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Journal Abstract Search


282 related items for PubMed ID: 21850188

  • 1. Critical role of SDF-1α-induced progenitor cell recruitment and macrophage VEGF production in the experimental corneal neovascularization.
    Liu G, Lu P, Li L, Jin H, He X, Mukaida N, Zhang X.
    Mol Vis; 2011; 17():2129-38. PubMed ID: 21850188
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  • 4. Enhanced experimental corneal neovascularization along with aberrant angiogenic factor expression in the absence of IL-1 receptor antagonist.
    Lu P, Li L, Liu G, Zhang X, Mukaida N.
    Invest Ophthalmol Vis Sci; 2009 Oct; 50(10):4761-8. PubMed ID: 19458323
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  • 7. Critical Role of IP-10 on Reducing Experimental Corneal Neovascularization.
    Liu G, Zhang W, Xiao Y, Lu P.
    Curr Eye Res; 2015 Sep; 40(9):891-901. PubMed ID: 25309995
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  • 8. Alkali-induced corneal neovascularization is independent of CXCR2-mediated neutrophil infiltration.
    Lu P, Li L, Mukaida N, Zhang X.
    Cornea; 2007 Feb; 26(2):199-206. PubMed ID: 17251813
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  • 9. Protective roles of the fractalkine/CX3CL1-CX3CR1 interactions in alkali-induced corneal neovascularization through enhanced antiangiogenic factor expression.
    Lu P, Li L, Kuno K, Wu Y, Baba T, Li YY, Zhang X, Mukaida N.
    J Immunol; 2008 Mar 15; 180(6):4283-91. PubMed ID: 18322241
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  • 10. Opposite roles of CCR2 and CX3CR1 macrophages in alkali-induced corneal neovascularization.
    Lu P, Li L, Liu G, van Rooijen N, Mukaida N, Zhang X.
    Cornea; 2009 Jun 15; 28(5):562-9. PubMed ID: 19421039
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  • 11. Activation of Toll-like receptor 3 promotes pathological corneal neovascularization by enhancement of SDF-1-mediated endothelial progenitor cell recruitment.
    Zhao R, Zhang J, Wang Y, Jin J, Zhou H, Chen J, Su SB.
    Exp Eye Res; 2019 Jan 15; 178():177-185. PubMed ID: 30321512
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  • 13. Inhibition of Experimental Corneal Neovascularization by the Tight Junction Protein ZO-1.
    Yao Q, Wu H, Ren H, Cao J, Shao Y, Liu G, Lu P.
    J Ocul Pharmacol Ther; 2024 Jan 15; 40(6):379-388. PubMed ID: 39172123
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  • 14. Comparison of genome-wide gene expression in suture- and alkali burn-induced murine corneal neovascularization.
    Jia C, Zhu W, Ren S, Xi H, Li S, Wang Y.
    Mol Vis; 2011 Jan 15; 17():2386-99. PubMed ID: 21921991
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  • 16. Upadacitinib inhibits corneal inflammation and neovascularization by suppressing M1 macrophage infiltration in the corneal alkali burn model.
    Yu J, Shen Y, Luo J, Jin J, Li P, Feng P, Guan H.
    Int Immunopharmacol; 2023 Mar 15; 116():109680. PubMed ID: 36739832
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  • 18. The effect of TC14012 on alkali burn-induced corneal neovascularization in mice.
    Shen M, Yuan F, Jin J, Yuan Y.
    Ophthalmic Res; 2014 Mar 15; 52(1):17-24. PubMed ID: 24853648
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  • 20. High-mobility group box-1-Toll-Like receptor 4 axis mediates the recruitment of endothelial progenitor cells in alkali-induced corneal neovascularization.
    Yang S, Yang TS, Wang F, Su SB.
    Int Immunopharmacol; 2015 Sep 15; 28(1):450-8. PubMed ID: 26202806
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