These tools will no longer be maintained as of December 31, 2024. Archived website can be found here. PubMed4Hh GitHub repository can be found here. Contact NLM Customer Service if you have questions.


Pubmed for Handhelds

PUBMED FOR HANDHELDS

Journal Abstract Search

162 related items for PubMed ID: 21768089

  • 1. Loss of granuphilin and loss of syntaxin-1A cause differential effects on insulin granule docking and fusion.
    Wang H, Ishizaki R, Kobayashi E, Fujiwara T, Akagawa K, Izumi T.
    J Biol Chem; 2011 Sep 16; 286(37):32244-50. PubMed ID: 21768089
    [Abstract] [Full Text] [Related]

  • 2. The Rab27a effector exophilin7 promotes fusion of secretory granules that have not been docked to the plasma membrane.
    Wang H, Ishizaki R, Xu J, Kasai K, Kobayashi E, Gomi H, Izumi T.
    Mol Biol Cell; 2013 Feb 16; 24(3):319-30. PubMed ID: 23223571
    [Abstract] [Full Text] [Related]

  • 3. Granuphilin molecularly docks insulin granules to the fusion machinery.
    Gomi H, Mizutani S, Kasai K, Itohara S, Izumi T.
    J Cell Biol; 2005 Oct 10; 171(1):99-109. PubMed ID: 16216924
    [Abstract] [Full Text] [Related]

  • 4. Slp4-a/granuphilin-a interacts with syntaxin-2/3 in a Munc18-2-dependent manner.
    Fukuda M, Imai A, Nashida T, Shimomura H.
    J Biol Chem; 2005 Nov 25; 280(47):39175-84. PubMed ID: 16186111
    [Abstract] [Full Text] [Related]

  • 5. Rab27 effector granuphilin promotes the plasma membrane targeting of insulin granules via interaction with syntaxin 1a.
    Torii S, Takeuchi T, Nagamatsu S, Izumi T.
    J Biol Chem; 2004 May 21; 279(21):22532-8. PubMed ID: 15028737
    [Abstract] [Full Text] [Related]

  • 6. Munc 18-1 and granuphilin collaborate during insulin granule exocytosis.
    Tomas A, Meda P, Regazzi R, Pessin JE, Halban PA.
    Traffic; 2008 May 21; 9(5):813-32. PubMed ID: 18208509
    [Abstract] [Full Text] [Related]

  • 7. Granuphilin modulates the exocytosis of secretory granules through interaction with syntaxin 1a.
    Torii S, Zhao S, Yi Z, Takeuchi T, Izumi T.
    Mol Cell Biol; 2002 Aug 21; 22(15):5518-26. PubMed ID: 12101244
    [Abstract] [Full Text] [Related]

  • 8. Granuphilin exclusively mediates functional granule docking to the plasma membrane.
    Mizuno K, Fujita T, Gomi H, Izumi T.
    Sci Rep; 2016 Apr 01; 6():23909. PubMed ID: 27032672
    [Abstract] [Full Text] [Related]

  • 9. Munc18-1 regulates first-phase insulin release by promoting granule docking to multiple syntaxin isoforms.
    Oh E, Kalwat MA, Kim MJ, Verhage M, Thurmond DC.
    J Biol Chem; 2012 Jul 27; 287(31):25821-33. PubMed ID: 22685295
    [Abstract] [Full Text] [Related]

  • 10. Munc13b stimulus-dependently accumulates on granuphilin-mediated, docked granules prior to fusion.
    Mizuno K, Izumi T.
    Cell Struct Funct; 2022 May 21; 47(1):31-41. PubMed ID: 35387942
    [Abstract] [Full Text] [Related]

  • 11. Docking is not a prerequisite but a temporal constraint for fusion of secretory granules.
    Kasai K, Fujita T, Gomi H, Izumi T.
    Traffic; 2008 Jul 21; 9(7):1191-203. PubMed ID: 18397364
    [Abstract] [Full Text] [Related]

  • 12. Snapin mediates insulin secretory granule docking, but not trans-SNARE complex formation.
    Somanath S, Partridge CJ, Marshall C, Rowe T, Turner MD.
    Biochem Biophys Res Commun; 2016 Apr 29; 473(2):403-7. PubMed ID: 26946359
    [Abstract] [Full Text] [Related]

  • 13. The Slp4-a linker domain controls exocytosis through interaction with Munc18-1.syntaxin-1a complex.
    Tsuboi T, Fukuda M.
    Mol Biol Cell; 2006 May 29; 17(5):2101-12. PubMed ID: 16481396
    [Abstract] [Full Text] [Related]

  • 14. Syntaxin 2 Acts as Inhibitory SNARE for Insulin Granule Exocytosis.
    Zhu D, Xie L, Kang Y, Dolai S, Bondo Hansen J, Qin T, Xie H, Liang T, Rubin DC, Osborne L, Gaisano HY.
    Diabetes; 2017 Apr 29; 66(4):948-959. PubMed ID: 28115395
    [Abstract] [Full Text] [Related]

  • 15. Contact-induced clustering of syntaxin and munc18 docks secretory granules at the exocytosis site.
    Gandasi NR, Barg S.
    Nat Commun; 2014 May 19; 5():3914. PubMed ID: 24835618
    [Abstract] [Full Text] [Related]

  • 16. Synaptotagmin-1 docks secretory vesicles to syntaxin-1/SNAP-25 acceptor complexes.
    de Wit H, Walter AM, Milosevic I, Gulyás-Kovács A, Riedel D, Sørensen JB, Verhage M.
    Cell; 2009 Sep 04; 138(5):935-46. PubMed ID: 19716167
    [Abstract] [Full Text] [Related]

  • 17. Munc18b is a major mediator of insulin exocytosis in rat pancreatic β-cells.
    Lam PP, Ohno M, Dolai S, He Y, Qin T, Liang T, Zhu D, Kang Y, Liu Y, Kauppi M, Xie L, Wan WC, Bin NR, Sugita S, Olkkonen VM, Takahashi N, Kasai H, Gaisano HY.
    Diabetes; 2013 Jul 04; 62(7):2416-28. PubMed ID: 23423569
    [Abstract] [Full Text] [Related]

  • 18. Extension of Helix 12 in Munc18-1 Induces Vesicle Priming.
    Munch AS, Kedar GH, van Weering JR, Vazquez-Sanchez S, He E, André T, Braun T, Söllner TH, Verhage M, Sørensen JB.
    J Neurosci; 2016 Jun 29; 36(26):6881-91. PubMed ID: 27358447
    [Abstract] [Full Text] [Related]

  • 19. Functional analysis of Rab27a effector granuphilin in insulin exocytosis.
    Izumi T, Gomi H, Torii S.
    Methods Enzymol; 2005 Jun 29; 403():216-29. PubMed ID: 16473589
    [Abstract] [Full Text] [Related]

  • 20. Munc18-2 regulates exocytotic membrane fusion positively interacting with syntaxin-3 in RBL-2H3 cells.
    Tadokoro S, Kurimoto T, Nakanishi M, Hirashima N.
    Mol Immunol; 2007 Jul 29; 44(13):3427-33. PubMed ID: 17408745
    [Abstract] [Full Text] [Related]

    Page: [Next] [New Search]
    of 9.