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Journal Abstract Search
658 related items for PubMed ID: 21834058
1. Stress signaling from the endoplasmic reticulum: A central player in the pathogenesis of amyotrophic lateral sclerosis. Walker AK, Atkin JD. IUBMB Life; 2011 Sep; 63(9):754-63. PubMed ID: 21834058 [Abstract] [Full Text] [Related]
4. Rab1-dependent ER-Golgi transport dysfunction is a common pathogenic mechanism in SOD1, TDP-43 and FUS-associated ALS. Soo KY, Halloran M, Sundaramoorthy V, Parakh S, Toth RP, Southam KA, McLean CA, Lock P, King A, Farg MA, Atkin JD. Acta Neuropathol; 2015 Nov; 130(5):679-97. PubMed ID: 26298469 [Abstract] [Full Text] [Related]
18. The role of heat shock proteins in Amyotrophic Lateral Sclerosis: The therapeutic potential of Arimoclomol. Kalmar B, Lu CH, Greensmith L. Pharmacol Ther; 2014 Jan 15; 141(1):40-54. PubMed ID: 23978556 [Abstract] [Full Text] [Related]
19. Mutant SOD1 inhibits ER-Golgi transport in amyotrophic lateral sclerosis. Atkin JD, Farg MA, Soo KY, Walker AK, Halloran M, Turner BJ, Nagley P, Horne MK. J Neurochem; 2014 Apr 15; 129(1):190-204. PubMed ID: 24134191 [Abstract] [Full Text] [Related]
20. Derlin-1 overexpression ameliorates mutant SOD1-induced endoplasmic reticulum stress by reducing mutant SOD1 accumulation. Mori A, Yamashita S, Uchino K, Suga T, Ikeda T, Takamatsu K, Ishizaki M, Koide T, Kimura E, Mita S, Maeda Y, Hirano T, Uchino M. Neurochem Int; 2011 Feb 15; 58(3):344-53. PubMed ID: 21185345 [Abstract] [Full Text] [Related] Page: [Next] [New Search]