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Journal Abstract Search


454 related items for PubMed ID: 21624135

  • 1. Histone deacetylase turnover and recovery in sulforaphane-treated colon cancer cells: competing actions of 14-3-3 and Pin1 in HDAC3/SMRT corepressor complex dissociation/reassembly.
    Rajendran P, Delage B, Dashwood WM, Yu TW, Wuth B, Williams DE, Ho E, Dashwood RH.
    Mol Cancer; 2011 May 30; 10():68. PubMed ID: 21624135
    [Abstract] [Full Text] [Related]

  • 2. Differential effects of sulforaphane on histone deacetylases, cell cycle arrest and apoptosis in normal prostate cells versus hyperplastic and cancerous prostate cells.
    Clarke JD, Hsu A, Yu Z, Dashwood RH, Ho E.
    Mol Nutr Food Res; 2011 Jul 30; 55(7):999-1009. PubMed ID: 21374800
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  • 3. A novel mechanism of chemoprotection by sulforaphane: inhibition of histone deacetylase.
    Myzak MC, Karplus PA, Chung FL, Dashwood RH.
    Cancer Res; 2004 Aug 15; 64(16):5767-74. PubMed ID: 15313918
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  • 4. HDAC turnover, CtIP acetylation and dysregulated DNA damage signaling in colon cancer cells treated with sulforaphane and related dietary isothiocyanates.
    Rajendran P, Kidane AI, Yu TW, Dashwood WM, Bisson WH, Löhr CV, Ho E, Williams DE, Dashwood RH.
    Epigenetics; 2013 Jun 15; 8(6):612-23. PubMed ID: 23770684
    [Abstract] [Full Text] [Related]

  • 5. Heterocyclic Analogs of Sulforaphane Trigger DNA Damage and Impede DNA Repair in Colon Cancer Cells: Interplay of HATs and HDACs.
    Okonkwo A, Mitra J, Johnson GS, Li L, Dashwood WM, Hegde ML, Yue C, Dashwood RH, Rajendran P.
    Mol Nutr Food Res; 2018 Sep 15; 62(18):e1800228. PubMed ID: 29924908
    [Abstract] [Full Text] [Related]

  • 6. In cortical neurons HDAC3 activity suppresses RD4-dependent SMRT export.
    Soriano FX, Hardingham GE.
    PLoS One; 2011 Sep 15; 6(6):e21056. PubMed ID: 21695276
    [Abstract] [Full Text] [Related]

  • 7. D,L-sulforaphane-induced apoptosis in human breast cancer cells is regulated by the adapter protein p66Shc.
    Sakao K, Singh SV.
    J Cell Biochem; 2012 Feb 15; 113(2):599-610. PubMed ID: 21956685
    [Abstract] [Full Text] [Related]

  • 8. Involvement of c-Jun N-terminal kinase in G2/M arrest and caspase-mediated apoptosis induced by sulforaphane in DU145 prostate cancer cells.
    Cho SD, Li G, Hu H, Jiang C, Kang KS, Lee YS, Kim SH, Lu J.
    Nutr Cancer; 2005 Feb 15; 52(2):213-24. PubMed ID: 16201852
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  • 15. Sulforaphane-induced G2/M phase cell cycle arrest involves checkpoint kinase 2-mediated phosphorylation of cell division cycle 25C.
    Singh SV, Herman-Antosiewicz A, Singh AV, Lew KL, Srivastava SK, Kamath R, Brown KD, Zhang L, Baskaran R.
    J Biol Chem; 2004 Jun 11; 279(24):25813-22. PubMed ID: 15073169
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  • 16. Formation of AR-SMRT binding in prostate cancer cells treated with natural histone deacetylase inhibitor.
    Trtková K, Pašková L, Matiješčuková N, Kolář Z.
    Cancer Biomark; 2010 Jun 11; 7(2):79-90. PubMed ID: 21178266
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  • 17. Activation of p53 transcriptional activity by SMRT: a histone deacetylase 3-independent function of a transcriptional corepressor.
    Adikesavan AK, Karmakar S, Pardo P, Wang L, Liu S, Li W, Smith CL.
    Mol Cell Biol; 2014 Apr 11; 34(7):1246-61. PubMed ID: 24449765
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  • 18. Targeting cell cycle machinery as a molecular mechanism of sulforaphane in prostate cancer prevention.
    Wang L, Liu D, Ahmed T, Chung FL, Conaway C, Chiao JW.
    Int J Oncol; 2004 Jan 11; 24(1):187-92. PubMed ID: 14654956
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  • 19. Dose-dependent effects of R-sulforaphane isothiocyanate on the biology of human mesenchymal stem cells, at dietary amounts, it promotes cell proliferation and reduces senescence and apoptosis, while at anti-cancer drug doses, it has a cytotoxic effect.
    Zanichelli F, Capasso S, Cipollaro M, Pagnotta E, Cartenì M, Casale F, Iori R, Galderisi U.
    Age (Dordr); 2012 Apr 11; 34(2):281-93. PubMed ID: 21465338
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