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150 related items for PubMed ID: 21773967

  • 1. High prevalence of TSHR/Gsα mutation-negative clonal hot thyroid nodules (HNs) in a Turkish cohort.
    Sancak S, Jaeschke H, Eren F, Tarcin O, Guellueoglu B, Sen LS, Sever Z, Gozu HI, Bircan R, Akalin S, Paschke R, Eszlinger M.
    Horm Metab Res; 2011 Jul; 43(8):562-8. PubMed ID: 21773967
    [Abstract] [Full Text] [Related]

  • 2. Similar prevalence of somatic TSH receptor and Gsalpha mutations in toxic thyroid nodules in geographical regions with different iodine supply in Turkey.
    Gozu HI, Bircan R, Krohn K, Müller S, Vural S, Gezen C, Sargin H, Yavuzer D, Sargin M, Cirakoglu B, Paschke R.
    Eur J Endocrinol; 2006 Oct; 155(4):535-45. PubMed ID: 16990652
    [Abstract] [Full Text] [Related]

  • 3. Detection of thyroid-stimulating hormone receptor and Gsalpha mutations: in 75 toxic thyroid nodules by denaturing gradient gel electrophoresis.
    Trülzsch B, Krohn K, Wonerow P, Chey S, Holzapfel HP, Ackermann F, Führer D, Paschke R.
    J Mol Med (Berl); 2001 Oct; 78(12):684-91. PubMed ID: 11434721
    [Abstract] [Full Text] [Related]

  • 4. Lack of mutations in the TSHr and Gsalpha genes in TSHr antibody negative Graves' disease.
    Elgadi A, Frisk T, Larsson C, Wallin G, Höög A, Zedenius J, Norgren S.
    Exp Clin Endocrinol Diabetes; 2005 Oct; 113(9):516-21. PubMed ID: 16235153
    [Abstract] [Full Text] [Related]

  • 5. Somatic mutations in thyroid nodular disease.
    Krohn K, Paschke R.
    Mol Genet Metab; 2002 Mar; 75(3):202-8. PubMed ID: 11914031
    [Abstract] [Full Text] [Related]

  • 6. Prevalence of TSH receptor and Gsalpha mutations in 45 autonomously functioning thyroid nodules in Japan.
    Nishihara E, Amino N, Maekawa K, Yoshida H, Ito M, Kubota S, Fukata S, Miyauchi A.
    Endocr J; 2009 Mar; 56(6):791-8. PubMed ID: 19550078
    [Abstract] [Full Text] [Related]

  • 7. Hyperfunctioning thyroid nodules in toxic multinodular goiter share activating thyrotropin receptor mutations with solitary toxic adenoma.
    Tonacchera M, Chiovato L, Pinchera A, Agretti P, Fiore E, Cetani F, Rocchi R, Viacava P, Miccoli P, Vitti P.
    J Clin Endocrinol Metab; 1998 Feb; 83(2):492-8. PubMed ID: 9467563
    [Abstract] [Full Text] [Related]

  • 8. Somatic mutations in the thyrotropin receptor gene and not in the Gs alpha protein gene in 31 toxic thyroid nodules.
    Führer D, Holzapfel HP, Wonerow P, Scherbaum WA, Paschke R.
    J Clin Endocrinol Metab; 1997 Nov; 82(11):3885-91. PubMed ID: 9360556
    [Abstract] [Full Text] [Related]

  • 9. Identification of constitutively activating somatic thyrotropin receptor mutations in a subset of toxic multinodular goiters.
    Holzapfel HP, Führer D, Wonerow P, Weinland G, Scherbaum WA, Paschke R.
    J Clin Endocrinol Metab; 1997 Dec; 82(12):4229-33. PubMed ID: 9398745
    [Abstract] [Full Text] [Related]

  • 10. Non-hyperfunctioning nodules from multinodular goiters: a minor role in pathogenesis for somatic activating mutations in the TSH-receptor and Gsalpha subunit genes.
    Derrien C, Sonnet E, Gicquel I, Le Gall JY, Poirier JY, David V, Maugendre D.
    J Endocrinol Invest; 2001 May; 24(5):321-5. PubMed ID: 11407651
    [Abstract] [Full Text] [Related]

