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Journal Abstract Search

336 related items for PubMed ID: 20890745

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  • 3. P2Y1 purinergic receptor identified as a diabetes target in a small-molecule screen to reverse circadian β-cell failure.
    Marcheva B, Weidemann BJ, Taguchi A, Perelis M, Ramsey KM, Newman MV, Kobayashi Y, Omura C, Manning Fox JE, Lin H, Macdonald PE, Bass J.
    Elife; 2022 Feb 21; 11():. PubMed ID: 35188462
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  • 4. Induction of Core Circadian Clock Transcription Factor Bmal1 Enhances β-Cell Function and Protects Against Obesity-Induced Glucose Intolerance.
    Rakshit K, Matveyenko AV.
    Diabetes; 2021 Jan 21; 70(1):143-154. PubMed ID: 33087455
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  • 6. Alteration in glucose homeostasis and persistence of the pancreatic clock in aged mPer2Luc mice.
    Novosadová Z, Polidarová L, Sládek M, Sumová A.
    Sci Rep; 2018 Aug 03; 8(1):11668. PubMed ID: 30076390
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  • 7. Clock genes, pancreatic function, and diabetes.
    Vieira E, Burris TP, Quesada I.
    Trends Mol Med; 2014 Dec 03; 20(12):685-93. PubMed ID: 25457619
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  • 9. Role of the clock gene Rev-erbα in metabolism and in the endocrine pancreas.
    Vieira E, Merino B, Quesada I.
    Diabetes Obes Metab; 2015 Sep 03; 17 Suppl 1():106-14. PubMed ID: 26332975
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  • 10. Bmal1 and β-cell clock are required for adaptation to circadian disruption, and their loss of function leads to oxidative stress-induced β-cell failure in mice.
    Lee J, Moulik M, Fang Z, Saha P, Zou F, Xu Y, Nelson DL, Ma K, Moore DD, Yechoor VK.
    Mol Cell Biol; 2013 Jun 03; 33(11):2327-38. PubMed ID: 23547261
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  • 12. Loss of melatonin signalling and its impact on circadian rhythms in mouse organs regulating blood glucose.
    Mühlbauer E, Gross E, Labucay K, Wolgast S, Peschke E.
    Eur J Pharmacol; 2009 Mar 15; 606(1-3):61-71. PubMed ID: 19374844
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