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Journal Abstract Search

1646 related items for PubMed ID: 21880712

  • 1. Tumor suppressor and aging biomarker p16(INK4a) induces cellular senescence without the associated inflammatory secretory phenotype.
    Coppé JP, Rodier F, Patil CK, Freund A, Desprez PY, Campisi J.
    J Biol Chem; 2011 Oct 21; 286(42):36396-403. PubMed ID: 21880712
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  • 2. P16 INK4a Deletion Ameliorated Renal Tubulointerstitial Injury in a Stress-induced Premature Senescence Model of Bmi-1 Deficiency.
    Jin J, Tao J, Gu X, Yu Z, Wang R, Zuo G, Li Q, Lv X, Miao D.
    Sci Rep; 2017 Aug 08; 7(1):7502. PubMed ID: 28790310
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  • 3. Differential roles for cyclin-dependent kinase inhibitors p21 and p16 in the mechanisms of senescence and differentiation in human fibroblasts.
    Stein GH, Drullinger LF, Soulard A, Dulić V.
    Mol Cell Biol; 1999 Mar 08; 19(3):2109-17. PubMed ID: 10022898
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  • 4. Targeted clearance of p21- but not p16-positive senescent cells prevents radiation-induced osteoporosis and increased marrow adiposity.
    Chandra A, Lagnado AB, Farr JN, Doolittle M, Tchkonia T, Kirkland JL, LeBrasseur NK, Robbins PD, Niedernhofer LJ, Ikeno Y, Passos JF, Monroe DG, Pignolo RJ, Khosla S.
    Aging Cell; 2022 May 08; 21(5):e13602. PubMed ID: 35363946
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  • 5. Tissue specificity of senescent cell accumulation during physiologic and accelerated aging of mice.
    Yousefzadeh MJ, Zhao J, Bukata C, Wade EA, McGowan SJ, Angelini LA, Bank MP, Gurkar AU, McGuckian CA, Calubag MF, Kato JI, Burd CE, Robbins PD, Niedernhofer LJ.
    Aging Cell; 2020 Mar 08; 19(3):e13094. PubMed ID: 31981461
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  • 6. Long-term growth arrest of PUVA-treated fibroblasts in G2/M in the absence of p16(INK4a) p21(CIP1) or p53.
    Ma W, Hommel C, Brenneisen P, Peters T, Smit N, Sedivy J, Scharffetter-Kochanek K, Wlaschek M.
    Exp Dermatol; 2003 Oct 08; 12(5):629-37. PubMed ID: 14705804
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  • 12. E2FBP1 antagonizes the p16(INK4A)-Rb tumor suppressor machinery for growth suppression and cellular senescence by regulating promyelocytic leukemia protein stability.
    Fukuyo Y, Takahashi A, Hara E, Horikoshi N, Pandita TK, Nakajima T.
    Int J Oral Sci; 2011 Oct 08; 3(4):200-8. PubMed ID: 22010578
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  • 14. Cells exhibiting strong p16INK4a promoter activation in vivo display features of senescence.
    Liu JY, Souroullas GP, Diekman BO, Krishnamurthy J, Hall BM, Sorrentino JA, Parker JS, Sessions GA, Gudkov AV, Sharpless NE.
    Proc Natl Acad Sci U S A; 2019 Feb 12; 116(7):2603-2611. PubMed ID: 30683717
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  • 15. CDK inhibitors (p16/p19/p21) induce senescence and autophagy in cancer-associated fibroblasts, "fueling" tumor growth via paracrine interactions, without an increase in neo-angiogenesis.
    Capparelli C, Chiavarina B, Whitaker-Menezes D, Pestell TG, Pestell RG, Hulit J, Andò S, Howell A, Martinez-Outschoorn UE, Sotgia F, Lisanti MP.
    Cell Cycle; 2012 Oct 01; 11(19):3599-610. PubMed ID: 22935696
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