These tools will no longer be maintained as of December 31, 2024. Archived website can be found here. PubMed4Hh GitHub repository can be found here. Contact NLM Customer Service if you have questions.


Pubmed for Handhelds

PUBMED FOR HANDHELDS

Journal Abstract Search

235 related items for PubMed ID: 21839364

  • 21. The podocyte as a direct target of immunosuppressive agents.
    Schönenberger E, Ehrich JH, Haller H, Schiffer M.
    Nephrol Dial Transplant; 2011 Jan; 26(1):18-24. PubMed ID: 20937691
    [Abstract] [Full Text] [Related]

  • 22. Role of p38 mitogen-activated protein kinase activation in podocyte injury and proteinuria in experimental nephrotic syndrome.
    Koshikawa M, Mukoyama M, Mori K, Suganami T, Sawai K, Yoshioka T, Nagae T, Yokoi H, Kawachi H, Shimizu F, Sugawara A, Nakao K.
    J Am Soc Nephrol; 2005 Sep; 16(9):2690-701. PubMed ID: 15987752
    [Abstract] [Full Text] [Related]

  • 23. Minimal change disease: a dysregulation of the podocyte CD80-CTLA-4 axis?
    Cara-Fuentes G, Wasserfall CH, Wang H, Johnson RJ, Garin EH.
    Pediatr Nephrol; 2014 Dec; 29(12):2333-40. PubMed ID: 25017621
    [Abstract] [Full Text] [Related]

  • 24. The role of podocytes in glomerular pathobiology.
    Asanuma K, Mundel P.
    Clin Exp Nephrol; 2003 Dec; 7(4):255-9. PubMed ID: 14712353
    [Abstract] [Full Text] [Related]

  • 25. Approach to Diagnosis and Management of Primary Glomerular Diseases Due to Podocytopathies in Adults: Core Curriculum 2020.
    Ahn W, Bomback AS.
    Am J Kidney Dis; 2020 Jun; 75(6):955-964. PubMed ID: 32331832
    [Abstract] [Full Text] [Related]

  • 26. [The role of podocyte damage in the pathogenesis of glomerulosclerosis and possible repair mechanisms].
    Ronconi E, Mazzinghi B, Sagrinati C, Angelotti ML, Ballerini L, Parente E, Romagnani P, Lazzeri E, Lasagni L.
    G Ital Nefrol; 2009 Jun; 26(6):660-9. PubMed ID: 19918748
    [Abstract] [Full Text] [Related]

  • 27. Interaction of CD80 with Neph1: a potential mechanism of podocyte injury.
    Khullar B, Balyan R, Oswal N, Jain N, Sharma A, Abdin MZ, Bagga A, Bhatnagar S, Wadhwa N, Natchu UCM, George A, Rath S, Bal V, Sopory S.
    Clin Exp Nephrol; 2018 Jun; 22(3):508-516. PubMed ID: 29022109
    [Abstract] [Full Text] [Related]

  • 28. Minimal change disease in graft versus host disease: a podocyte response to the graft?
    Huskey J, Rivard C, Myint H, Lucia S, Smith M, Shimada M, Ishimoto T, Araya C, Garin EH, Johnson RJ.
    Clin Nephrol; 2013 Dec; 80(6):469-73. PubMed ID: 23006339
    [Abstract] [Full Text] [Related]

  • 29. [Pathogenesis and treatment of idiopathic nephrotic syndrome in adults].
    Sahali D, Audard V, Rémy P, Lang P.
    Nephrol Ther; 2012 Jun; 8(3):180-92. PubMed ID: 22425458
    [Abstract] [Full Text] [Related]

  • 30. Role of the immune system in the pathogenesis of idiopathic nephrotic syndrome.
    van den Berg JG, Weening JJ.
    Clin Sci (Lond); 2004 Aug; 107(2):125-36. PubMed ID: 15157184
    [Abstract] [Full Text] [Related]

  • 31. Pediatric nephrotic syndrome: from the simple to the complex.
    Lane JC, Kaskel FJ.
    Semin Nephrol; 2009 Jul; 29(4):389-98. PubMed ID: 19615560
    [Abstract] [Full Text] [Related]

  • 32. [Minimal change glomerulonephritis, diffuse mesangioproliferative glomerulonephritis and focal and segmental glomerular sclerosis and hyalinosis].
    Belovezhdov N.
    Vutr Boles; 1984 Jul; 23(4):42-6. PubMed ID: 6506660
    [Abstract] [Full Text] [Related]

  • 33. Podocytes are firmly attached to glomerular basement membrane in kidneys with heavy proteinuria.
    Lahdenkari AT, Lounatmaa K, Patrakka J, Holmberg C, Wartiovaara J, Kestilä M, Koskimies O, Jalanko H.
    J Am Soc Nephrol; 2004 Oct; 15(10):2611-8. PubMed ID: 15466265
    [Abstract] [Full Text] [Related]

  • 34. Case series: CTLA4-IgG1 therapy in minimal change disease and focal segmental glomerulosclerosis.
    Garin EH, Reiser J, Cara-Fuentes G, Wei C, Matar D, Wang H, Alachkar N, Johnson RJ.
    Pediatr Nephrol; 2015 Mar; 30(3):469-77. PubMed ID: 25239302
    [Abstract] [Full Text] [Related]

  • 35. [Nephrotic syndrome in childhood].
    Reusz G, Szabó A, Fekete A.
    Orv Hetil; 2006 Nov 26; 147(47):2251-60. PubMed ID: 17380687
    [Abstract] [Full Text] [Related]

  • 36. Modulation of podocyte phenotype in collapsing glomerulopathies.
    Barisoni L, Kopp JB.
    Microsc Res Tech; 2002 May 15; 57(4):254-62. PubMed ID: 12012394
    [Abstract] [Full Text] [Related]

  • 37. [The fate of podocytes in proteinuric nephropathies].
    Ortiz A, Marrón B, Ramos A.
    Nefrologia; 2002 May 15; 22(5):425-31. PubMed ID: 12497743
    [No Abstract] [Full Text] [Related]

  • 38. Glomerular anionic sites in minimal change nephrotic syndrome and focal segmental glomerulosclerosis.
    Kitano Y, Yoshikawa N, Nakamura H.
    Clin Nephrol; 1993 Oct 15; 40(4):199-204. PubMed ID: 8261675
    [Abstract] [Full Text] [Related]

  • 39. Podocyte hypertrophy, "adaptation," and "decompensation" associated with glomerular enlargement and glomerulosclerosis in the aging rat: prevention by calorie restriction.
    Wiggins JE, Goyal M, Sanden SK, Wharram BL, Shedden KA, Misek DE, Kuick RD, Wiggins RC.
    J Am Soc Nephrol; 2005 Oct 15; 16(10):2953-66. PubMed ID: 16120818
    [Abstract] [Full Text] [Related]

  • 40. Dendrin expression in glomerulogenesis and in human minimal change nephrotic syndrome.
    Dunér F, Patrakka J, Xiao Z, Larsson J, Vlamis-Gardikas A, Pettersson E, Tryggvason K, Hultenby K, Wernerson A.
    Nephrol Dial Transplant; 2008 Aug 15; 23(8):2504-11. PubMed ID: 18356187
    [Abstract] [Full Text] [Related]

    Page: [Previous] [Next] [New Search]
    of 12.