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Journal Abstract Search

311 related items for PubMed ID: 21078175

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  • 2. Comparative analysis of the time-dependent functional and molecular changes in spinal cord degeneration induced by the G93A SOD1 gene mutation and by mechanical compression.
    Malaspina A, Jokic N, Huang WL, Priestley JV.
    BMC Genomics; 2008 Oct 23; 9():500. PubMed ID: 18947433
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  • 6. Progressive impairment of CaV1.1 function in the skeletal muscle of mice expressing a mutant type 1 Cu/Zn superoxide dismutase (G93A) linked to amyotrophic lateral sclerosis.
    Beqollari D, Romberg CF, Dobrowolny G, Martini M, Voss AA, Musarò A, Bannister RA.
    Skelet Muscle; 2016 Oct 23; 6():24. PubMed ID: 27340545
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  • 7. Different human copper-zinc superoxide dismutase mutants, SOD1G93A and SOD1H46R, exert distinct harmful effects on gross phenotype in mice.
    Pan L, Yoshii Y, Otomo A, Ogawa H, Iwasaki Y, Shang HF, Hadano S.
    PLoS One; 2012 Oct 23; 7(3):e33409. PubMed ID: 22438926
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  • 10. Galectin-1 deficiency improves axonal swelling of motor neurones in SOD1(G93A) transgenic mice.
    Kobayakawa Y, Sakumi K, Kajitani K, Kadoya T, Horie H, Kira J, Nakabeppu Y.
    Neuropathol Appl Neurobiol; 2015 Feb 23; 41(2):227-44. PubMed ID: 24707896
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  • 12. Overexpression of Abeta is associated with acceleration of onset of motor impairment and superoxide dismutase 1 aggregation in an amyotrophic lateral sclerosis mouse model.
    Li QX, Mok SS, Laughton KM, McLean CA, Volitakis I, Cherny RA, Cheung NS, White AR, Masters CL.
    Aging Cell; 2006 Apr 23; 5(2):153-65. PubMed ID: 16626394
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  • 13. Novel behavioural characteristics of the superoxide dismutase 1 G93A (SOD1G93A ) mouse model of amyotrophic lateral sclerosis include sex-dependent phenotypes.
    Kreilaus F, Guerra S, Masanetz R, Menne V, Yerbury J, Karl T.
    Genes Brain Behav; 2020 Feb 23; 19(2):e12604. PubMed ID: 31412164
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  • 15. Glycine receptor channels in spinal motoneurons are abnormal in a transgenic mouse model of amyotrophic lateral sclerosis.
    Chang Q, Martin LJ.
    J Neurosci; 2011 Feb 23; 31(8):2815-27. PubMed ID: 21414903
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  • 16. Transcriptomic indices of fast and slow disease progression in two mouse models of amyotrophic lateral sclerosis.
    Nardo G, Iennaco R, Fusi N, Heath PR, Marino M, Trolese MC, Ferraiuolo L, Lawrence N, Shaw PJ, Bendotti C.
    Brain; 2013 Nov 23; 136(Pt 11):3305-32. PubMed ID: 24065725
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  • 17. Rats expressing human cytosolic copper-zinc superoxide dismutase transgenes with amyotrophic lateral sclerosis: associated mutations develop motor neuron disease.
    Nagai M, Aoki M, Miyoshi I, Kato M, Pasinelli P, Kasai N, Brown RH, Itoyama Y.
    J Neurosci; 2001 Dec 01; 21(23):9246-54. PubMed ID: 11717358
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  • 18. Proteins that bind to misfolded mutant superoxide dismutase-1 in spinal cords from transgenic amyotrophic lateral sclerosis (ALS) model mice.
    Zetterström P, Graffmo KS, Andersen PM, Brännström T, Marklund SL.
    J Biol Chem; 2011 Jun 10; 286(23):20130-6. PubMed ID: 21493711
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  • 19. Mutant SOD1 prevents normal functional recovery through enhanced glial activation and loss of motor neuron innervation after peripheral nerve injury.
    Schram S, Chuang D, Schmidt G, Piponov H, Helder C, Kerns J, Gonzalez M, Song F, Loeb JA.
    Neurobiol Dis; 2019 Apr 10; 124():469-478. PubMed ID: 30594811
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  • 20. Trehalose decreases mutant SOD1 expression and alleviates motor deficiency in early but not end-stage amyotrophic lateral sclerosis in a SOD1-G93A mouse model.
    Li Y, Guo Y, Wang X, Yu X, Duan W, Hong K, Wang J, Han H, Li C.
    Neuroscience; 2015 Jul 09; 298():12-25. PubMed ID: 25841320
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