptosis by uncoupling of mitochondrial respiration in human leukemic CEM cells.
    Hao JH, Yu M, Liu FT, Newland AC, Jia L.
    Cancer Res; 2004 May 15; 64(10):3607-16. PubMed ID: 15150119
    [Abstract] [Full Text] [Related]


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