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Journal Abstract Search

315 related items for PubMed ID: 21486770

  • 41. Transcriptomic indices of fast and slow disease progression in two mouse models of amyotrophic lateral sclerosis.
    Nardo G, Iennaco R, Fusi N, Heath PR, Marino M, Trolese MC, Ferraiuolo L, Lawrence N, Shaw PJ, Bendotti C.
    Brain; 2013 Nov; 136(Pt 11):3305-32. PubMed ID: 24065725
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  • 49. Mutant SOD1(G93A) microglia are more neurotoxic relative to wild-type microglia.
    Xiao Q, Zhao W, Beers DR, Yen AA, Xie W, Henkel JS, Appel SH.
    J Neurochem; 2007 Sep; 102(6):2008-2019. PubMed ID: 17555556
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  • 51. Glycinergic innervation of motoneurons is deficient in amyotrophic lateral sclerosis mice: a quantitative confocal analysis.
    Chang Q, Martin LJ.
    Am J Pathol; 2009 Feb; 174(2):574-85. PubMed ID: 19116365
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  • 52. Dismutase-competent SOD1 mutant accumulation in myelinating Schwann cells is not detrimental to normal or transgenic ALS model mice.
    Turner BJ, Ackerley S, Davies KE, Talbot K.
    Hum Mol Genet; 2010 Mar 01; 19(5):815-24. PubMed ID: 20008901
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  • 54. Inhibitory synaptic regulation of motoneurons: a new target of disease mechanisms in amyotrophic lateral sclerosis.
    Martin LJ, Chang Q.
    Mol Neurobiol; 2012 Feb 01; 45(1):30-42. PubMed ID: 22072396
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  • 57. Muscle cells and motoneurons differentially remove mutant SOD1 causing familial amyotrophic lateral sclerosis.
    Onesto E, Rusmini P, Crippa V, Ferri N, Zito A, Galbiati M, Poletti A.
    J Neurochem; 2011 Jul 01; 118(2):266-80. PubMed ID: 21554318
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  • 58. Voltage-dependent sodium channels in spinal cord motor neurons display rapid recovery from fast inactivation in a mouse model of amyotrophic lateral sclerosis.
    Zona C, Pieri M, Carunchio I.
    J Neurophysiol; 2006 Dec 01; 96(6):3314-22. PubMed ID: 16899637
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  • 59. Low Ca2+ buffering in hypoglossal motoneurons of mutant SOD1 (G93A) mice.
    von Lewinski F, Fuchs J, Vanselow BK, Keller BU.
    Neurosci Lett; 2008 Nov 21; 445(3):224-8. PubMed ID: 18782598
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  • 60. Trehalose decreases mutant SOD1 expression and alleviates motor deficiency in early but not end-stage amyotrophic lateral sclerosis in a SOD1-G93A mouse model.
    Li Y, Guo Y, Wang X, Yu X, Duan W, Hong K, Wang J, Han H, Li C.
    Neuroscience; 2015 Jul 09; 298():12-25. PubMed ID: 25841320
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