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135 related items for PubMed ID: 21376702
1. Hypertrophic cardiomyopathy-causing Asp175asn and Glu180gly Tpm1 mutations shift tropomyosin strands further towards the open position during the ATPase cycle. Borovikov YS, Rysev NA, Karpicheva OE, Redwood CS. Biochem Biophys Res Commun; 2011 Apr 01; 407(1):197-201. PubMed ID: 21376702 [Abstract] [Full Text] [Related]
2. The effect of the Asp175Asn and Glu180Gly TPM1 mutations on actin-myosin interaction during the ATPase cycle. Rysev NA, Karpicheva OE, Redwood CS, Borovikov YS. Biochim Biophys Acta; 2012 Feb 01; 1824(2):366-73. PubMed ID: 22155441 [Abstract] [Full Text] [Related]
7. Effect of hypertrophic cardiomyopathy mutations in human cardiac muscle alpha -tropomyosin (Asp175Asn and Glu180Gly) on the regulatory properties of human cardiac troponin determined by in vitro motility assay. Bing W, Knott A, Redwood C, Esposito G, Purcell I, Watkins H, Marston S. J Mol Cell Cardiol; 2000 Aug 01; 32(8):1489-98. PubMed ID: 10900175 [Abstract] [Full Text] [Related]
11. The nemaline myopathy-causing E117K mutation in β-tropomyosin reduces thin filament activation. Karpicheva OE, Robinson P, Piers A, Borovikov YS, Redwood CS. Arch Biochem Biophys; 2013 Aug 01; 536(1):25-30. PubMed ID: 23689010 [Abstract] [Full Text] [Related]
12. Myopathy-causing Q147P TPM2 mutation shifts tropomyosin strands further towards the open position and increases the proportion of strong-binding cross-bridges during the ATPase cycle. Karpicheva OE, Simonyan AO, Kuleva NV, Redwood CS, Borovikov YS. Biochim Biophys Acta; 2016 Mar 01; 1864(3):260-267. PubMed ID: 26708479 [Abstract] [Full Text] [Related]
13. The cMyBP-C HCM variant L348P enhances thin filament activation through an increased shift in tropomyosin position. Mun JY, Kensler RW, Harris SP, Craig R. J Mol Cell Cardiol; 2016 Feb 01; 91():141-7. PubMed ID: 26718724 [Abstract] [Full Text] [Related]