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Journal Abstract Search

95 related items for PubMed ID: 21762452

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  • 2. Scn2a sodium channel mutation results in hyperexcitability in the hippocampus in vitro.
    Kile KB, Tian N, Durand DM.
    Epilepsia; 2008 Mar; 49(3):488-99. PubMed ID: 18031550
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  • 3. Cacna1g is a genetic modifier of epilepsy caused by mutation of voltage-gated sodium channel Scn2a.
    Calhoun JD, Hawkins NA, Zachwieja NJ, Kearney JA.
    Epilepsia; 2016 Jun; 57(6):e103-7. PubMed ID: 27112236
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  • 4. Hippocampal neurodegeneration, spontaneous seizures, and mossy fiber sprouting in the F344 rat model of temporal lobe epilepsy.
    Rao MS, Hattiangady B, Reddy DS, Shetty AK.
    J Neurosci Res; 2006 May 01; 83(6):1088-105. PubMed ID: 16493685
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  • 6. CaMKII modulates sodium current in neurons from epileptic Scn2a mutant mice.
    Thompson CH, Hawkins NA, Kearney JA, George AL.
    Proc Natl Acad Sci U S A; 2017 Feb 14; 114(7):1696-1701. PubMed ID: 28137877
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  • 7. Epileptogenesis and chronic seizures in a mouse model of temporal lobe epilepsy are associated with distinct EEG patterns and selective neurochemical alterations in the contralateral hippocampus.
    Arabadzisz D, Antal K, Parpan F, Emri Z, Fritschy JM.
    Exp Neurol; 2005 Jul 14; 194(1):76-90. PubMed ID: 15899245
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  • 11. Temporal lobe epilepsy and GEFS+ phenotypes associated with SCN1B mutations.
    Scheffer IE, Harkin LA, Grinton BE, Dibbens LM, Turner SJ, Zielinski MA, Xu R, Jackson G, Adams J, Connellan M, Petrou S, Wellard RM, Briellmann RS, Wallace RH, Mulley JC, Berkovic SF.
    Brain; 2007 Jan 14; 130(Pt 1):100-9. PubMed ID: 17020904
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  • 15. Spontaneous epileptiform discharges in a mouse model of Alzheimer's disease are suppressed by antiepileptic drugs that block sodium channels.
    Ziyatdinova S, Gurevicius K, Kutchiashvili N, Bolkvadze T, Nissinen J, Tanila H, Pitkänen A.
    Epilepsy Res; 2011 Mar 14; 94(1-2):75-85. PubMed ID: 21300523
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  • 19. Low frequency stimulation decreases seizure activity in a mutation model of epilepsy.
    Kile KB, Tian N, Durand DM.
    Epilepsia; 2010 Sep 14; 51(9):1745-53. PubMed ID: 20659150
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  • 20. Epileptic tolerance is associated with enduring neuroprotection and uncoupling of the relationship between CA3 damage, neuropeptide Y rearrangement and spontaneous seizures following intra-amygdala kainic acid-induced status epilepticus in mice.
    Jimenez-Mateos EM, Mouri G, Conroy RM, Henshall DC.
    Neuroscience; 2010 Dec 01; 171(2):556-65. PubMed ID: 20837105
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