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73 related items for PubMed ID: 2167088

  • 21. Mutant Phe788 --> Leu of the Na+,K+-ATPase is inhibited by micromolar concentrations of potassium and exhibits high Na+-ATPase activity at low sodium concentrations.
    Vilsen B.
    Biochemistry; 1999 Aug 31; 38(35):11389-400. PubMed ID: 10471289
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  • 22. Consequences of mutations to the phosphorylation site of the alpha-subunit of Na, K-ATPase for ATP binding and E1-E2 conformational equilibrium.
    Pedersen PA, Rasmussen JH, Jørgensen PL.
    Biochemistry; 1996 Dec 17; 35(50):16085-93. PubMed ID: 8973179
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  • 23. New evidence for ATP binding induced catalytic subunit interactions in pig kidney Na/K-ATPase.
    Tanoue K, Kaya S, Hayashi Y, Abe K, Imagawa T, Taniguchi K, Sakaguchi K.
    J Biochem; 2006 Oct 17; 140(4):599-607. PubMed ID: 16987945
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  • 24. Na,K-ATPase structure/function relationships probed by the denaturant urea.
    Esmann M, Fedosova NU, Olesen C.
    Biochim Biophys Acta; 2015 May 17; 1848(5):1212-23. PubMed ID: 25687971
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  • 30. Differences between CTP and ATP as substrates for the (Na + K)-ATPase.
    Robinson JD.
    Arch Biochem Biophys; 1982 Feb 17; 213(2):650-7. PubMed ID: 6280617
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  • 34. Unexpected Effects of K+ and Adenosine Triphosphate on the Thermal Stability of Na+,K+-ATPase.
    Placenti MA, Kaufman SB, González Flecha FL, González Lebrero RM.
    J Phys Chem B; 2017 May 18; 121(19):4949-4957. PubMed ID: 28441865
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  • 40. Na+, K+-dependent adenosine triphosphate phosphohydrolase. Two types of kinetics.
    Boldyrev AA, Svinukhova IA.
    Biochim Biophys Acta; 1982 Sep 22; 707(1):167-70. PubMed ID: 6291616
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