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Journal Abstract Search


129 related items for PubMed ID: 2158404

  • 1. Selective depression of maximum rate of depolarization of guinea-pig ventricular action potentials by amiodarone and lignocaine in simulated ischaemia: comparison with encainide.
    Campbell TJ, Hemsworth PD.
    Clin Exp Pharmacol Physiol; 1990 Feb; 17(2):135-45. PubMed ID: 2158404
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  • 2. Effects of hyperkalemia, acidosis, and hypoxia on the depression of maximum rate of depolarization by class I antiarrhythmic drugs in guinea pig myocardium: differential actions of class Ib and Ic agents.
    Campbell TJ, Wyse KR, Hemsworth PD.
    J Cardiovasc Pharmacol; 1991 Jul; 18(1):51-9. PubMed ID: 1719292
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  • 3. Block of cardiac sodium channels by amiodarone studied by using Vmax of action potential in single ventricular myocytes.
    Honjo H, Kodama I, Kamiya K, Toyama J.
    Br J Pharmacol; 1991 Mar; 102(3):651-6. PubMed ID: 1364834
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  • 4. Resting and rate-dependent depression of maximum rate of depolarisation (Vmax) in guinea pig ventricular action potentials by mexiletine, disopyramide, and encainide.
    Campbell TJ.
    J Cardiovasc Pharmacol; 1983 Mar; 5(2):291-6. PubMed ID: 6188905
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  • 16. Antifibrillatory efficacy of encainide, loracainide and ORG 6001 compared with lignocaine in isolated hearts of rabbits and guinea-pigs.
    Almotrefi AA, Baker JB.
    Br J Pharmacol; 1981 Jun; 73(2):373-7. PubMed ID: 6786403
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  • 17. Supersensitivity to tetrodotoxin and lignocaine of sea anemone toxin II-treated sodium channel in guinea-pig ventricular muscle.
    Nishio M, Ohmura T, Kigoshi S, Muramatsu I.
    Br J Pharmacol; 1991 Oct; 104(2):504-8. PubMed ID: 1686737
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