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7. Facilitated cross-bridge interactions with thin filaments by familial hypertrophic cardiomyopathy mutations in α-tropomyosin. Wang F, Brunet NM, Grubich JR, Bienkiewicz EA, Asbury TM, Compton LA, Mihajlović G, Miller VF, Chase PB. J Biomed Biotechnol; 2011; 2011():435271. PubMed ID: 22187526 [Abstract] [Full Text] [Related]
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