These tools will no longer be maintained as of December 31, 2024. Archived website can be found here. PubMed4Hh GitHub repository can be found here. Contact NLM Customer Service if you have questions.


Pubmed for Handhelds

PUBMED FOR HANDHELDS

Journal Abstract Search

303 related items for PubMed ID: 22052354

  • 1.
    ; . PubMed ID:
    [No Abstract] [Full Text] [Related]

  • 2.
    ; . PubMed ID:
    [No Abstract] [Full Text] [Related]

  • 3. Inclusions in frontotemporal lobar degeneration with TDP-43 proteinopathy (FTLD-TDP) and amyotrophic lateral sclerosis (ALS), but not FTLD with FUS proteinopathy (FTLD-FUS), have properties of amyloid.
    Bigio EH, Wu JY, Deng HX, Bit-Ivan EN, Mao Q, Ganti R, Peterson M, Siddique N, Geula C, Siddique T, Mesulam M.
    Acta Neuropathol; 2013 Mar; 125(3):463-5. PubMed ID: 23378033
    [No Abstract] [Full Text] [Related]

  • 4. FUS/TLS forms cytoplasmic aggregates, inhibits cell growth and interacts with TDP-43 in a yeast model of amyotrophic lateral sclerosis.
    Kryndushkin D, Wickner RB, Shewmaker F.
    Protein Cell; 2011 Mar; 2(3):223-36. PubMed ID: 21452073
    [Abstract] [Full Text] [Related]

  • 5. Implications of the prion-related Q/N domains in TDP-43 and FUS.
    Udan M, Baloh RH.
    Prion; 2011 Mar; 5(1):1-5. PubMed ID: 21135580
    [Abstract] [Full Text] [Related]

  • 6. Molecular determinants and genetic modifiers of aggregation and toxicity for the ALS disease protein FUS/TLS.
    Sun Z, Diaz Z, Fang X, Hart MP, Chesi A, Shorter J, Gitler AD.
    PLoS Biol; 2011 Apr; 9(4):e1000614. PubMed ID: 21541367
    [Abstract] [Full Text] [Related]

  • 7.
    ; . PubMed ID:
    [No Abstract] [Full Text] [Related]

  • 8. Using yeast models to probe the molecular basis of amyotrophic lateral sclerosis.
    Bastow EL, Gourlay CW, Tuite MF.
    Biochem Soc Trans; 2011 Oct; 39(5):1482-7. PubMed ID: 21936838
    [Abstract] [Full Text] [Related]

  • 9.
    ; . PubMed ID:
    [No Abstract] [Full Text] [Related]

  • 10. Aberrant interaction between FUS and SFPQ in neurons in a wide range of FTLD spectrum diseases.
    Ishigaki S, Riku Y, Fujioka Y, Endo K, Iwade N, Kawai K, Ishibashi M, Yokoi S, Katsuno M, Watanabe H, Mori K, Akagi A, Yokota O, Terada S, Kawakami I, Suzuki N, Warita H, Aoki M, Yoshida M, Sobue G.
    Brain; 2020 Aug 01; 143(8):2398-2405. PubMed ID: 32770214
    [Abstract] [Full Text] [Related]

  • 11.
    ; . PubMed ID:
    [No Abstract] [Full Text] [Related]

  • 12.
    ; . PubMed ID:
    [No Abstract] [Full Text] [Related]

  • 13.
    ; . PubMed ID:
    [No Abstract] [Full Text] [Related]

  • 14.
    ; . PubMed ID:
    [No Abstract] [Full Text] [Related]

  • 15. The prion-like nature of amyotrophic lateral sclerosis.
    McAlary L, Yerbury JJ, Cashman NR.
    Prog Mol Biol Transl Sci; 2020 Aug 01; 175():261-296. PubMed ID: 32958236
    [Abstract] [Full Text] [Related]

  • 16. Molecular basis of amyotrophic lateral sclerosis.
    Liscic RM, Breljak D.
    Prog Neuropsychopharmacol Biol Psychiatry; 2011 Mar 30; 35(2):370-2. PubMed ID: 20655970
    [Abstract] [Full Text] [Related]

  • 17.
    ; . PubMed ID:
    [No Abstract] [Full Text] [Related]

  • 18.
    ; . PubMed ID:
    [No Abstract] [Full Text] [Related]

  • 19. Fused in Sarcoma: Properties, Self-Assembly and Correlation with Neurodegenerative Diseases.
    Chen C, Ding X, Akram N, Xue S, Luo SZ.
    Molecules; 2019 Apr 24; 24(8):. PubMed ID: 31022909
    [Abstract] [Full Text] [Related]

  • 20.
    ; . PubMed ID:
    [No Abstract] [Full Text] [Related]

    Page: [Next] [New Search]
    of 16.