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PUBMED FOR HANDHELDS

Journal Abstract Search


508 related items for PubMed ID: 21839714

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  • 4. The Calcium-Dependent Protease Calpain-1 Links TRPC6 Activity to Podocyte Injury.
    Verheijden KAT, Sonneveld R, Bakker-van Bebber M, Wetzels JFM, van der Vlag J, Nijenhuis T.
    J Am Soc Nephrol; 2018 Aug; 29(8):2099-2109. PubMed ID: 29954830
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  • 5. TRPC6 mutations associated with focal segmental glomerulosclerosis cause constitutive activation of NFAT-dependent transcription.
    Schlöndorff J, Del Camino D, Carrasquillo R, Lacey V, Pollak MR.
    Am J Physiol Cell Physiol; 2009 Mar; 296(3):C558-69. PubMed ID: 19129465
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  • 8. Activation of NFAT signaling in podocytes causes glomerulosclerosis.
    Wang Y, Jarad G, Tripathi P, Pan M, Cunningham J, Martin DR, Liapis H, Miner JH, Chen F.
    J Am Soc Nephrol; 2010 Oct; 21(10):1657-66. PubMed ID: 20651158
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  • 9. Transient receptor potential channel 6 (TRPC6) protects podocytes during complement-mediated glomerular disease.
    Kistler AD, Singh G, Altintas MM, Yu H, Fernandez IC, Gu C, Wilson C, Srivastava SK, Dietrich A, Walz K, Kerjaschki D, Ruiz P, Dryer S, Sever S, Dinda AK, Faul C, Reiser J.
    J Biol Chem; 2013 Dec 20; 288(51):36598-609. PubMed ID: 24194522
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  • 12. Gq signaling causes glomerular injury by activating TRPC6.
    Wang L, Jirka G, Rosenberg PB, Buckley AF, Gomez JA, Fields TA, Winn MP, Spurney RF.
    J Clin Invest; 2015 May 20; 125(5):1913-26. PubMed ID: 25844902
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  • 13. Angiotensin II has acute effects on TRPC6 channels in podocytes of freshly isolated glomeruli.
    Ilatovskaya DV, Palygin O, Chubinskiy-Nadezhdin V, Negulyaev YA, Ma R, Birnbaumer L, Staruschenko A.
    Kidney Int; 2014 Sep 20; 86(3):506-14. PubMed ID: 24646854
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  • 15. Angiotensin II Type I Receptor Agonistic Autoantibody Induces Podocyte Injury via Activation of the TRPC6- Calcium/Calcineurin Pathway in Pre-Eclampsia.
    Yu Y, Zhang L, Xu G, Wu Z, Li Q, Gu Y, Niu J.
    Kidney Blood Press Res; 2018 Sep 20; 43(5):1666-1676. PubMed ID: 30380548
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  • 16. TRPC6 channel as an emerging determinant of the podocyte injury susceptibility in kidney diseases.
    Ilatovskaya DV, Staruschenko A.
    Am J Physiol Renal Physiol; 2015 Sep 01; 309(5):F393-7. PubMed ID: 26084930
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  • 17. Inhibition of TRPC6 Signal Pathway Alleviates Podocyte Injury Induced by TGF-β1.
    Huang H, You Y, Lin X, Tang C, Gu X, Huang M, Qin Y, Tan J, Huang F.
    Cell Physiol Biochem; 2017 Sep 01; 41(1):163-172. PubMed ID: 28214865
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  • 18. TRPC6 enhances angiotensin II-induced albuminuria.
    Eckel J, Lavin PJ, Finch EA, Mukerji N, Burch J, Gbadegesin R, Wu G, Bowling B, Byrd A, Hall G, Sparks M, Zhang ZS, Homstad A, Barisoni L, Birbaumer L, Rosenberg P, Winn MP.
    J Am Soc Nephrol; 2011 Mar 01; 22(3):526-35. PubMed ID: 21258036
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  • 19. β-Arrestin pathway activation by selective ATR1 agonism promotes calcium influx in podocytes, leading to glomerular damage.
    Semenikhina M, Fedoriuk M, Stefanenko M, Klemens CA, Cherezova A, Marshall B, Hall G, Levchenko V, Solanki AK, Lipschutz JH, Ilatovskaya DV, Staruschenko A, Palygin O.
    Clin Sci (Lond); 2023 Dec 22; 137(24):1789-1804. PubMed ID: 38051199
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  • 20. Insulin Increases Expression of TRPC6 Channels in Podocytes by a Calcineurin-Dependent Pathway.
    Xia S, Liu Y, Li X, Thilo F, Tepel M.
    Cell Physiol Biochem; 2016 Dec 22; 38(2):659-69. PubMed ID: 26849622
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