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Journal Abstract Search

235 related items for PubMed ID: 21933012

  • 1.
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  • 2. Restoring endoplasmic reticulum homeostasis improves functional recovery after spinal cord injury.
    Ohri SS, Hetman M, Whittemore SR.
    Neurobiol Dis; 2013 Oct; 58():29-37. PubMed ID: 23659896
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  • 3. Attenuating the endoplasmic reticulum stress response improves functional recovery after spinal cord injury.
    Ohri SS, Maddie MA, Zhao Y, Qiu MS, Hetman M, Whittemore SR.
    Glia; 2011 Oct; 59(10):1489-502. PubMed ID: 21638341
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  • 5. Activating Transcription Factor-6α Deletion Modulates the Endoplasmic Reticulum Stress Response after Spinal Cord Injury but Does Not Affect Locomotor Recovery.
    Saraswat Ohri S, Mullins A, Hetman M, Whittemore SR.
    J Neurotrauma; 2018 Feb 01; 35(3):486-491. PubMed ID: 26842780
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  • 6. Oligodendrocyte-specific deletion of Xbp1 exacerbates the endoplasmic reticulum stress response and restricts locomotor recovery after thoracic spinal cord injury.
    Saraswat Ohri S, Howard RM, Liu Y, Andres KR, Shepard CT, Hetman M, Whittemore SR.
    Glia; 2021 Feb 01; 69(2):424-435. PubMed ID: 32926479
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  • 9. Hypoxia-inducible factor prolyl hydroxylase domain (PHD) inhibition after contusive spinal cord injury does not improve locomotor recovery.
    Wei GZ, Saraswat Ohri S, Khattar NK, Listerman AW, Doyle CH, Andres KR, Karuppagounder SS, Ratan RR, Whittemore SR, Hetman M.
    PLoS One; 2021 Feb 01; 16(4):e0249591. PubMed ID: 33819286
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  • 10. Acute Neural and Proteostasis Messenger Ribonucleic Acid Levels Predict Chronic Locomotor Recovery after Contusive Spinal Cord Injury.
    Saraswat Ohri S, Burke DA, Andres KR, Hetman M, Whittemore SR.
    J Neurotrauma; 2021 Feb 01; 38(3):365-372. PubMed ID: 33076743
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  • 12. Deletion of endoplasmic reticulum stress-induced CHOP protects microvasculature post-spinal cord injury.
    Fassbender JM, Saraswat-Ohri S, Myers SA, Gruenthal MJ, Benton RL, Whittemore SR.
    Curr Neurovasc Res; 2012 Nov 01; 9(4):274-81. PubMed ID: 22873727
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  • 13. [Effects of electroacupuncture at "Jiaji"(EX-B2) on autophagy and endoplasmic reticulum stress in spinal cord injury mice].
    Dai P, Huang SQ, Tang CL, Dai N, Zhao HD, Tan YL, Yang YH, Tao CH.
    Zhen Ci Yan Jiu; 2021 Jan 25; 46(1):45-51. PubMed ID: 33559425
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  • 16. Oligodendrocyte-selective deletion of the eIF2α kinase Perk/Eif2ak3 limits functional recovery after spinal cord injury.
    Saraswat Ohri S, Forston MD, Myers SA, Brown BL, Andres KR, Howard RM, Gao Y, Liu Y, Cavener DR, Hetman M, Whittemore SR.
    Glia; 2024 Jul 25; 72(7):1259-1272. PubMed ID: 38587137
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  • 18. Behavioral, histological, and ex vivo magnetic resonance imaging assessment of graded contusion spinal cord injury in mice.
    Nishi RA, Liu H, Chu Y, Hamamura M, Su MY, Nalcioglu O, Anderson AJ.
    J Neurotrauma; 2007 Apr 25; 24(4):674-89. PubMed ID: 17439350
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  • 19. Nerve growth factor improves functional recovery by inhibiting endoplasmic reticulum stress-induced neuronal apoptosis in rats with spinal cord injury.
    Zhang H, Wu F, Kong X, Yang J, Chen H, Deng L, Cheng Y, Ye L, Zhu S, Zhang X, Wang Z, Shi H, Fu X, Li X, Xu H, Lin L, Xiao J.
    J Transl Med; 2014 May 15; 12():130. PubMed ID: 24884850
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