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84 related items for PubMed ID: 22069262
1. Role of TLR2 in the pathogenesis of autoimmune diabetes and its therapeutic implication. Lee MS, Kim DH, Lee JC, Kim S, Kim HS. Diabetes Metab Res Rev; 2011 Nov; 27(8):797-801. PubMed ID: 22069262 [Abstract] [Full Text] [Related]
2. Inhibition of autoimmune diabetes by TLR2 tolerance. Kim DH, Lee JC, Kim S, Oh SH, Lee MK, Kim KW, Lee MS. J Immunol; 2011 Nov 15; 187(10):5211-20. PubMed ID: 21998452 [Abstract] [Full Text] [Related]
3. Toll-like receptor 2 senses beta-cell death and contributes to the initiation of autoimmune diabetes. Kim HS, Han MS, Chung KW, Kim S, Kim E, Kim MJ, Jang E, Lee HA, Youn J, Akira S, Lee MS. Immunity; 2007 Aug 15; 27(2):321-33. PubMed ID: 17707128 [Abstract] [Full Text] [Related]
4. Marginal-zone B-cells of nonobese diabetic mice expand with diabetes onset, invade the pancreatic lymph nodes, and present autoantigen to diabetogenic T-cells. Mariño E, Batten M, Groom J, Walters S, Liuwantara D, Mackay F, Grey ST. Diabetes; 2008 Feb 15; 57(2):395-404. PubMed ID: 18025414 [Abstract] [Full Text] [Related]
5. Transfusion of apoptotic beta-cells induces immune tolerance to beta-cell antigens and prevents type 1 diabetes in NOD mice. Xia CQ, Peng R, Qiu Y, Annamalai M, Gordon D, Clare-Salzler MJ. Diabetes; 2007 Aug 15; 56(8):2116-23. PubMed ID: 17496235 [Abstract] [Full Text] [Related]
6. Differential role of MyD88 and TRIF signaling in myeloid cells in the pathogenesis of autoimmune diabetes. Androulidaki A, Wachsmuth L, Polykratis A, Pasparakis M. PLoS One; 2018 Aug 15; 13(3):e0194048. PubMed ID: 29522531 [Abstract] [Full Text] [Related]
12. CD8+ T cells in type 1 diabetes. Tsai S, Shameli A, Santamaria P. Adv Immunol; 2008 Aug 15; 100():79-124. PubMed ID: 19111164 [Abstract] [Full Text] [Related]
13. Heightened interferon-alpha/beta response causes myeloid cell dysfunction and promotes T1D pathogenesis in NOD mice. Peng RH, Paek E, Xia CQ, Tennyson N, Clare-Salzler MJ. Ann N Y Acad Sci; 2006 Oct 15; 1079():99-102. PubMed ID: 17130537 [Abstract] [Full Text] [Related]