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Journal Abstract Search

228 related items for PubMed ID: 21984545

  • 1. Functional consequences of stably expressing a mutant calsequestrin (CASQ2D307H) in the CASQ2 null background.
    Kalyanasundaram A, Viatchenko-Karpinski S, Belevych AE, Lacombe VA, Hwang HS, Knollmann BC, Gyorke S, Periasamy M.
    Am J Physiol Heart Circ Physiol; 2012 Jan 01; 302(1):H253-61. PubMed ID: 21984545
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  • 4. Calsequestrin mutation and catecholaminergic polymorphic ventricular tachycardia: a simulation study of cellular mechanism.
    Faber GM, Rudy Y.
    Cardiovasc Res; 2007 Jul 01; 75(1):79-88. PubMed ID: 17531962
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  • 7. The role of mutant protein level in autosomal recessive catecholamine dependent polymorphic ventricular tachycardia (CPVT2).
    Katz G, Shainberg A, Hochhauser E, Kurtzwald-Josefson E, Issac A, El-Ani D, Aravot D, Afek A, Seidman JG, Seidman CE, Eldar M, Arad M.
    Biochem Pharmacol; 2013 Dec 01; 86(11):1576-83. PubMed ID: 24070655
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  • 8. Prevention of ventricular arrhythmia and calcium dysregulation in a catecholaminergic polymorphic ventricular tachycardia mouse model carrying calsequestrin-2 mutation.
    Alcalai R, Wakimoto H, Arad M, Planer D, Konno T, Wang L, Seidman JG, Seidman CE, Berul CI.
    J Cardiovasc Electrophysiol; 2011 Mar 01; 22(3):316-24. PubMed ID: 20807279
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  • 11. Abnormal interactions of calsequestrin with the ryanodine receptor calcium release channel complex linked to exercise-induced sudden cardiac death.
    Terentyev D, Nori A, Santoro M, Viatchenko-Karpinski S, Kubalova Z, Gyorke I, Terentyeva R, Vedamoorthyrao S, Blom NA, Valle G, Napolitano C, Williams SC, Volpe P, Priori SG, Gyorke S.
    Circ Res; 2006 May 12; 98(9):1151-8. PubMed ID: 16601229
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  • 16. Ablation of HRC alleviates cardiac arrhythmia and improves abnormal Ca handling in CASQ2 knockout mice prone to CPVT.
    Liu B, Ho HT, Brunello L, Unudurthi SD, Lou Q, Belevych AE, Qian L, Kim DH, Cho C, Janssen PM, Hund TJ, Knollmann BC, Kranias EG, Györke S.
    Cardiovasc Res; 2015 Nov 01; 108(2):299-311. PubMed ID: 26410369
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  • 17. Impaired calcium-calmodulin-dependent inactivation of Cav1.2 contributes to loss of sarcoplasmic reticulum calcium release refractoriness in mice lacking calsequestrin 2.
    Kryshtal DO, Gryshchenko O, Gomez-Hurtado N, Knollmann BC.
    J Mol Cell Cardiol; 2015 May 01; 82():75-83. PubMed ID: 25758429
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