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237 related items for PubMed ID: 22086197
1. Sequential activation of hypoxia-inducible factor 1 and specificity protein 1 is required for hypoxia-induced transcriptional stimulation of Abcc8. Woo SK, Kwon MS, Geng Z, Chen Z, Ivanov A, Bhatta S, Gerzanich V, Simard JM. J Cereb Blood Flow Metab; 2012 Mar; 32(3):525-36. PubMed ID: 22086197 [Abstract] [Full Text] [Related]
9. Characterization of the mouse sulfonylurea receptor 1 promoter and its regulation. Hernández-Sánchez C, Ito Y, Ferrer J, Reitman M, LeRoith D. J Biol Chem; 1999 Jun 25; 274(26):18261-70. PubMed ID: 10373428 [Abstract] [Full Text] [Related]
10. Glibenclamide improves neurological function in neonatal hypoxia-ischemia in rats. Zhou Y, Fathali N, Lekic T, Tang J, Zhang JH. Brain Res; 2009 May 13; 1270():131-9. PubMed ID: 19306849 [Abstract] [Full Text] [Related]
19. Mechanism of KATP hyperactivity and sulfonylurea tolerance due to a diabetogenic mutation in L0 helix of sulfonylurea receptor 1 (ABCC8). Babenko AP, Vaxillaire M. FEBS Lett; 2011 Nov 16; 585(22):3555-9. PubMed ID: 22020219 [Abstract] [Full Text] [Related]
20. Compound heterozygous mutations in the SUR1 (ABCC 8) subunit of pancreatic K(ATP) channels cause neonatal diabetes by perturbing the coupling between Kir6.2 and SUR1 subunits. Lin YW, Akrouh A, Hsu Y, Hughes N, Nichols CG, De León DD. Channels (Austin); 2012 Nov 16; 6(2):133-8. PubMed ID: 22562119 [Abstract] [Full Text] [Related] Page: [Next] [New Search]