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Journal Abstract Search
170 related items for PubMed ID: 22189709
1. Acm1 contributes to nuclear positioning by inhibiting Cdh1-substrate interactions. Martinez JS, Hall H, Bartolowits MD, Hall MC. Cell Cycle; 2012 Jan 15; 11(2):384-94. PubMed ID: 22189709 [Abstract] [Full Text] [Related]
2. Modulation of the mitotic regulatory network by APC-dependent destruction of the Cdh1 inhibitor Acm1. Enquist-Newman M, Sullivan M, Morgan DO. Mol Cell; 2008 May 23; 30(4):437-46. PubMed ID: 18498748 [Abstract] [Full Text] [Related]
3. Acm1 is a negative regulator of the CDH1-dependent anaphase-promoting complex/cyclosome in budding yeast. Martinez JS, Jeong DE, Choi E, Billings BM, Hall MC. Mol Cell Biol; 2006 Dec 23; 26(24):9162-76. PubMed ID: 17030612 [Abstract] [Full Text] [Related]
4. Pseudosubstrate inhibition of the anaphase-promoting complex by Acm1: regulation by proteolysis and Cdc28 phosphorylation. Ostapenko D, Burton JL, Wang R, Solomon MJ. Mol Cell Biol; 2008 Aug 23; 28(15):4653-64. PubMed ID: 18519589 [Abstract] [Full Text] [Related]
10. The pseudosubstrate inhibitor Acm1 inhibits the anaphase-promoting complex/cyclosome by combining high-affinity activator binding with disruption of Doc1/Apc10 function. Qin L, Mizrak A, Guimarães DSPSF, Tambrin HM, Morgan DO, Hall MC. J Biol Chem; 2019 Nov 15; 294(46):17249-17261. PubMed ID: 31562243 [Abstract] [Full Text] [Related]
11. Cdc28 and Cdc14 control stability of the anaphase-promoting complex inhibitor Acm1. Hall MC, Jeong DE, Henderson JT, Choi E, Bremmer SC, Iliuk AB, Charbonneau H. J Biol Chem; 2008 Apr 18; 283(16):10396-407. PubMed ID: 18287090 [Abstract] [Full Text] [Related]
12. Timely activation of budding yeast APCCdh1 involves degradation of its inhibitor, Acm1, by an unconventional proteolytic mechanism. Melesse M, Choi E, Hall H, Walsh MJ, Geer MA, Hall MC. PLoS One; 2014 Apr 18; 9(7):e103517. PubMed ID: 25072887 [Abstract] [Full Text] [Related]
13. Inhibition of APCCdh1 activity by Cdh1/Acm1/Bmh1 ternary complex formation. Dial JM, Petrotchenko EV, Borchers CH. J Biol Chem; 2007 Feb 23; 282(8):5237-48. PubMed ID: 17178718 [Abstract] [Full Text] [Related]
14. "Reductional anaphase" in replication-defective cells is caused by ubiquitin-conjugating enzyme Cdc34-mediated deregulation of the spindle. Khong JH, Zhang T, Gunaratne J, Blackstock W, Surana U. Cell Cycle; 2012 Aug 01; 11(15):2896-910. PubMed ID: 22805765 [Abstract] [Full Text] [Related]
15. Expression of constitutively active CDK1 stabilizes APC-Cdh1 substrates and potentiates premature spindle assembly and checkpoint function in G1 cells. Ma Y, Yuan X, Wyatt WR, Pomerening JR. PLoS One; 2012 Aug 01; 7(3):e33835. PubMed ID: 22479455 [Abstract] [Full Text] [Related]
16. Anaphase promoting complex-dependent degradation of transcriptional repressors Nrm1 and Yhp1 in Saccharomyces cerevisiae. Ostapenko D, Solomon MJ. Mol Biol Cell; 2011 Jul 01; 22(13):2175-84. PubMed ID: 21562221 [Abstract] [Full Text] [Related]
18. The regulation of Cdc20 proteolysis reveals a role for APC components Cdc23 and Cdc27 during S phase and early mitosis. Prinz S, Hwang ES, Visintin R, Amon A. Curr Biol; 1998 Jun 18; 8(13):750-60. PubMed ID: 9651679 [Abstract] [Full Text] [Related]
20. Inactivation of Cdh1 by synergistic action of Cdk1 and polo kinase is necessary for proper assembly of the mitotic spindle. Crasta K, Lim HH, Giddings TH, Winey M, Surana U. Nat Cell Biol; 2008 Jun 18; 10(6):665-75. PubMed ID: 18500339 [Abstract] [Full Text] [Related] Page: [Next] [New Search]