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Journal Abstract Search

298 related items for PubMed ID: 21856905

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  • 3. A high-lipid diet potentiates left ventricular dysfunction in nitric oxide synthase 3-deficient mice after chronic pressure overload.
    Ahmadie R, Santiago JJ, Walker J, Fang T, Le K, Zhao Z, Azordegan N, Bage S, Lytwyn M, Rattan S, Dixon IM, Kardami E, Moghadasian MH, Jassal DS.
    J Nutr; 2010 Aug; 140(8):1438-44. PubMed ID: 20554900
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  • 4. Pressure overload-induced LV hypertrophy and dysfunction in mice are exacerbated by congenital NOS3 deficiency.
    Ichinose F, Bloch KD, Wu JC, Hataishi R, Aretz HT, Picard MH, Scherrer-Crosbie M.
    Am J Physiol Heart Circ Physiol; 2004 Mar; 286(3):H1070-5. PubMed ID: 14644766
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  • 9. Sepiapterin prevents left ventricular hypertrophy and dilatory remodeling induced by pressure overload in rats.
    Yoshioka K, Otani H, Shimazu T, Fujita M, Iwasaka T, Shiojima I.
    Am J Physiol Heart Circ Physiol; 2015 Nov 15; 309(10):H1782-91. PubMed ID: 26408540
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  • 10. Protein kinase g iα inhibits pressure overload-induced cardiac remodeling and is required for the cardioprotective effect of sildenafil in vivo.
    Blanton RM, Takimoto E, Lane AM, Aronovitz M, Piotrowski R, Karas RH, Kass DA, Mendelsohn ME.
    J Am Heart Assoc; 2012 Oct 15; 1(5):e003731. PubMed ID: 23316302
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  • 12. Effects of adiponectin deficiency on structural and metabolic remodeling in mice subjected to pressure overload.
    O'Shea KM, Chess DJ, Khairallah RJ, Rastogi S, Hecker PA, Sabbah HN, Walsh K, Stanley WC.
    Am J Physiol Heart Circ Physiol; 2010 Jun 15; 298(6):H1639-45. PubMed ID: 20348222
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  • 13. Cardiomyocyte-specific overexpression of nitric oxide synthase 3 improves left ventricular performance and reduces compensatory hypertrophy after myocardial infarction.
    Janssens S, Pokreisz P, Schoonjans L, Pellens M, Vermeersch P, Tjwa M, Jans P, Scherrer-Crosbie M, Picard MH, Szelid Z, Gillijns H, Van de Werf F, Collen D, Bloch KD.
    Circ Res; 2004 May 14; 94(9):1256-62. PubMed ID: 15044322
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  • 16. A novel mtDNA repair fusion protein attenuates maladaptive remodeling and preserves cardiac function in heart failure.
    Bradley JM, Li Z, Organ CL, Polhemus DJ, Otsuka H, Islam KN, Bhushan S, Gorodnya OM, Ruchko MV, Gillespie MN, Wilson GL, Lefer DJ.
    Am J Physiol Heart Circ Physiol; 2018 Feb 01; 314(2):H311-H321. PubMed ID: 29101177
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  • 17. Endothelial nitric oxide synthase limits left ventricular remodeling after myocardial infarction in mice.
    Scherrer-Crosbie M, Ullrich R, Bloch KD, Nakajima H, Nasseri B, Aretz HT, Lindsey ML, Vançon AC, Huang PL, Lee RT, Zapol WM, Picard MH.
    Circulation; 2001 Sep 11; 104(11):1286-91. PubMed ID: 11551881
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  • 18. Knockout of insulin receptors in cardiomyocytes attenuates coronary arterial dysfunction induced by pressure overload.
    Symons JD, Hu P, Yang Y, Wang X, Zhang QJ, Wende AR, Sloan CL, Sena S, Abel ED, Litwin SE.
    Am J Physiol Heart Circ Physiol; 2011 Jan 11; 300(1):H374-81. PubMed ID: 20971769
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  • 19. Oxidant stress from nitric oxide synthase-3 uncoupling stimulates cardiac pathologic remodeling from chronic pressure load.
    Takimoto E, Champion HC, Li M, Ren S, Rodriguez ER, Tavazzi B, Lazzarino G, Paolocci N, Gabrielson KL, Wang Y, Kass DA.
    J Clin Invest; 2005 May 11; 115(5):1221-31. PubMed ID: 15841206
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  • 20. Adipose tissue ATGL modifies the cardiac lipidome in pressure-overload-induced left ventricular failure.
    Salatzki J, Foryst-Ludwig A, Bentele K, Blumrich A, Smeir E, Ban Z, Brix S, Grune J, Beyhoff N, Klopfleisch R, Dunst S, Surma MA, Klose C, Rothe M, Heinzel FR, Krannich A, Kershaw EE, Beule D, Schulze PC, Marx N, Kintscher U.
    PLoS Genet; 2018 Jan 11; 14(1):e1007171. PubMed ID: 29320510
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