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Journal Abstract Search

316 related items for PubMed ID: 21936838

  • 1. Using yeast models to probe the molecular basis of amyotrophic lateral sclerosis.
    Bastow EL, Gourlay CW, Tuite MF.
    Biochem Soc Trans; 2011 Oct; 39(5):1482-7. PubMed ID: 21936838
    [Abstract] [Full Text] [Related]

  • 2. Aberrant localization of FUS and TDP43 is associated with misfolding of SOD1 in amyotrophic lateral sclerosis.
    Pokrishevsky E, Grad LI, Yousefi M, Wang J, Mackenzie IR, Cashman NR.
    PLoS One; 2012 Oct; 7(4):e35050. PubMed ID: 22493728
    [Abstract] [Full Text] [Related]

  • 3. The small heat shock protein B8 (HspB8) promotes autophagic removal of misfolded proteins involved in amyotrophic lateral sclerosis (ALS).
    Crippa V, Sau D, Rusmini P, Boncoraglio A, Onesto E, Bolzoni E, Galbiati M, Fontana E, Marino M, Carra S, Bendotti C, De Biasi S, Poletti A.
    Hum Mol Genet; 2010 Sep 01; 19(17):3440-56. PubMed ID: 20570967
    [Abstract] [Full Text] [Related]

  • 4. FUS and TARDBP but not SOD1 interact in genetic models of amyotrophic lateral sclerosis.
    Kabashi E, Bercier V, Lissouba A, Liao M, Brustein E, Rouleau GA, Drapeau P.
    PLoS Genet; 2011 Aug 01; 7(8):e1002214. PubMed ID: 21829392
    [Abstract] [Full Text] [Related]

  • 5. Pathological Modification of TDP-43 in Amyotrophic Lateral Sclerosis with SOD1 Mutations.
    Jeon GS, Shim YM, Lee DY, Kim JS, Kang M, Ahn SH, Shin JY, Geum D, Hong YH, Sung JJ.
    Mol Neurobiol; 2019 Mar 01; 56(3):2007-2021. PubMed ID: 29982983
    [Abstract] [Full Text] [Related]

  • 6. Rab1-dependent ER-Golgi transport dysfunction is a common pathogenic mechanism in SOD1, TDP-43 and FUS-associated ALS.
    Soo KY, Halloran M, Sundaramoorthy V, Parakh S, Toth RP, Southam KA, McLean CA, Lock P, King A, Farg MA, Atkin JD.
    Acta Neuropathol; 2015 Nov 01; 130(5):679-97. PubMed ID: 26298469
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  • 8. SOD1 aggregation and ALS: role of metallation states and disulfide status.
    Sheng Y, Chattopadhyay M, Whitelegge J, Valentine JS.
    Curr Top Med Chem; 2012 Nov 01; 12(22):2560-72. PubMed ID: 23339308
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  • 10. The role of heat shock proteins in Amyotrophic Lateral Sclerosis: The therapeutic potential of Arimoclomol.
    Kalmar B, Lu CH, Greensmith L.
    Pharmacol Ther; 2014 Jan 01; 141(1):40-54. PubMed ID: 23978556
    [Abstract] [Full Text] [Related]

  • 11. Shared Molecular Mechanisms in Alzheimer's Disease and Amyotrophic Lateral Sclerosis: Neurofilament-Dependent Transport of sAPP, FUS, TDP-43 and SOD1, with Endoplasmic Reticulum-Like Tubules.
    Muresan V, Ladescu Muresan Z.
    Neurodegener Dis; 2016 Jan 01; 16(1-2):55-61. PubMed ID: 26605911
    [Abstract] [Full Text] [Related]

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  • 13. Protein folding alterations in amyotrophic lateral sclerosis.
    Parakh S, Atkin JD.
    Brain Res; 2016 Oct 01; 1648(Pt B):633-649. PubMed ID: 27064076
    [Abstract] [Full Text] [Related]

  • 14. Pathological TDP-43 distinguishes sporadic amyotrophic lateral sclerosis from amyotrophic lateral sclerosis with SOD1 mutations.
    Mackenzie IR, Bigio EH, Ince PG, Geser F, Neumann M, Cairns NJ, Kwong LK, Forman MS, Ravits J, Stewart H, Eisen A, McClusky L, Kretzschmar HA, Monoranu CM, Highley JR, Kirby J, Siddique T, Shaw PJ, Lee VM, Trojanowski JQ.
    Ann Neurol; 2007 May 01; 61(5):427-34. PubMed ID: 17469116
    [Abstract] [Full Text] [Related]

  • 15. TDP-43 or FUS-induced misfolded human wild-type SOD1 can propagate intercellularly in a prion-like fashion.
    Pokrishevsky E, Grad LI, Cashman NR.
    Sci Rep; 2016 Mar 01; 6():22155. PubMed ID: 26926802
    [Abstract] [Full Text] [Related]

  • 16. From nucleation to widespread propagation: A prion-like concept for ALS.
    Maniecka Z, Polymenidou M.
    Virus Res; 2015 Sep 02; 207():94-105. PubMed ID: 25656065
    [Abstract] [Full Text] [Related]

  • 17. Mutant SOD1 mediated pathogenesis of Amyotrophic Lateral Sclerosis.
    Kaur SJ, McKeown SR, Rashid S.
    Gene; 2016 Feb 15; 577(2):109-18. PubMed ID: 26657039
    [Abstract] [Full Text] [Related]

  • 18. Modeling ALS and FTLD proteinopathies in yeast: an efficient approach for studying protein aggregation and toxicity.
    Kryndushkin D, Shewmaker F.
    Prion; 2011 Feb 15; 5(4):250-7. PubMed ID: 22052354
    [Abstract] [Full Text] [Related]

  • 19. Connecting RNA-Modifying Similarities of TDP-43, FUS, and SOD1 with MicroRNA Dysregulation Amidst A Renewed Network Perspective of Amyotrophic Lateral Sclerosis Proteinopathy.
    Pham J, Keon M, Brennan S, Saksena N.
    Int J Mol Sci; 2020 May 14; 21(10):. PubMed ID: 32422969
    [Abstract] [Full Text] [Related]

  • 20. SOD1 and TDP-43 animal models of amyotrophic lateral sclerosis: recent advances in understanding disease toward the development of clinical treatments.
    Joyce PI, Fratta P, Fisher EM, Acevedo-Arozena A.
    Mamm Genome; 2011 Aug 14; 22(7-8):420-48. PubMed ID: 21706386
    [Abstract] [Full Text] [Related]

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