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315 related items for PubMed ID: 21486770

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  • 3. Dysregulation of persistent inward and outward currents in spinal motoneurons of symptomatic SOD1-G93A mice.
    Deutsch AJ, Elbasiouny SM.
    J Physiol; 2024 Aug; 602(15):3715-3736. PubMed ID: 38924530
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  • 4. Postnatal electrical and morphological abnormalities in lumbar motoneurons from transgenic mouse models of amyotrophic lateral sclerosis.
    Amendola J, Gueritaud JP, Lamotte d'Incamps B, Bories C, Liabeuf S, Allene C, Pambo-Pambo A, Durand J.
    Arch Ital Biol; 2007 Nov; 145(3-4):311-23. PubMed ID: 18075124
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  • 6. Depressed excitability and ion currents linked to slow exocytotic fusion pore in chromaffin cells of the SOD1(G93A) mouse model of amyotrophic lateral sclerosis.
    Calvo-Gallardo E, de Pascual R, Fernández-Morales JC, Arranz-Tagarro JA, Maroto M, Nanclares C, Gandía L, de Diego AM, Padín JF, García AG.
    Am J Physiol Cell Physiol; 2015 Jan 01; 308(1):C1-19. PubMed ID: 25377090
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  • 7. Accumulation of SOD1 mutants in postnatal motoneurons does not cause motoneuron pathology or motoneuron disease.
    Lino MM, Schneider C, Caroni P.
    J Neurosci; 2002 Jun 15; 22(12):4825-32. PubMed ID: 12077179
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  • 10. Hyperexcitability of cultured spinal motoneurons from presymptomatic ALS mice.
    Kuo JJ, Schonewille M, Siddique T, Schults AN, Fu R, Bär PR, Anelli R, Heckman CJ, Kroese AB.
    J Neurophysiol; 2004 Jan 15; 91(1):571-5. PubMed ID: 14523070
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  • 11. Motoneuron subtypes show specificity in glycine receptor channel abnormalities in a transgenic mouse model of amyotrophic lateral sclerosis.
    Chang Q, Martin LJ.
    Channels (Austin); 2011 Jan 15; 5(4):299-303. PubMed ID: 21558795
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  • 13. The vulnerability of spinal motoneurons and soma size plasticity in a mouse model of amyotrophic lateral sclerosis.
    Dukkipati SS, Garrett TL, Elbasiouny SM.
    J Physiol; 2018 May 01; 596(9):1723-1745. PubMed ID: 29502344
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  • 14. Spinal motoneurones are intrinsically more responsive in the adult G93A SOD1 mouse model of amyotrophic lateral sclerosis.
    Jensen DB, Kadlecova M, Allodi I, Meehan CF.
    J Physiol; 2020 Oct 01; 598(19):4385-4403. PubMed ID: 32716521
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  • 15. Early electrophysiological abnormalities in lumbar motoneurons in a transgenic mouse model of amyotrophic lateral sclerosis.
    Bories C, Amendola J, Lamotte d'Incamps B, Durand J.
    Eur J Neurosci; 2007 Jan 01; 25(2):451-9. PubMed ID: 17284186
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  • 17. Preferential potentiation of AMPA-mediated currents in brainstem hypoglossal motoneurons by subchronic exposure of mice expressing the human superoxide dismutase 1 G93A gene mutation to neurotoxicant methylmercury in vivo.
    Yuan Y, Bailey JM, Rivera-Lopez GM, Atchison WD.
    Neurotoxicology; 2024 Jan 01; 100():72-84. PubMed ID: 38065418
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  • 18. Excitatory action of low frequency depolarizing GABA/glycine synaptic inputs is prevalent in prenatal spinal SOD1G93A motoneurons.
    Zhu H, Dalvi U, Cazenave W, Cattaert D, Branchereau P.
    J Physiol; 2024 Mar 01; 602(5):913-932. PubMed ID: 38345477
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