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Journal Abstract Search

234 related items for PubMed ID: 2158884

  • 1. Cholera toxin differentially decreases membrane levels of alpha and beta subunits of G proteins in NG108-15 cells.
    Klinz FJ, Costa T.
    Eur J Biochem; 1990 Mar 30; 188(3):567-76. PubMed ID: 2158884
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  • 2. Identification of three separate guanine nucleotide-binding proteins that interact with the delta-opioid receptor in NG108-15 neuroblastoma x glioma hybrid cells.
    Roerig SC, Loh HH, Law PY.
    Mol Pharmacol; 1992 May 30; 41(5):822-31. PubMed ID: 1317000
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  • 3. Cholera toxin impairment of opioid-mediated inhibition of adenylate cyclase in neuroblastoma x glioma hybrid cells is due to a toxin-induced decrease in opioid receptor levels.
    McKenzie FR, Milligan G.
    Biochem J; 1991 Apr 01; 275 ( Pt 1)(Pt 1):175-81. PubMed ID: 1673334
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  • 4. Biphasic regulation of adenylate cyclase by cholera toxin in neuroblastoma x glioma hybrid cells is due to the activation and subsequent loss of the alpha subunit of the stimulatory GTP binding protein (GS).
    Macleod KG, Milligan G.
    Cell Signal; 1990 Apr 01; 2(2):139-51. PubMed ID: 2119204
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  • 5. Functional modification by cholera-toxin-catalyzed ADP-ribosylation of a guanine-nucleotide-binding regulatory protein serving as the substrate of pertussis toxin.
    Iiri T, Ohoka Y, Ui M, Katada T.
    Eur J Biochem; 1991 Dec 05; 202(2):635-41. PubMed ID: 1662135
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  • 10. Determination of G-protein levels, ADP-ribosylation by cholera and pertussis toxins and the regulation of adenylyl cyclase activity in liver plasma membranes from lean and genetically diabetic (db/db) mice.
    Palmer TM, Houslay MD.
    Biochim Biophys Acta; 1991 Oct 21; 1097(3):193-204. PubMed ID: 1932144
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  • 12. Pertussis toxin B-subunit-induced Ca2(+)-fluxes in Jurkat human lymphoma cells: the action of long-term pre-treatment with cholera and pertussis holotoxins.
    Sommermeyer H, Resch K.
    Cell Signal; 1990 Oct 21; 2(2):115-28. PubMed ID: 2169284
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  • 13. Cholera and pertussis toxins reveal multiple regulation of cAMP levels in the rabbit carotid body.
    Cachero TG, Rigual R, Rocher A, Gonzalez C.
    Eur J Neurosci; 1996 Nov 21; 8(11):2320-7. PubMed ID: 8950096
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  • 14. An arginine residue is the site of receptor-stimulated, cholera toxin-catalysed ADP-ribosylation of pertussis toxin-sensitive G-proteins.
    Milligan G, Mitchell FM.
    Cell Signal; 1993 Jul 21; 5(4):485-93. PubMed ID: 8396964
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  • 17. Cholera toxin ADP-ribosylates the receptor-coupled form of pertussis toxin-sensitive G-proteins.
    Klinz FJ, Costa T.
    Biochem Biophys Res Commun; 1989 Dec 15; 165(2):554-60. PubMed ID: 2557017
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  • 19. Gs alpha is a substrate for mono(ADP-ribosyl)transferase of NG108-15 cells. ADP-ribosylation regulates Gs alpha activity and abundance.
    Donnelly LE, Boyd RS, MacDermot J.
    Biochem J; 1992 Nov 15; 288 ( Pt 1)(Pt 1):331-6. PubMed ID: 1280114
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  • 20. Stimulatory guanine nucleotide binding protein in pig epidermis: transient increase of the 45KDA cholera toxin substrate (Gs alpha) in the tape stripping-induced hyperproliferative state.
    Tsutsui M, Tamura T, Takahashi H, Hashimoto Y, Iizuka H.
    Epithelial Cell Biol; 1994 Nov 15; 3(4):161-7. PubMed ID: 7550608
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