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158 related items for PubMed ID: 22094641
1. Levels of kinesin light chain and dynein intermediate chain are reduced in the frontal cortex in Alzheimer's disease: implications for axoplasmic transport. Morel M, Héraud C, Nicaise C, Suain V, Brion JP. Acta Neuropathol; 2012 Jan; 123(1):71-84. PubMed ID: 22094641 [Abstract] [Full Text] [Related]
2. Presenilin influences glycogen synthase kinase-3 β (GSK-3β) for kinesin-1 and dynein function during axonal transport. Dolma K, Iacobucci GJ, Hong Zheng K, Shandilya J, Toska E, White JA, Spina E, Gunawardena S. Hum Mol Genet; 2014 Mar 01; 23(5):1121-33. PubMed ID: 24105467 [Abstract] [Full Text] [Related]
9. Abeta exacerbates the neuronal dysfunction caused by human tau expression in a Drosophila model of Alzheimer's disease. Folwell J, Cowan CM, Ubhi KK, Shiabh H, Newman TA, Shepherd D, Mudher A. Exp Neurol; 2010 Jun 01; 223(2):401-9. PubMed ID: 19782075 [Abstract] [Full Text] [Related]
10. LMTK2 binds to kinesin light chains to mediate anterograde axonal transport of cdk5/p35 and LMTK2 levels are reduced in Alzheimer's disease brains. Mórotz GM, Glennon EB, Gomez-Suaga P, Lau DHW, Robinson ED, Sedlák É, Vagnoni A, Noble W, Miller CCJ. Acta Neuropathol Commun; 2019 May 08; 7(1):73. PubMed ID: 31068217 [Abstract] [Full Text] [Related]
14. Atlas stumbled: kinesin light chain-1 variant E triggers a vicious cycle of axonal transport disruption and amyloid-β generation in Alzheimer's disease. Gan KJ, Morihara T, Silverman MA. Bioessays; 2015 Feb 08; 37(2):131-41. PubMed ID: 25394182 [Abstract] [Full Text] [Related]
20. The retinoic acid and brain-derived neurotrophic factor differentiated SH-SY5Y cell line as a model for Alzheimer's disease-like tau phosphorylation. Jämsä A, Hasslund K, Cowburn RF, Bäckström A, Vasänge M. Biochem Biophys Res Commun; 2004 Jul 02; 319(3):993-1000. PubMed ID: 15184080 [Abstract] [Full Text] [Related] Page: [Next] [New Search]