  • 11. Clonal origin of toxic thyroid nodules with constitutively activating thyrotropin receptor mutations.
    Krohn K, Führer D, Holzapfel HP, Paschke R.
    J Clin Endocrinol Metab; 1998 Jan; 83(1):130-4. PubMed ID: 9435429
    [Abstract] [Full Text] [Related]

  • 12. Mutations in the thyrotropin receptor signal transduction pathway in the hyperfunctioning thyroid nodules from multinodular goiters: a study in the Turkish population.
    Gozu H, Avsar M, Bircan R, Sahin S, Deyneli O, Cirakoglu B, Akalin S.
    Endocr J; 2005 Oct; 52(5):577-85. PubMed ID: 16284436
    [Abstract] [Full Text] [Related]

  • 13. Activating thyrotropin receptor mutations are present in nonadenomatous hyperfunctioning nodules of toxic or autonomous multinodular goiter.
    Tonacchera M, Agretti P, Chiovato L, Rosellini V, Ceccarini G, Perri A, Viacava P, Naccarato AG, Miccoli P, Pinchera A, Vitti P.
    J Clin Endocrinol Metab; 2000 Jun; 85(6):2270-4. PubMed ID: 10852462
    [Abstract] [Full Text] [Related]

  • 14. Sensitive Sequencing Analysis Suggests Thyrotropin Receptor and Guanine Nucleotide-Binding Protein G Subunit Alpha as Sole Driver Mutations in Hot Thyroid Nodules.
    Stephenson A, Eszlinger M, Stewardson P, McIntyre JB, Boesenberg E, Bircan R, Sancak S, Gozu HI, Ghaznavi S, Krohn K, Paschke R.
    Thyroid; 2020 Oct; 30(10):1482-1489. PubMed ID: 32284013
    [Abstract] [Full Text] [Related]

  • 15. Molecular aspects of the pathogenesis of nodular goiters, thyroid nodules and adenomas.
    Derwahl M.
    Exp Clin Endocrinol Diabetes; 1996 Oct; 104 Suppl 4():32-5. PubMed ID: 8980997
    [Abstract] [Full Text] [Related]

  • 16. Activating thyrotropin receptor mutations in histologically heterogeneous hyperfunctioning nodules of multinodular goiter.
    Tonacchera M, Vitti P, Agretti P, Giulianetti B, Mazzi B, Cavaliere R, Ceccarini G, Fiore E, Viacava P, Naccarato A, Pinchera A, Chiovato L.
    Thyroid; 1998 Jul; 8(7):559-64. PubMed ID: 9709907
    [Abstract] [Full Text] [Related]

  • 17. Functioning and nonfunctioning thyroid adenomas involve different molecular pathogenetic mechanisms.
    Tonacchera M, Vitti P, Agretti P, Ceccarini G, Perri A, Cavaliere R, Mazzi B, Naccarato AG, Viacava P, Miccoli P, Pinchera A, Chiovato L.
    J Clin Endocrinol Metab; 1999 Nov; 84(11):4155-8. PubMed ID: 10566665
    [Abstract] [Full Text] [Related]

  • 18. Ras mutations are rare in solitary cold and toxic thyroid nodules.
    Krohn K, Reske A, Ackermann F, Müller A, Paschke R.
    Clin Endocrinol (Oxf); 2001 Aug; 55(2):241-8. PubMed ID: 11531932
    [Abstract] [Full Text] [Related]

  • 19. Diversity and prevalence of somatic mutations in the thyrotropin receptor and Gs alpha genes as a cause of toxic thyroid adenomas.
    Parma J, Duprez L, Van Sande J, Hermans J, Rocmans P, Van Vliet G, Costagliola S, Rodien P, Dumont JE, Vassart G.
    J Clin Endocrinol Metab; 1997 Aug; 82(8):2695-701. PubMed ID: 9253356
    [Abstract] [Full Text] [Related]

  • 20. Constitutively activating TSH receptor mutations as the cause of toxic thyroid adenoma, multinodular toxic goiter and autosomal dominant non autoimmune hyperthyroidism.
    Paschke R.
    Exp Clin Endocrinol Diabetes; 1996 Aug; 104 Suppl 4():129-32. PubMed ID: 8981020
    [Abstract] [Full Text] [Related]

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