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Journal Abstract Search

478 related items for PubMed ID: 21915802

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  • 3. Hyperparathyroidism in hereditary syndromes: special expressions and special managements.
    Marx SJ, Simonds WF, Agarwal SK, Burns AL, Weinstein LS, Cochran C, Skarulis MC, Spiegel AM, Libutti SK, Alexander HR, Chen CC, Chang R, Chandrasekharappa SC, Collins FS.
    J Bone Miner Res; 2002 Nov; 17 Suppl 2():N37-43. PubMed ID: 12412776
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  • 4. Molecular pathogenesis of primary hyperparathyroidism.
    Cetani F, Pardi E, Borsari S, Marcocci C.
    J Endocrinol Invest; 2011 Jul; 34(7 Suppl):35-9. PubMed ID: 21985978
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  • 8. Genetic analyses in familial isolated hyperparathyroidism: implication for clinical assessment and surgical management.
    Cetani F, Pardi E, Ambrogini E, Lemmi M, Borsari S, Cianferotti L, Vignali E, Viacava P, Berti P, Mariotti S, Pinchera A, Marcocci C.
    Clin Endocrinol (Oxf); 2006 Feb; 64(2):146-52. PubMed ID: 16430712
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  • 10. Management of familial hyperparathyroidism syndromes: MEN1, MEN2, MEN4, HPT-Jaw tumour, Familial isolated hyperparathyroidism, FHH, and neonatal severe hyperparathyroidism.
    Cristina EV, Alberto F.
    Best Pract Res Clin Endocrinol Metab; 2018 Dec; 32(6):861-875. PubMed ID: 30665551
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  • 12. Familial parathyroid tumours-comparison of clinical profiles between syndromes.
    Figueiredo AA, Saramago A, Cavaco BM, Simões-Pereira J, Leite V.
    J Endocrinol Invest; 2023 Sep; 46(9):1799-1806. PubMed ID: 36780067
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  • 13. Tumor suppressor gene mutation in a patient with a history of hyperparathyroidism-jaw tumor syndrome and healed generalized osteitis fibrosa cystica: a case report and genetic pathophysiology review.
    Parfitt J, Harris M, Wright JM, Kalamchi S.
    J Oral Maxillofac Surg; 2015 Jan; 73(1):194.e1-9. PubMed ID: 25511968
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  • 14. Inactivating calcium-sensing receptor mutations in patients with primary hyperparathyroidism.
    Frank-Raue K, Leidig-Bruckner G, Haag C, Schulze E, Lorenz A, Schmitz-Winnenthal H, Raue F.
    Clin Endocrinol (Oxf); 2011 Jul; 75(1):50-5. PubMed ID: 21521328
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  • 17. Familial isolated primary hyperparathyroidism/hyperparathyroidism-jaw tumour syndrome caused by germline gross deletion or point mutations of CDC73 gene in Chinese.
    Kong J, Wang O, Nie M, Shi J, Hu Y, Jiang Y, Li M, Xia W, Meng X, Xing X.
    Clin Endocrinol (Oxf); 2014 Aug; 81(2):222-30. PubMed ID: 24716902
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  • 19. Familial isolated hyperparathyroidism is rarely caused by germline mutation in HRPT2, the gene for the hyperparathyroidism-jaw tumor syndrome.
    Simonds WF, Robbins CM, Agarwal SK, Hendy GN, Carpten JD, Marx SJ.
    J Clin Endocrinol Metab; 2004 Jan; 89(1):96-102. PubMed ID: 14715834
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  • 20. Detection of the first gross CDC73 germline deletion in an HPT-JT syndrome family.
    Cascón A, Huarte-Mendicoa CV, Javier Leandro-García L, Letón R, Suela J, Santana A, Costa MB, Comino-Méndez I, Landa I, Sánchez L, Rodríguez-Antona C, Cigudosa JC, Robledo M.
    Genes Chromosomes Cancer; 2011 Nov; 50(11):922-9. PubMed ID: 21837707
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